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62 Cards in this Set
- Front
- Back
Pt. at risk for F&E imbalence: Addison's Disease
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too little aldosterone which causes loss in sodium and retention of potassium..
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Pt. at risk for F&E imbalence:
Peristalsis |
No peristalisis so water is pushed into 3rd space (where it is not supposed to be)
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Pt. at risk for F&E imbalence:
Cushing's Disease |
too much aldosterone which causes retention of sodium (pt. becomes poofy and swollen)
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Tubules of the Kidneys:
If opened ______ if closed _______ |
If opened you pee out and if closed you retain and do not pee.
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Glomerular Filtration Rate
Increased - Decreased |
Incleased you pee more (fluid loss) if decreased you don't pee (fluid retention)
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Where is ADH made and where is ADH stored?
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ADH is made in the hypothalamus and stored in the posterior pituitary.
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When ADH increases___
When ADH decreases ___ |
increases = water retention
decreases = water loss |
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Where is aldosterone stored and what is resposible for the secretion of aldosterone?
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Stored in the Adrenal gland and secreted by the adrenal cortex
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Increased sodium=__ aldosterone
Decreased sodium= ___ aldosterone |
Decreased aldosterone
increased aldosterone |
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Parathyroid is responsible for what?
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Monitor calcium levels in our blood vessels
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Calcitonin does what?
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Pulls calcium from the blood back into the bone
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What takes calcium from the bone into the blood?
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Parathryoid
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Extracellular fluid is what % of total body weight and what does it consist of?
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20% and consists of the interstital which accounts for 75% of the total extracelluar total (fluid between plasma and cells) and intravascular which accounts for 25% of the total extracellular (plasma)
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What accounts for 40% of the total body weight?
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Intracellular
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Osmosis is what?
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movement of water from low to high concentration. dilute to less dilute
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Diffusion is what?
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particles move from high to low - no energy is required
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Facilitated diffusion is what?
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particles move from high to low - require a carrier molecule (glucose)
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Filtration is what?
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Hydrostatic pressure - movement of fluid from high to low pressure. higher pressure on arterial end (moves out) and lower pressure on venous end (moves in - absorption)
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Active Transport is what?
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movement from low to high- requires energy (sodium potassium pump)
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Cations
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Positivly charged electrolytes
Sodium Na+ , Potassium K+, Calcium Ca++ and magnesium Ma+ |
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Anions
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Negativly charged electrolytes
Chloride Cl-, Bicarbonate HC03- and Phosphate P04- |
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Major intracellular
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Potassium and phosphate
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Major extracellular
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Sodium and chloride
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Isotonic
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same osmolarity as blood. No fluid shifts, stays in the intravascular space. Normal saline and lactated ringers
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Hypotonic
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Lower osmolarity than blood. fluid shifts out of vascular space to hydrate the cells. D5W, 1/2 NS and water we drink. used for dehydrated pts.
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Hypertonic
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osmolarity greater than blood. fluid shifts from cells into the vascualr space (cells strink). D10W, D50W , .3%NaCl
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Isotonic Dehydration
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Water and electrolytes are lost in the same amount..
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What caused Isotonic dehydration?
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GI losses, Hemorrhage, Loop diuretics and profuse sweating
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S&S of Isotonic dehydration
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Decreased BP, Increased Pulse, Flat neck veins, decreased urine output and weight loss
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Treatment for Isotonic dehydration
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Give fluids and sodium diet (salty broths) if can not tolerate give isotonic solution (NS & LR)
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Hypertonic Dehydration
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Loses more water than electrolytes. Fluid shifts from ICF to ECF (cells strink)
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Causes of hypertonic Dehydration
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Renal failure, kidney failure, diabetic isipidus and diarrhea
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S&S of hypertonic dehydration
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Thirst, Deep tendon reflexes, and furrow coating on tounge
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Treatment for hypertonic dehydration
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hypotonic orally (water) if need and IV(1/2 NS and D5W) for diabetes give insulin
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Hypotonic dehydration
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Lose more electrolytes than water. fluid shifts from ECf to ICF (cells swell)
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Hypotonic dehydration causes
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Renal failure, pt. on low sodium diet that is taking diuretics and malnutrition.
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Hypotonic dehydration S&S
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decrease urine output, non pitting edema, decreased BP and increased pulse and mental status changes
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Hypotonic dehydration treatment
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hypertonic solutions (.3%NaCl but slowly because potent to veint)
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Isotonic Fluid Volume Excess
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No fluid shift fluids and electrolyes are gained in equal amounts
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Isotonic Fluid Volume Excess causes
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Renal, Heart and liver failure
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Isotonic Fluid Volume Excess S&S
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Rapid weight gain, increased BP, pitting edema and 3rd spacing.
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Isotonic Fluid Volume Excess treatment
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Fluid restriction and sodium restriction. administer diuretics (heart OK kidney NOT OK) daily weights and strict I&O
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Hypotonic Fluid Volume Excess
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Lose more electrolytes than fluids. fluid shifts from ECF to ICF (cells swell)
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Hypotonic Fluid Volume Excess Causes
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SIADH, repeated tap water enemas and rapid infusions
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Hypotonic Fluid Volume Excess S&S
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Incease BP, convulsions if servere, mental status changes and warm moist skin.
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Hypotonic Fluid Volume Excess Treatment
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Water Restriction, weigh daily or use hypertonic solution .3%NaCl
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Hypertonic Fluid Volume Excess
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VERY RARE. Excessive ingestion of sodium!
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SIADH
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Increase ADH = retain water
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Diabetes insipidus
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Descrease ADH = lose water (pee alot)
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Sodium Normal Values
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135-145
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Hypernatremia
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Skin flushed, Aggitated, Low grade fever and Thirst. Causes include water loss(diuretic and diabetes insipidus) and sodium gains (Cushings) Treatment= water replacement, restrict sodium in diet and diuretics to promote sodium excretion
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Hyponatremia
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Nausea & vomiting, Abdominal distention, Altered LOC, Achy head, Anorexia and Positive Muscle Twitching. Causes= SIADH, addisons disease. treatment= fluid restriction replace oral with salty broths IV with .3% NaCl, assess mental status and seizure precautions
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Potassium Normal Values
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3.5-5.0
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Hypokalemia
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SUCTIONING.Skeletal muscle weakness, U wave(EKG changes), Constipation, toxicity of digoxin, irregular weak pulse, orthostatic hypotension and numbness. Causes= cushings, gastric suction, N&V and loop diuretics. Treatment= potassium supplements or foods high in potassium (bananas, avacodaos and cantalopes) and monitor dig level.
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Hyperkalemia
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MOST DEANGEROUS. Danger of V- fib, Abdominal cramping, Nausea, tingling and numbness, EKG changes, Rate irragular, Cardiac output decreases, unable to move- flaccid paralysis and slow heartrate- bradycardia. causes= excessive K+ intake, use of postassium sparing diuretics, massive cell destruction, acidosis and addisons disease. treatment= give insulin, sodium bicarb, calcium gluconate, kayexalate.
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Phosphate Normal Values
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2.7-4.5
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Calcium Normal Values
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8.5-10.5
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Calcium is decreased and ____ is increased
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Phosphate
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Phosphorus is eliminated from ___
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Bowel movements
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Main cause of hyperphosphateemia =
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alcoholisim
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Treat deficiency of phosphate with
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oral replacement of phosphates and Vitamin D
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Treat excess of phosphate with
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albumin hdroxide with meals to bind to phosphrous
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