• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/116

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

116 Cards in this Set

  • Front
  • Back
where do you get an intestinal voluvlus
at the root of the mesentery
omentum and all mesentery gets twisted- no blood supply
when will a cow start colicing like a horse
when they have intestinal volvulus, they will throw themselves on the ground, kick their belly, and vocalize
rectally what can you palpate with a IV
going to feel the voluvlus and the loops of dilated bowel
tx for IV
surgery
trichobenzoars in cows
will usually sit in the rumen, sometimes travel into the intestines and cause blockage
CS of trichobenzoars
usually seen in calves
go off feed
abdomen may be distended
pain inc., dehydration
how can you dx trichobenzoars
US
tx for trichobenzoars
sx correction
intuss in cow CS
vague signs of colic
abd pain inc. HR
abdomen distends
what will you see on US with intuss
bulls eye lesion
what happens to fecal production in cows with intuss
stops
mesenteric fat necrosis
inflammatory response around degenerating adipose cells
what cause mesenteric fat necorsosi
when grass has endophyes on it causes the fat to calcify, intestines cant get normal peristaltic motion
CS of mesenteric fat
wt los
anorexia
diarrhea
abdominal enlargement
off-feed pings
what kind of cows get mesenterinc fat necrosis
guenersy
what can you find on rectal exam with a cow with mesenteric fat necoriss
rectal strictures may occur, making rectal exam of the abdomen impossible
dx of mesenteric fat necrosis
exploratory lapatotomy
necropsy
tx of mesenteric fat necrosis
no tx approved for cattle in US
isoprotihilane is a fungicide that has been used experimentally used with limited success
cecal dilatation
tend to occur in dairy cattle, usually within 60 days after parturition, can occur anytime, escae of fermentable carbs fromt he foresomach
pathophys of cecal dilatation
cecal flora metabolize these carbs into VFAs, methane, and CO2
this reduces motility and inc. gas production
CS of cecal dilation
reduced appetitte
dec. milk production right PLF (paralumbar fossa) may be distending
dec. manure
dx of CD
rectal
sometimes these cecum can torse on themselves and cause a volvulus
CS of cecal volucua
anorexia
aglacita
tx of cecal volulus dilatoin
dilation- antacids, laxatives, oral/IV fluid, high fiber diet (HAY!)
tx of cecal volvulus
sx
IV fluids
antibiotics
NSAIDs
atresia coli
neonate normal at birth
abdomen becomes distended after several feedings
colic/depression with no fecal production
atresia ani
can you put the thermometer in, nope
dx of atresia coli
hx, PE, have to do a contrast radiograph
rectal prolapse who gets it
small ruminants, feed lot cattle
what causes rectal prolapse
coccidia or pneumonia
rectal prolapse in sheep
when their tails are docked too short and you mess up their nerves
tx for rectal prolapse
tx underlying causes
then do purse string
clostridium black leg
snovii find them dead, or emphysematous legs
what can you use for tx of clostridium
peniliciin
clostridium perfingens
anaerobic spore former
a, c and d are most common
difficult to dx
they all die
dx of clostridium perfingens
PCR
need samples from freshly dead animal
hemorrhagic bowel syndrome
acute, fatal dz of dairy cattle
type A clostridium perfingens
what does type A c. per fringes cause in the GIT of dairy cows
intrualminal clot lunn saw one that was 12 feet long
signalment of HBS
all stages of lactation but tend to be <100 days in milk
median days 104
often foudn dead or moribund
CS of HBS
+/- colic
inconsistent
pale mm
cold extremities
dehydration
rapid, weak pulse
what happens over time with a HBS cow
their abdomen keeps expanding
rectal palpation of HBS
small bowel distension
what will you see on US of HBS
put caual PLF you can see a big clot in dilated intestines
if you're not convinced or you don't have a US for HBS what can you do
measure the abdominal circumference
clin path HBS
hyperglycemia, glucosuria, azotemia, low Na+, low k+, low Cl-, metabolic acidosis
unlike a DA what will clin path be on HBS
low sodium too
tx of HBS
systemic abx (pen, oxytet)
IV fluid
clostridium perfringens C and D antitoxin
laxatives, adsorbents PO
pro kinetic drugs- metoclopramide, castor oil, neostigmine
sx of HBS
right paralumbar lapartomy- they don't deal well with standing sx for this so do general anesthesia.
pre-op tx for sx of HBS
IV fluids, NSAIDs, narcotics (morpine IV and/ or epidural)
what will lidocaine do if you give it epidural that morphine wont
takes away motor function
clostridium perfringens A combines with what
aspergillus
dx of C. perfrigens type A
isolate from lesion site in high numbers and with aspergillus
fecal cutters are not diagnostics
prog of HBS
poor
what is omnigen
dietary supplement with yeast and b-vitamins somehow kinda helps with prevention of HBS in a herd
is there a vax for type a c. perf
yes
px of c. perf
make sure you don't feed them moldy food
enterotoxemia
clostridium perfringens type C and D
overeating dz, pulpy kidney dz
classic history of enter toxemia
fast growing lambs or kids
lush pasture or high grain diet or lost of milk
over-abundance of carbs in the intestines leads to overgrowth of clostridium
what do you recommend for lambs or kids that are eating too much
make their milk cold so they slow down
which c. perf causes an exotoxin
type DX
what does the exotoxin do
increases intestinal permeability
necrosis of mucosa and submucosa
edema in a variety of organs, notably te lngs
pathophys c.perf
epsiolon exotoxin leads to encephmalacia neuro signs may develop in some aniasl
what is the result of th epsilon-toxicity
hyperglycemia and glucosura are the result of mass ice hepatic glycogen release caused by this toxin
CS of c. perf
severe depression
complete anorexia
colic/pain
diarrhea (may be bloody)
fever
CS of c. perf cont
sudden death of well fed, rapidly growing aiimasl
ataxia, trembling, stiff gait, opisthonous, confulsoins, coma
Clin path of c. perf
neutorphillic
azotemia
hyperglycemia
glucosurea
dx of c. perf
PCR of gut contents- must be fresh
gross necropsy may be enough, bright red from hyperemia
tx of c. perf
antibiotics (penicillin)
NSAIDs
fluids
type D antitoxin (saanen goats may have a rxn)
px of c. perf
diet adjusted to bring the carb level down
graze for 10-20 min, working up full access by the end of the week
vaccination- two doses give 14-56 days apart before heavy grain feeding
is it OK to not give c.perf vaccines to small ruminants or camelids
its malpractice if you don't
johnes dz
diarrhea pipe stream, BAR, skinny
what is the organism of johnes
mycobacterium paratuberculosis
how long is the incubation time for johns
> 2 years
when do cows pick up johnes
when they are babies, its transmitted fecal-oral
what happens with johnes ?
PLE, weight loss, diarrhea (not sm. ruminants wont get screaming diarrhea from it since they have pelleted feces)
CS of johnes ind. signs
about 95% wont ever show signs
5% show wt. loss then intermittent diarrhea
CS in animals > 2 yo
culled before dx usually
herd signs of johnes
one animal with chronic diarrhea then another several months later
what does johnes do to the intestsintes paht
granulomatous response all over the intestine cant absorb
pathophy of johnes
fecal-oral, M-cells myobacterium from intestines to peyers patches on ileum, phagocytozed by macrophages (intracellular pathogen), granulomatous infiltrate causes malabsorption takes 15 mo. from infection to CS
Type I immune jonhes
degranulation of mast cells- diarrhea
type III immune johnes
Ag/Ab complex
type IV johnes immunological
delayed type hypersensitivity
CS of johnes
often in two or three yo cow near calving
starts out as intermittent diarrhea
progresses to severe diarrhea
CS of johnes cont
dehydration is not a prominent signs
chronic wt. loss
hypoproteinemia (bottle jaw)
death or culling due to the dz is inevitable
clin path of johnes
hypoproteinemia
loss of albumin
who is most susceptible to getting johnes
< 6 mo infection depedn on number of organisms ingested
defense of host
infected calves may recover spontaneously , become carriers, develop CS
mode of transmission johnes
manure, semen, urine
milk and colostrum if they don't clean udder before milking her
newborn calves are susceptible
fecal-oral transmission
in utero
dx of johnes
AGID and ElISA good sensitivity if animal has CS
takes time to build up Ab titer
skinny cow with diarrhea- good candidate for testing
normal cow may be positive but not enough Ab.s to test positive
gold standard of johnes dx
fecal culuture but its very slow growing takes about 12-16 wks
good for testing the whole herd
who are fecal cultures for johnes not reliable in
sheep
more dx for johnes
can do rectal scraping/biopsy to see acid-fast organism not extremely sensitive good specificity
necropsy- thickening of ileocecal jxn, enlarged mesenteric ln.
who doesnt really show anything with johnes
sheep
testing and control of johnes
most herd test in is a combination of ELISA and fecal culture.
ELISA will detect less than 50% of positive animals
control of johnes
protect calves from adults, milk and manure can infect
biosecruity of johnes
reduce manure contamination
for milk- pasteurize the milk it will kill off the johnes
tx for johnes
no legal treatment
what kind of drug can be used for sm. pet ruminants with johnes
isopurinum its the old TB drug for humans
NEVER do this is FOOD ANIMALS
salmonella
zoonotic
can kill
most dz is subclinical
can have acute endotoxic form
true carrier states- asymptomatic carriers, contamination of milk and colostrum
what is the most likely salmonella to be shed to humans
s. typhimurium zoonotic, drug resistant
what is the host adapted salmonella in cattle
s. dublin
lifelong carrier status possible
what is the samonellosis pathophys
host resistance- infectious dose-strain virulence
what population of cows are susceptive for infection of salmonella
babies
and cows about to calve
stressed
transmission of salmonella
fecal oral can be from rodents or birds
where does salmonella like to live
its intracellular, loves the lymphatic system these guys get pneumonia as well
acute salmonellosis
cows near time calving
fever, depression, severe drop in milk production, fetid, watery diarrhea (inflammatory process)
anorexia promotes rumen growth of salmonella (VFA inhib)
neonatal salmonellosis
less than 2 months of age, fed milk from chronic carrier, fever, dehydration, weakness, watery diarrhea (mucosa, fibrin, blood)
septicemic slamonellosis
s. dublin
all ages affected but calves more likely to become seriously ill
zoonotic, endotoxemia, enteritis, meningitis, pneumonia, arthritis sometimes they are so dehydrated and septicemic that they wil slough their ears
clin path of salmonella
neutropenia/ lymphopenia (early)
neutrophllic (later)
fibriogen inc
azotemia
total protein may decrease due to albumin loss
dx of salmonellosis
fecal culture
if carrier have to do serial cultures
rectal biopsy and culture
serology- ELISA for S. dublin
herd screeinging for carrier animals
PCR-bulk milk screening
tx of salmonelloisis
supportve care
iv fluids
NSAIDs
for septic form only abx
florfenicol
ceftiofur
TMS
ampicillin
ISOLATION
control of salmonellosis
host adapted strains
pasteurization of colostrum
testing for carriers and culling
ELISA, culture
vaccine- more against gram neg core Ag , will build up some immunity
if its s. typhimruim
look for rodents and birds
winter dysentery
winter diarrhea in dairy cows in a barn
morb high
coronavirus
recovery spontaneous
CS os winter dysentery
pipe stream diarrhea for about 2 days
anorexia
spontaneously get better
do winter dysentery cows need tx
no they usually recovery w/o tx at most oral fluids
CS of winter dysentery cont
high morb low mort
low grade fever some coughing