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59 Cards in this Set

  • Front
  • Back
what does IHSS stand for?
idiopathic hypertrophic subaortic stenosis=SAM + ASH
what is sam caused by?
venturi effect(suction of the anterior mitral valve leaflet)
-the closer the leaflet comes to being in contact w/ the IVS, the more severe the obstruction
differentiate btw mild sam, mod sam, and severe sam?
mild-leaflet>10mm from IVS
mod-leaflet within 10mm of IVS
severe-leaflet touches IVS for >30% of systole
explain dynamic stenosis? HOw does this change the appearance of the doppler waveform?
-occurs only in mid-late systole and severity of the obstruction can be altered by loading conditions(preload)
-by changing preload, the doppler waveform changes to a dagger shape
what part of the cardiac cycle does dynamic stensois occur? What can you do to increase the velocity?
mid-late systole=preload dependant
-do valsalva to increase velocity
what is the m-mode of subaortic stenosis like?
-ASH, concentric LVC, apical hypertrophy, mid ventricular hypertrophy
-SAM-may creat LVOTO
-b-notch of MV-increased LVEP
-mid systolic closure of the arotic valve due to a sudden decrease in cardiac output
what is the b-notch on m-mode due to?
increased LVEP w/ subaortic stenosis
what part of the cardiac cycle does the AO close w/ IHSS?
mid systole due to sudden decrease in cardiac output
what is the doppler like w/ IHSS? what should you do?
-w/ LVOTO, obtain max PSV by doing valsalva
-diff LVOT from MR
-evaluate diastolic dysfunction
how do you differentiate LVOT from MR?
MR is higher velocity and holosystolic
differentiate amyloidosis from IHSS?
amyloidosis:
-LVH
-Has small PE

IHSS:
-thickened basal septum
-mid-systolic closure
what is seen on echo w/ marfans?
-AO dialated(if >5, surgery is done)
-MVP
-MR and AI
why is the valsalva done for TEE bubble studies?
-decrease in preload
-pressures in Rt are higher than left, so it goes from RT to left.
what is the normal flow velocity for the LAA?
46cm/sec
where are pseudoanerurysms most commonly seen?
inferior wall; true aneurysms in apex
dresslers syndrome
pericarditits caused by myocardial infarct(seen 6 weeks later)
what is the difference btw true and false aneurysms
true:
-involves all 3 layers
-large neck

Pseudo:
-doesn't involve all 3 layers
-small neck
-to/fro flow
What can MAC cause?
a-fib
hemochromatossi
iron overload
hypovolemia
decrease in blood plasma
low fluid which can cause LV to look small
SV formula
Area x TVI
EDV-ESV
between preload and afterload, which one refers to stenosis, and which one refers to regurge?
preload=regurge
afterload=stenosis
what equasions do you use to figure out stenosis severity?
AO stenosis=continuity; can use TVI or peak velocity
MV=plaminetry or 220/PHT
when does the PHT for stenosis not work to accurately assess mitral stenosis?
-AO regurge-due to increase in LV pressures; underestimates the severity of stenosis
-restrictive diastolic dysfunction also underestimates the severity of stenosis because the slope is very sharp to begin with.
what does stenosis look like on 2D?
domes
w/ mitral stenosis, what will the "a" look like on m-mode and doppler?
M-mode=no a
doppler=exaggerated A
when do the anterior and posterior septum move together?
90% of the time w/ mitral stenosis
would tissue doppler be effected w/ tamponade?
no, because you're assessing the myocardium w/ tissue doppler, not the pericardium
what is another name for the sawtooth appearance
cutoff
what e-velocity of the MV will tell us that there is significant MR?
>1,7
what will the vena contracta measure with severe regurge of the AO, and MV?
Ao=>0.7
MV>1.0cm
what is the most common cause of thrombus in the LA?
a-fib
where are myxomas attached?
-the secundum or foramen ovale
what should worry about w/ marfans'
dissection
what usually causes pulmonary stenosis
usually congenital causes
what sign will you see w/ pulmonary hypertension?
flying w
what is normal accel time for the pulmonary valve?
120-130ms
what are the 2 most common thingsthat associated w/ ebsteins?
-ASD
-WPW-wolf parkinson white
eisenmenger's is secondary to what?
pulmonary hypertension
what does cor triatriatum mimic?
mitral stenosis
what do you need to figure out RVSP?
-systolic BP
-peak velocity of VSD or PDA
what echo signs will you see w/ turners syndrome?
-bicuspid AO valve
-coarctation
how often do people w/ coarctations have a abicuspid AO valve?
50% of the time
with williams syndrome, what will you see on echo?
supravalvular stenosis
what indicates the severity of ebsteins?
-see how atrialized the ventricle is
->20mm btw TV and MV=ebsteins
How do you tell the diff. btw coronary sinus, and desc. ao in PSLA?
-Desc. ao is bigger, and outside the heart
-coronary sinus is smaller, and inside the heart
if you see an enlarged coronary sinus, what might you suspect?
-anomolous pulmonary venous return into coronary sinus
what makes something cyanotic?
higher right sided pressures
how are dissections classified?
-stanford and debakey
what is the 1st structure you see on tee?
LA
raffe
bit of tissue that occurs w/ a bicuspid AO valve
what is the main complication of bioprosthetic and mechanical valves?
bioprosthetic=degeneration
mechanical=thrombus
what do you always see w/ disecting aneurysm?
-intimal flap
-true lumen is smaller than false aneurysm
what is the anatomical location of the PV and TV
PV=most superior and anterior
TV=most inferior and posterior
what do you see w/ kawasaki's? what is the best view?
-coronary artery aneurysms and infarcts, abnormal wall segments
-best veiw=short axis
what procedures fix transposiiton?
Jantene and mustard procedure
Jantene=great artery switch
Mustard=atrial switch
if you see a saw tooth, and flow continuous in diastole when dopplering the desc. ao in suprasternal, what should you ssupect?
-coarct
where are most sinus of venous ASD's seen?
-close to the SVC
what should you do if you suspect SEC?
turn up gain to see blood swirling.