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35 Cards in this Set

  • Front
  • Back
What are the 3 diseases we are vaccinating for in cats? Give their causative agents.
1. Feline Panleukopaenia/ Feline Infectious Enteritis : Caused by feline panleukopenia virus
2. Feline respiratory disease: Feline Herpesvirus (FHV-1), Feline Rhinotracheitis Virus (FRV)
3. Feline calicivirus (FCV)
What are the risk groups for feline panleukopaenia virus and mode of transmission?
Affects all cats.
Direct faecal oral route or indiret via environment.
What are the risk groups for Feline herpes virus/Feline rhinotracheitis virus and mode of transmission?
Multicat establishments.
Direct and indirect contacts with aerosols / fomites
What are the risk groups for Feline calicivirus and mode of transmission?
Multi cat establishments and spread via cat to cat contact or fomites.
What are the clinical signs of feline enteriris?
Affects mainly kittens causing GI signs, pyrexia,depression, dehydration and shock. Panleucopenia on blood tests. Queens may abort or kittens are born with cerebellar hypoplasia.
Describe what two types of vaccine are for Feline panleukopaenia ? what categories are they unsafe for?
1. killed vaccine = sade in pregnant queens/kittens
2. Modified vaccine with long lasting immunity is unsafe in pregnant queens, requires second dose after 12 weeks of age.
What are the clinical signs for feline herpes/Feline rhinotracheitis ?
Flu- like signs of sneezing, ocular discharge, fever, anorexia, malaise. Serious signs incluse conjunctivitis, oedema, corneal ulceration, keratitis.
Queens may abort.
What type of vaccine is recommended to cover feline respiratory diseases?
killed vaccines preferred to reduce chances of reversion to virulence.
What clinical signs does calici virus present with ?
Causes flu like signs of sneezing, ocular discharge, fever, anorexia, malaise.
What are the 3 non- core vaccines?
Chlamydiosis , FeLV and FIV
What are the clinical signs of Chlamydophila felis?
They are sneezing, ocular discharge, fever, anorexia, malaise. Conjunctivitis or chemosis of one or both eyes.
What are the problems of vaccinating for chlamydia?
1. Both modified and killed vaccines do not prevent infections or shedding of organism.
2. Affects kittens less than 6 weeks. hence to prevent, vaccinate queens.
3. Adult cats dont need it.
4. Problems of catteries.
What can you use to treat chlamydophila felis?
Doxycycline.
Feline Leukaemia Virus affects which class of animals? What can it cause?
Mosty kittens, can lead to neoplasia, immunosuppression. Will kill the kittens when they are young.
When should we vaccinate for Feline Leukaemia Virus?
Outdoor cats or cats living with FeLV positive cats. Test prior as positive cats will not benefit.
Where should you vaccinate F3?
Left forelimb as distally as possible.
What types of cats are affected by FIV? who should you vaccinate?
Mostly male older cats as its spread by biting.
Vaccinate outdoor cats or if its living with FIV +ve cat.
Ideally test the cat. if positive, keep indoors, if negative vaccinate.
Compare the pros and cons of modified live vaccines and inactivated killed vaccines.
Modified:
Rapid onset of protection.
Disadvantages: vaccine induced disease, especially pregnant or immunocompromised animals.
Reversion to virulence is possible.

Inactivated vaccines:
Pros: Reversion to virulence cannot occur. No shedding of virus.
Safe in pregnant /immunocompromised animals
More highly associated with inflammatory reactions at vaccination sites and serious reactions such as anaphylaxis.
Where should we vaccinate FeLV?
In the distal left hind.
When should you vaccinate in kittens?
6-8 weeks, 12 weeks, 16-18 weeks.
Annual booster recommended.
Cats presented over 12 weeks of age with unknown vaccination status. How do you vaccinate for them?
Require 2 vaccinations 4 weeks apart.
What are the vaccination protocols with FeLV?
require two boosters 2-3 weeks apart with the first one after nine weeks of age.
What are common vaccination reactions?
Pyrexia, anorexia, malaise, depression, pain. Lameness with live calicivirus vaccine.
May lead to sarcoma.
What is in c3?
Canine parvovirus(CPV), Canine Distemper Virus and canine adenovirus-1 ( canine infectious hepatitis).
What are the mode of transmission of parvovirus?
Direct and indirect oronasal contact with virus particles ( shed in faecal material).
What is the pathogenesis of parvovirus?
The virus replicates initially in the lymphoid tissue of the oropharynx, mesenteric lymph nodes and thymus. The virus then disseminates to the intestinal crypts of the small intestimes causing destruction and collapse of the epithelium. The virus also further infects GI epithelium, lymphoid tissue and other tissue.
What are the clinical signs of parvo virus?
Vomiting, diarrhoea( BLOODY), anorexia, dehydration, leokopaenia, pyrexia.Occasionally neurological signs, cardiac dysfunction, myocarditis. Occurs to puppies <1 year. Pups are prone to gastroenteritis.
How is canine distemper transmitted?
Direct (oronasal) contact with infected respiratory respiratory droplets or aerosols; also urine
What are the 3 phases of canine distemper disease?
1. Pyrexia/Fever with leukopaenia
2. Mucosal phase: ocular and nasal discharge, coughing, vomiting, diarrhoea, callusing of the nose and foot pads.
3. Neurologic phase: muscle spasms, convulsions and progressive paralysis.
How is Canine Adenovirus transmitted?
Direct oronasal contact with infected secretions, often urine.
What agents does canine cough consists of?
Various, but most commonly Bordetella bronchoseptica. Canine paranfluenza virus, canine adenovirus-2. Bordetella bronchoseptica may act as a primary pathogen, especially in dogs less than 6 months old.
What is the mode of transmission of kennel cough?
Direct and indirect oronasal contact with infected respiratory secretion, including aerosols.
Bronchoseptica: what is the pathogenesis? Differentiate pathogenesis between CPV and CAV-2.
BB attaches to and replicates on cilia of respiratory epithelium. It produces toxins that impair phagocytic function and induce cilostasis.
CPiV replicates within and damages upper respiratory tract epithelium.
CAV-2 replicates in the epithelium of the nasal mucosa, phyranx and respiratory tract epithelium.
What are the clinical signs of kennel cough?
Honking cough and retching, ofen with expectoration of foamy mucus.
Mucoid to ucopurulent nasal and ocular discharge, bronchopneumonia, lethargy, anorexia, dyspnoea.
BB: rhinitis, mucous nasal discharge, cough.
CPiC- dy hacking cough, serous nasal discharge.
CAV-2 tonsilitis and or lungs consolidation.
What are the vaccination protocol for a puppy?
6-8 weeks : C3 ( KC optional)
10-12 weeks: C3 (KC optional)
14-16 weeks : vaccination if they did not recieve one at 6-8 weeks of age.