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59 Cards in this Set
- Front
- Back
what does IHSS stand for?
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idiopathic hypertrophic subaortic stenosis=SAM + ASH
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what is sam caused by?
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venturi effect(suction of the anterior mitral valve leaflet)
-the closer the leaflet comes to being in contact w/ the IVS, the more severe the obstruction |
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differentiate btw mild sam, mod sam, and severe sam?
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mild-leaflet>10mm from IVS
mod-leaflet within 10mm of IVS severe-leaflet touches IVS for >30% of systole |
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explain dynamic stenosis? HOw does this change the appearance of the doppler waveform?
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-occurs only in mid-late systole and severity of the obstruction can be altered by loading conditions(preload)
-by changing preload, the doppler waveform changes to a dagger shape |
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what part of the cardiac cycle does dynamic stensois occur? What can you do to increase the velocity?
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mid-late systole=preload dependant
-do valsalva to increase velocity |
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what is the m-mode of subaortic stenosis like?
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-ASH, concentric LVC, apical hypertrophy, mid ventricular hypertrophy
-SAM-may creat LVOTO -b-notch of MV-increased LVEP -mid systolic closure of the arotic valve due to a sudden decrease in cardiac output |
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what is the b-notch on m-mode due to?
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increased LVEP w/ subaortic stenosis
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what part of the cardiac cycle does the AO close w/ IHSS?
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mid systole due to sudden decrease in cardiac output
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what is the doppler like w/ IHSS? what should you do?
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-w/ LVOTO, obtain max PSV by doing valsalva
-diff LVOT from MR -evaluate diastolic dysfunction |
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how do you differentiate LVOT from MR?
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MR is higher velocity and holosystolic
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differentiate amyloidosis from IHSS?
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amyloidosis:
-LVH -Has small PE IHSS: -thickened basal septum -mid-systolic closure |
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what is seen on echo w/ marfans?
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-AO dialated(if >5, surgery is done)
-MVP -MR and AI |
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why is the valsalva done for TEE bubble studies?
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-decrease in preload
-pressures in Rt are higher than left, so it goes from RT to left. |
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what is the normal flow velocity for the LAA?
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46cm/sec
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where are pseudoanerurysms most commonly seen?
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inferior wall; true aneurysms in apex
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dresslers syndrome
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pericarditits caused by myocardial infarct(seen 6 weeks later)
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what is the difference btw true and false aneurysms
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true:
-involves all 3 layers -large neck Pseudo: -doesn't involve all 3 layers -small neck -to/fro flow |
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What can MAC cause?
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a-fib
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hemochromatossi
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iron overload
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hypovolemia
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decrease in blood plasma
low fluid which can cause LV to look small |
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SV formula
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Area x TVI
EDV-ESV |
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between preload and afterload, which one refers to stenosis, and which one refers to regurge?
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preload=regurge
afterload=stenosis |
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what equasions do you use to figure out stenosis severity?
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AO stenosis=continuity; can use TVI or peak velocity
MV=plaminetry or 220/PHT |
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when does the PHT for stenosis not work to accurately assess mitral stenosis?
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-AO regurge-due to increase in LV pressures; underestimates the severity of stenosis
-restrictive diastolic dysfunction also underestimates the severity of stenosis because the slope is very sharp to begin with. |
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what does stenosis look like on 2D?
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domes
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w/ mitral stenosis, what will the "a" look like on m-mode and doppler?
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M-mode=no a
doppler=exaggerated A |
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when do the anterior and posterior septum move together?
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90% of the time w/ mitral stenosis
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would tissue doppler be effected w/ tamponade?
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no, because you're assessing the myocardium w/ tissue doppler, not the pericardium
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what is another name for the sawtooth appearance
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cutoff
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what e-velocity of the MV will tell us that there is significant MR?
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>1,7
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what will the vena contracta measure with severe regurge of the AO, and MV?
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Ao=>0.7
MV>1.0cm |
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what is the most common cause of thrombus in the LA?
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a-fib
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where are myxomas attached?
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-the secundum or foramen ovale
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what should worry about w/ marfans'
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dissection
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what usually causes pulmonary stenosis
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usually congenital causes
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what sign will you see w/ pulmonary hypertension?
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flying w
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what is normal accel time for the pulmonary valve?
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120-130ms
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what are the 2 most common thingsthat associated w/ ebsteins?
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-ASD
-WPW-wolf parkinson white |
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eisenmenger's is secondary to what?
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pulmonary hypertension
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what does cor triatriatum mimic?
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mitral stenosis
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what do you need to figure out RVSP?
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-systolic BP
-peak velocity of VSD or PDA |
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what echo signs will you see w/ turners syndrome?
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-bicuspid AO valve
-coarctation |
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how often do people w/ coarctations have a abicuspid AO valve?
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50% of the time
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with williams syndrome, what will you see on echo?
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supravalvular stenosis
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what indicates the severity of ebsteins?
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-see how atrialized the ventricle is
->20mm btw TV and MV=ebsteins |
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How do you tell the diff. btw coronary sinus, and desc. ao in PSLA?
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-Desc. ao is bigger, and outside the heart
-coronary sinus is smaller, and inside the heart |
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if you see an enlarged coronary sinus, what might you suspect?
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-anomolous pulmonary venous return into coronary sinus
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what makes something cyanotic?
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higher right sided pressures
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how are dissections classified?
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-stanford and debakey
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what is the 1st structure you see on tee?
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LA
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raffe
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bit of tissue that occurs w/ a bicuspid AO valve
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what is the main complication of bioprosthetic and mechanical valves?
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bioprosthetic=degeneration
mechanical=thrombus |
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what do you always see w/ disecting aneurysm?
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-intimal flap
-true lumen is smaller than false aneurysm |
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what is the anatomical location of the PV and TV
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PV=most superior and anterior
TV=most inferior and posterior |
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what do you see w/ kawasaki's? what is the best view?
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-coronary artery aneurysms and infarcts, abnormal wall segments
-best veiw=short axis |
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what procedures fix transposiiton?
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Jantene and mustard procedure
Jantene=great artery switch Mustard=atrial switch |
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if you see a saw tooth, and flow continuous in diastole when dopplering the desc. ao in suprasternal, what should you ssupect?
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-coarct
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where are most sinus of venous ASD's seen?
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-close to the SVC
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what should you do if you suspect SEC?
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turn up gain to see blood swirling.
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