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20 Cards in this Set
- Front
- Back
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causes hyperkalemia and gynecomastia?
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spironolactone
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decreases mortality
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ACE-I,
ARB, Nitrates $ hydralazine, B-blockers, aldosterone antagonist(spironolactone and eplerenone) |
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ototoxicity and metabolic alkalosis
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diuretics
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NYHA class III-IV
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aldosterone antagonist
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c/I of aldosterone antagonists
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crcl < 30ml/min
K > 5 |
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Ca2+ dependent tissues in which myocardial cells?
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SA and AV node
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Na+ dependent tissues in which myocardial cells?
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Atria, ventricles and purkinje fibers
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heart's dominant center of automaticity
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SA node
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where does sinus bradycardia and tachycardia orignate?
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SA node
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resting range is/
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60-100bpm
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what are the 3 conduction pathways in the RIGHT atrial from SA node to AV node
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Internodal tracts
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tract of depolarization of the LEFT atrium
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Interatrial tract - bachman's bundle
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alternative pathway between the atria and ventricles during formation of heart
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wolff-parkinson-white syndeome
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highest conduction velocity - where?
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purkinje fibers
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where does the normal sinus rhythm originate from?
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sinus node
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where is spontaneous automaticity generated?
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in latent pacemakers (atrial fibers, fibers of AV junction and purkinje fibers)
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increased slop of phase 4 depolarization can lead to what kind of tacchyarrhythmia?
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spontaneous automaticity
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what is the transient depolarization that occurs during repolarization (phase 3)?
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early afterdepolarization - a form of triggered automaticity
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transient membrane depolarization that occurs after repolarization but prior to phase 4
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delayed afterdepolarization
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what kind of triggered automaticity is precipitated by:
1> catecholamines, digoxin 2> hypokalemia, antiarrhythmic drugs |
1> DAD
2> EAD |
