• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/34

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

34 Cards in this Set

  • Front
  • Back
Type III Atresia of SI
blind pouches with no attachment
Atreasia of SI: like type III’s, but have a v-shaped mesenteric defect.
Type IV
Failure of intestines to return to abdominal wall at 10-11 weeks; enclosed herniation from umbilical stump
Omphaloceal
Nonenclosed paraumbilical defect
Gastroschisis
Persistent viteline duct usually involving ilium.
Meckel's Diverticula (may contain gastric mucosa or pancreatic tissue (most common); may cause intussusception)
Mesenteric Diverticula
congenital defect in muscular wall
Heterotopic pancreas tissue MC in what part of GI tract?
duodenum (least in jejunum); usually without islets
May act as a lead point for intussusception
Heterotpic pancreatic tissue
One portion of the small bowel becomes telescoped into a distal portion and is propelled forward by peristalsis.
Intussusception (MC in kids)
MC place for a volvulus to occur in the GI tract
Sigmoid, then cecum, then small bowel
DIfference between arterial and venous occulsions of a Transmural Infarction (Acute)
Arterial occlusions usually have sharp borders while those caused by venous occlusion fade into normal mucosa
Part of GI tract most susceptible to a transmural infarction (acute)
The splenic flexure is most susceptible: it is a “watershed,” between portions of the bowel fed by the superior and inferior mesenteric arteries. It is often hypo-perfused during episodes of shock, or hemodynamic compromise
Usual cause of chronic or low grade infarction of ischemic bowel disease
mostly venous cause (atrophic changes and fibrous scarring of the lamina propria develop with rare stricture formation
)
SB ischemia where Mucosal infarcted, but muscular wall and serosa are not; due to nonocclusive hypoperfusion (sepsis, shock, heart failure)
Hemorrhagic Gastroenteropathy
the alcohol-soluble portion of gluten
Gliadin
Gliadin peptides induce some epithelial cells to produce ____ in Celiac sprue
IL-15
Genetic susceptibility to Celiac sprue
HLA DQ2 and DQ8
Dermatitis herpetiformis correlated with this disease
Celiac sprue
Increased incidence of what cancers in Celiac sprue?
T-cell lymphoma and small bowel adenocarcinoma
mucosa flat, villi lost (profound loss of absorptive surface), with infiltration of lymphocyte (CD8+).
Celiac sprue
Tropical sprue may be related to what?
ETEC, cyclospora
Type of anemia assoc with Tropical Sprue
Megaloblastic (folate and/or B12 <)
> breath H2
Lactase Def
Individual cannot secrete triglyceride rich lipoproteins.
Abetalipoproteinemia
Leads to vitamin deficiency and membrane defects (burr RBCs) due to inability to absorb fats
Abetalipoproteinemia
Rx for Abetalipoproteinemia
Avoid dietary fat; massive Vit E supplements
MC neoplasms of small bowel
Adenoma
Brunner's Gland "Adenoma"
Small mass of normal elements; beleived to be hyperplasia; really NOT a neoplasm
Bands of smooth muscle support glands (hamartomas)
Peutz-Jeghers Syndrome Polyps
Where do most adenocarcinomas of the small bowel arise?
duodenum; however, MUCH less common than in the colon
MOST COMMON MALIGNANT TUMOR OF SMALL BOWEL
Carcinoid Tumor
MC location of Carcinoid tumor
appendix or ileum
MC concurrent malignancy of Carcinoid Tumor
Gastric adenocarcinoma
Neoplasm of SI that is Composed of 3 cell types: endocrine cells, ganglion cells, and Schwann-like cells.
Gangliocytic Paraganglioma