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71 Cards in this Set

  • Front
  • Back
Gestational age
-premature
-full term
-post mature
-premature-37 weeks
-full term-38-42
-post mature-42 weeks
Low birth weight (LBW)
very low birth weight (VLBW)
extremely low birth weight (ELBW)
-LBW-<2500g
-VLBW-<1500g
-ELBW-<1000g
AGA-appropriate for gestational age
-when the birth weight falls between 10th-90th% intrauterine growth curves
Small for gestational age (SGA)
-birth weight below 10%
-rate of intrauterine growth restricted
-also called small for date (SFD)
Intrauterine growth restriction (IUGR)
-does not grow as expected in utero
-symmetric IUGR-infant's weight, length, and head circumference are all affected
-asymmetrical IUGR-birth wt below 10%; head circ is normal
Large for gestational age (LGA)
-birth weight falls above 90%
-infant of diabetic mom
Ballards exam (gestational age) has two components
-physical maturity
-neurologic and or neuromuscular development evaluations
-score given in each area
-added up=gestational age
-also needed weight, length, head circ.
Preterm infants risks include
-immature organs
-lack physical ability to live outside of mom
-wide range of birth wts and problems
-lower the weight and gestational age, the less chance of survival
Incidence of preterm births are highest among?
-low socioeconomic groups
-lack prenatal care
causes of preterm births
-age
-smoking
-poor nutrition
-placental problems
-preeclampsia/eclampsia
-incompetent cervix
-multiple babies
-infections
-exposure to harful substances
Severity of problems can be related to?
-baby's age
-greater chance of complication the earlier infant is born
Respiratory complications in preterm baby's
-decreased/immature # of alveoli
-develop in utero at 26-28
-decreased surfactant
-smaller lumen in resp system
-immature/fragile capillaries in lungs
-skeletal muscles weak-difficulty positioning head/neck, problems maintaining patent airway
Surfactant does what
-lines the lung tissue
-acts like a detergent
-when present helps alveoli open easily
-when not present difficult to expand lungs
-infant must exert great effort with each breath
-eventually becomes exhaused-atelectasis
All resp problems can lead to
-respiratory distress
-expiratory grunting
-retractions
-tachypnea
-skin color pale/cyanotic
Retraction types
-subcostal -low bottom of ribs
-substernal
-intercostal
-suprasternal-high by clavicals
Apnea
-no breathing for at least 20 seconds
periodic breathing
-common in preterm infants
-5-10 sec resp pauses followed by 10-15 seconds of compensatory rapid respirations
Interventions/Treatment for resp distress
-assess resp status q 1-2 hrs
-cont pulse ox
-arterial blood gases
-HR, BP, RR-monitored continuously
-admin of O2
Goal of administration of O2
-provide adequate oxygen
-avoid negative effects of O2 administration
Surfactant replacement therapy
-Adj to O2 and ventilation therapy
-infants <29 wks with resp distress syndrome (RDS)
-given via ET
Betamethasone for mom does what
-enhances fetal lung development
-needs to be within 24 hrs of birth
Potential long term effects with preterm respiratory problems
-respiratory distress syndrome
-chronic lung disease (bronchopulmonary dysphasia)
-retinopathy of prematurity (ROP)-damage to retinea cause of O2
S/S of cardiovascular function problems that you would report
-hypotension
-cap refill >3 sec
-tachycardia (initially, than bradycardia)
-cont. resp. distress
Why are preterm infants at risk for poor themperature regulation?
-poor muscle tone
-immature terperature-regulating center in brain
-less fat
-cold stress can lead to man problems
Sources of external heat
-radiant warmer
-incubator or isolette
-kangaroo care
Meathods to help maintain a neutral thermal environment (NTE)
-minimize drafts
-prewarm all surface
-bathing-keep covered, warm water
-knitted caps and booties
-warm moist O2
CNS is well developed by?
24 weeks
Preterm babies are susceptible to injury due to?
-birth trauma
-bleeding from fragile capillaries
-impaired coagulation
-episodes of hypoxia
-susceptible to hypoglycemia
5 areas to look for in preterm infants
-seizure activity
-hyperirritability
-CNS depression
-increased intracranial pressure
-abnormal movements-reflexes should be present and normal for infant's age
Digestive system characteristics of preemies
-stomach small
-poor muscle tone (cause vomiting)
-gag and cough reflexes poor
-decreased absorption of fat
-limited ability to convet glucose to glycogen
-lacks sucking until 32-34 wks
-gavage feedings may be necessary until sucking reflex occurs
-give baby soft preemie nipple to stimulate sucking as they receive gavage feedings
1-2 cc/hr..always check residual!
Liver/Renal issues
-immature kidneys
-immature liver
-cannot conjugate billirubin=jaundice
-treat with phototherapy
Ways to encourage bonding w/ parent
-visiting-parents/sibilings
-kangaroo care
-twins co-bedding
-positioning
-Small for gestation age (SGA)
-less than 10% on the newborn classification chart
-two types of SGA's
Symmetric
-infant looks normal but is very small
-usually problem happens during first trimester (infection)

Asymmetric
-later in pregnancy
-long arms/legs; looks like a "skinny old man"
-usually weight <10%; length hc >10%
Maternal Factors contributing to SGA
-poor nutrition
-adv diabetes
-preg induced HTN
-moms who are smokers or drug users
-age over 35
-placental problems r/t nutritional needs
Fetal causes of SGA
-intrauterine infection
-chromosomal abnormalities and malformations
Assessment findings of the skin in SGA
-loose and dry
-little fat
-little muscle mass
other assessment findings of SGA
-small body
-sunken abdomen
-thin, dry umbilical cord
-little scalp hair
-wide scalp sutures
-respiratory distress
-hypoglycemic
-tremors
-weak cry
-lethargic
Large for gestational age )LGA) newborn
-birth weight >90% on newborn classification chart
-may be preterm, term or post-term
Causes of LGA
-mother with poorly controlled DM
-multiparity
-infant w/ trnasposition of the great vessels
-genetic predisposition
Problems associated with LGA
-may require c-section
-higher incidence of birth truma w/ vaginal delivery-clavical, skull fx, cephalhematomas
-fetal distress
-hypoglycemia
-polycythemia-hyperbilirubinemia
Physical findings in LGA
-weight > 4000g (8-14.5)
-caput seccedaneum
-cephalhematoma
-facial nerve damage
caput seccedaneum
-edema on top of head where is pushed against cervix during labor
cephalhematoma
-blood collection due to rupturing during birth
Infant of DM mother major problem
-hypoglycemia-glucose <40 mg/dl
Other symptoms R/T DM mother
-jitteriness/tremors
-lethargy
-tachypnea
-hyperbilirubineamia->12
-gavage if resp >60

-feeding difficulities
Interventions of infant with DM mother
-monitor glucose at birth
-every 2 hrs for first 8 hrs
-every 4 hrs for 24 hrs
-offer glucose, breast milk , formula before 4 hrs
Group B strep (GBS)
-major cause of infection in newborns
-natural inhabitant of female genital tract
- penumonia 20% die in 24 hrs
-meningitis 2-4 wks of age, 50% w/ brain damage
TORCH
-group of maternal infectious diseases
-can lead to serious complications in embryo, fetus and neonate
-symptoms often not seen in mom
TORCH stands for?
T-toxoplasmosis
O-other; hep B, HIV, syphilis, gonorrhea, varicella zoster
R- rubella
C-CMV cytomegalovirus
H- herpes simplex virus (HSV)
toxoplasmosis
-from cat box
-raw meat
-often spontanously abort
-still births, neonatal deaths, severe congenital anomalies, seizures, coma
rubella
-greatest risk is in 1st trimester
-effects congenital heart disease, IUGR, cataracts, mental retardation, hearing impairments, microcephaly, extensive fetal malformations
CMV cyomegalovirus
-member of herpes virus
-transmitted via placenta or cervix
-cause of viral infections in the fetus are brain, liver, blood damage
-common cause of MRDD
-effects hearing SGA infant
-Antiviral drugs cannot prevent CMV or treat the neonate
Herpes virus type II
-fetus is exposed from placenta or genitalia during delivery
-may be asymptomatic 2-12 days
-can develope jaundice, seizures, fever, vesicular lesions, stomatitis
treatment for herpes virus type II
-c-section
-acyclovir 21 days to infant
-health care workers with active lesions cannot care for babies
syphillis S/S
-vesicular lesions on soles, palms; irritabilty
-SGA, failure to thrive, rhinitis, red rash at mpouth and anus, copper rash of face, soles, palms
Treatment for syphillis
-PCN, isolation
-cover baby hands to prevent skin trauma from scratching
Hep B
babies routinely vacc at birth
-babies + moms given immunoglobin to decrease infection possibility
Hemolytic disease of the newborn
--occurs when blood group of mom and baby are different
Rh incompatibiltiy
-isoimmunization or Rh sensitization
-10-15% caucasian couples
-5% african american couples
Rh incompatibility affects who
-only Rh+ offspring of an Rh- mother is at risk
Rh incompatibility is
-some Rh + blood enters mom's blood
-as placenta separates, more enters mom's blood
mom becomes sensitized to this Rh + blood and makes antibodies
-next pregnancy fetus compenstaes and produces large numbers of immature erythrocytes to replace the destroyed ones
-Cont RBC destruction = anemia = jaundice and marked fetal edema = CHF
Erythroblastosis fetalis
red blood cells dying or being destroyed
Kernicterus
yellow staining in the brain, nerological damage
sensitization of the Rh factor can occur during
-pregnancy
-birth
-abortion
-amniocentesis
Rogam
-covers the mom's antibodies so they won't find and destroy the babies RBC's
Indirect Coombs test
-on mother
-to determine if Rh- mom has developed antibodies to Rh antigen
Direct Coombs test
-on baby's blood
-ID's maternal antibodies attached to fetal RBC's
-if + can start getting jaundice
How soon does the Rh immune globuline have to be give after birth to prevent sensitization in a mom with fetomaternal transfusion
72 hours after birth
-also given at 28 weeks
Kleihauer-Betke test
-if large fetomaternal transfusion is suspected this test detects amount of fetal blood in maternal circulation
ABO incompatibility
-more common than Rh incompatibility
-causes less severe problems
-mom's blood is O and fetus blood is A, B, or AB
-first borns can be affected