Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
62 Cards in this Set
- Front
- Back
really deep Q waves can indicate.... |
an old heart attack |
|
QT = ? |
length of ventricular contraction (systole) interval should not be too long! |
|
what kinds of meds lengthen QT interval? |
psych meds ondansetron (zofran) |
|
LV hypertrophy implies... |
longstanding ventricular HTN |
|
RV hypertrophy implies... |
longstanding pulmonary HTN |
|
change in axis = |
either electrically dead tissue OR more muscle tissue than expected |
|
in LVH, PMI is displaced... |
laterally and inferiorly |
|
pericarditis etiology |
usually viral, usuallys elf-limiting |
|
hemopericardium and pericardial effusion both squeeze the heart, so... |
LV can't stretch/dilate - backup of blood where it can't fill heart = cardiac tamponade = pressure on heart from fluid in pericardial sack |
|
chordae tendinae get stretched... |
when ventricle squeezes, valve backflows = murmur |
|
ionotropic drugs |
increase squeeze (contractility_ ex. epinephrine |
|
endocarditis etiology |
valves are vulnerable to bacteria that like to attach to leaflets (no pain receptors there, so no pain - mental status changes/fever/fatigue) |
|
IV drug users w/ endocarditis - which valve most often infected? |
tricuspid |
|
automaticity |
heart can start and maintain itself for a wile if sugar and electrolytes are tehre |
|
rest phase of heart is to prevent _____ |
tetany prevents rapid depol and repol past a certain point |
|
beta-blockers slow down _____ NS |
sympathetic |
|
sympathetic NS ____ time to threshold for SA node cells |
decreases - increases HR overall! |
|
BP = |
CO x peripheral resistance |
|
CO = |
SV x HR |
|
preload |
pressure required to stretch ventricle as far as it can go |
|
afterload |
how much pressure does it take to empty the ventricle against the pressure in the periphery |
|
preload is usually ____, afterload is usually ____ |
fluid arterial constriction |
|
the greater the size of a lumen (such as in ____), the greater the amount of wall tension/pressure needed to equalize pressure/push out fluid |
dilated cardiomyopathy |
|
beta-blockers reduce ______ |
after-load and slow down HR* |
|
hormone control mechs for BP |
ADH, epi/norepi, antiotensin II, ANP JGA in kidney Thyroxine |
|
neurologic factors for BP |
B1 and B2 adrenergic activity baroreceptors in carotid sinuses/aortic arch |
|
endothelial factors in BP |
nitric oxide prostacyclin thromboxane A2 |
|
walk balance b/w keeping pt wet enough (?) and dry enough (?) |
wet - for kidneys - keep low creatinine dry - don't drown - preload |
|
PR interval is ____ QRS is ______ QT is ______ |
PR <1 big box QRS <3 small boxes QT <2 big boxes |
|
narrow QRS wave means |
signals came from above ventricles (normal contraction) |
|
high circulating badykinin can cause.... |
edema (ACE-Is) |
|
cold lower extremities concerning for... |
oor arterial perfusionp |
|
NSAIDS can reduce the effect of all hypertensive drugs EXCEPT _____ |
calcium channel blockers |
|
thrombolytic therapy drugs |
TPA (converts plasminogen to plasmin, promoting fibrinolysis) Streptokinase |
|
Must constider _____ in adolescent pt with sudden onset SOB with exertion |
hypertrophic ostructive cardiomyopathy (HOCM) |
|
burning chest pain |
GERD |
|
tearingchest pain |
AAA |
|
sharp chest pain |
PE |
|
crushing chest pan |
MI |
|
prinzmetal angina |
pain NOT dependenty on activity - spasm of coronary arteries hearlthy women in teh morning |
|
pleuritic chest pain |
think PE |
|
A1 receptors |
activate = vasoconstrict |
|
A2 receptors |
activate = vasodilate |
|
B1 receptors |
activate = increase conduction velocity/contractility/HR |
|
B2 receptors |
activate = bronchodilation blcok = bronchoconstriction |
|
history questions for angina |
quality, location, timing, onset, precipitating factors, duration, radiation, relief with NT? |
|
NTG can also relieve.... |
gallbladder pain |
|
UA on EKG/biomarkers |
can have ST depression but NO positive biomarkers |
|
you want rate control in an antihypertensive? |
NDHP! CCBs Verapimil and Diltiazem |
|
"typical" MI symptoms may not be seen in... |
elderly, females, DM, psych pts |
|
counseling pts about htn |
"if we can aggressively treat your risk factors, we can prevent you from having these diseases!" |
|
2 things that can show tracheal deviation? |
Pneumothorax and SVC syndrome |
|
Hypertensive emergency is MOSTLY caused by |
acute CHF with pulmonary edema then cerebral infarction/encephalopathy |
|
oxidation of LDL causes |
endothelial injury direct inactiviation of nitric oxide by excess production of free radicals --> reduced nitric oxide causes increase in paltelet adhesion + pro-coagulant effects |
|
effects of tobacco |
independently increases CHD risk potentiates HTN decreases exercise tolerance increases propensity for thrombus MI risk declines 50% after one year of cessation |
|
DM effects on HTN |
hyperglycemia increases production of free radicals --> apoptosis also reduces availability of nitric oxide --> decreased vasodilation --> decreased blood flow |
|
wide S1 split |
RBBB, PVC, VT |
|
shh-dub |
systolic murmur |
|
lub-shh |
diastolic murmur |
|
IJP _____ with deep inspiration |
decreases - inspiration decreases the thoracic pressure and increases blood movement intothe heart (venous return) |
|
causes of rhythm disturbance |
hypoxia ischemia/irritability drugs sympathetic stimulation bradycardia electrolyte distrubance stretch |
|
rales |
pulmonary edema |