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20 Cards in this Set

  • Front
  • Back
causes hyperkalemia and gynecomastia?
spironolactone
decreases mortality
ACE-I,
ARB,
Nitrates $ hydralazine,
B-blockers,
aldosterone antagonist(spironolactone and eplerenone)
ototoxicity and metabolic alkalosis
diuretics
NYHA class III-IV
aldosterone antagonist
c/I of aldosterone antagonists
crcl < 30ml/min
K > 5
Ca2+ dependent tissues in which myocardial cells?
SA and AV node
Na+ dependent tissues in which myocardial cells?
Atria, ventricles and purkinje fibers
heart's dominant center of automaticity
SA node
where does sinus bradycardia and tachycardia orignate?
SA node
resting range is/
60-100bpm
what are the 3 conduction pathways in the RIGHT atrial from SA node to AV node
Internodal tracts
tract of depolarization of the LEFT atrium
Interatrial tract - bachman's bundle
alternative pathway between the atria and ventricles during formation of heart
wolff-parkinson-white syndeome
highest conduction velocity - where?
purkinje fibers
where does the normal sinus rhythm originate from?
sinus node
where is spontaneous automaticity generated?
in latent pacemakers (atrial fibers, fibers of AV junction and purkinje fibers)
increased slop of phase 4 depolarization can lead to what kind of tacchyarrhythmia?
spontaneous automaticity
what is the transient depolarization that occurs during repolarization (phase 3)?
early afterdepolarization - a form of triggered automaticity
transient membrane depolarization that occurs after repolarization but prior to phase 4
delayed afterdepolarization
what kind of triggered automaticity is precipitated by:
1> catecholamines, digoxin
2> hypokalemia, antiarrhythmic drugs
1> DAD
2> EAD