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22 Cards in this Set

  • Front
  • Back
what are the life threating arrhythmias
Ventricular tachy
MI c PVC's
SVT c hypotension
what is the difference btw stable and unstable V Tach
mental status and BP
sick sinus synd
tachy and brady rythms usually seen in the elderly
near syncope and syncope
Pre-excitation synd
short PR interval
or
Delta wave c tachydysrythmia
what should you do to confirm CAD
a stress test
signs of thyrotoxicosis
Tachy
fever
dehydration
exopthalmos
thyromegaly
Hx of thyroid disease
signs of pheochromocytoma
HTN
Tachy
ele catecholamines
what are the characteristic heart sounds for an MVP
Mid-systolic click c late systolic murmur
"click-murmur syndrome"
what is the most common click
systolic ejection click (aortic or pulmonic valves)
key points to remember about SVT
narrow QRS
vagal maneuvers reduce them (like valsalva)
what do you order if you suspect MVP?
an ECHO
MVP key points
More common in females age 14 - 30
most asymptomatic
ECHO shows LV dilation and inc LV wall thickness
Mid to late systolic click followed by murmur
sudden death is a rare complication
EKG- ventricular arrhythmia's (paroxysmal SVT & VT)
What has the greatest effect on mortality rates in the treatment of MI's
if you can initiate thrombolytic therapy within the first 1-2 hours
mortality=1.3%
what are the benefits to thrombolytic therapy for MI's within 2-12 hours
though there wont be a great deal of myocardial salvage, there will be: less adverse ventricular remodeling, reduced aneurysms, and improved electrophysiologic stability
how do you recognize an acute MI based on history
Pressure, squeezing, heaviness, something sitting on my chest
radiation of pain to arms, jaw, teeth
diaphoresis, N/V, dyspnea, syncope, palpitations, edema, cough, confusion, weakness
DM, Male, smoker, over 50, FH
clinical presentations of MI on PE
altered mental status
poor perfusion
diaphoresis
rales
JVD
S3 S4
bradycardia
EKG of AMI, inferior wall
ST segment elevation in leads I, II, aVF, V1-4

Must run serial EKGs
initial therapeutic intervention for MI
Airway
Breathing- supp O2
Circulation- distal pulse and BP
Access line immediately
further therapeutic intervention for MI (after ABCs)
1- thrombolytic therapy
2- B-blockers and Ca++ channel blockers
3- NTG
4- anti-inflammitory agents
5- Aspirin and heparin
effects of NTG treatment
dec preload and afterload
vasodilation of CAs

poss hypotension- resolved w/ IV saline 250-500 cc bolus
effects of morphine use in MIs
analgesic
anti-anxiety
dec O2 consumption
vasodilation

(2-5 mg every 5 to 30 min)
use of and contraindications for B-blockers in the treatment of MI
decreased HR, contractility

contra: CHF, COPD, reactive airway disease, Bradycardia, Hypotension