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62 Cards in this Set

  • Front
  • Back

really deep Q waves can indicate....

an old heart attack

QT = ?

length of ventricular contraction (systole)




interval should not be too long!





what kinds of meds lengthen QT interval?

psych meds




ondansetron (zofran)

LV hypertrophy implies...

longstanding ventricular HTN

RV hypertrophy implies...

longstanding pulmonary HTN



change in axis =

either electrically dead tissue OR more muscle tissue than expected

in LVH, PMI is displaced...

laterally and inferiorly

pericarditis etiology

usually viral, usuallys elf-limiting

hemopericardium and pericardial effusion both squeeze the heart, so...

LV can't stretch/dilate - backup of blood where it can't fill heart = cardiac tamponade = pressure on heart from fluid in pericardial sack

chordae tendinae get stretched...

when ventricle squeezes, valve backflows = murmur

ionotropic drugs

increase squeeze (contractility_ ex. epinephrine

endocarditis etiology

valves are vulnerable to bacteria that like to attach to leaflets (no pain receptors there, so no pain - mental status changes/fever/fatigue)

IV drug users w/ endocarditis - which valve most often infected?

tricuspid

automaticity

heart can start and maintain itself for a wile if sugar and electrolytes are tehre

rest phase of heart is to prevent _____

tetany




prevents rapid depol and repol past a certain point

beta-blockers slow down _____ NS

sympathetic

sympathetic NS ____ time to threshold for SA node cells

decreases - increases HR overall!

BP =

CO x peripheral resistance

CO =

SV x HR

preload

pressure required to stretch ventricle as far as it can go

afterload

how much pressure does it take to empty the ventricle against the pressure in the periphery

preload is usually ____, afterload is usually ____

fluid


arterial constriction

the greater the size of a lumen (such as in ____), the greater the amount of wall tension/pressure needed to equalize pressure/push out fluid

dilated cardiomyopathy

beta-blockers reduce ______

after-load and slow down HR*

hormone control mechs for BP

ADH, epi/norepi, antiotensin II, ANP


JGA in kidney


Thyroxine

neurologic factors for BP

B1 and B2 adrenergic activity


baroreceptors in carotid sinuses/aortic arch



endothelial factors in BP

nitric oxide


prostacyclin


thromboxane A2

walk balance b/w keeping pt wet enough (?) and dry enough (?)

wet - for kidneys - keep low creatinine


dry - don't drown - preload

PR interval is ____


QRS is ______


QT is ______

PR <1 big box


QRS <3 small boxes


QT <2 big boxes

narrow QRS wave means

signals came from above ventricles (normal contraction)

high circulating badykinin can cause....

edema (ACE-Is)

cold lower extremities concerning for...

oor arterial perfusionp

NSAIDS can reduce the effect of all hypertensive drugs EXCEPT _____

calcium channel blockers

thrombolytic therapy drugs

TPA (converts plasminogen to plasmin, promoting fibrinolysis)




Streptokinase

Must constider _____ in adolescent pt with sudden onset SOB with exertion

hypertrophic ostructive cardiomyopathy (HOCM)

burning chest pain

GERD

tearingchest pain

AAA

sharp chest pain

PE

crushing chest pan

MI

prinzmetal angina

pain NOT dependenty on activity - spasm of coronary arteries




hearlthy women in teh morning

pleuritic chest pain

think PE

A1 receptors

activate = vasoconstrict



A2 receptors

activate = vasodilate

B1 receptors

activate = increase conduction velocity/contractility/HR

B2 receptors

activate = bronchodilation


blcok = bronchoconstriction

history questions for angina

quality, location, timing, onset, precipitating factors, duration, radiation, relief with NT?

NTG can also relieve....

gallbladder pain

UA on EKG/biomarkers

can have ST depression but NO positive biomarkers

you want rate control in an antihypertensive?

NDHP! CCBs




Verapimil and Diltiazem

"typical" MI symptoms may not be seen in...

elderly, females, DM, psych pts

counseling pts about htn

"if we can aggressively treat your risk factors, we can prevent you from having these diseases!"



2 things that can show tracheal deviation?

Pneumothorax and SVC syndrome

Hypertensive emergency is MOSTLY caused by

acute CHF with pulmonary edema




then cerebral infarction/encephalopathy

oxidation of LDL causes

endothelial injury




direct inactiviation of nitric oxide by excess production of free radicals --> reduced nitric oxide causes increase in paltelet adhesion + pro-coagulant effects

effects of tobacco

independently increases CHD risk


potentiates HTN


decreases exercise tolerance


increases propensity for thrombus


MI risk declines 50% after one year of cessation

DM effects on HTN

hyperglycemia increases production of free radicals --> apoptosis




also reduces availability of nitric oxide --> decreased vasodilation --> decreased blood flow

wide S1 split

RBBB, PVC, VT

shh-dub

systolic murmur

lub-shh

diastolic murmur

IJP _____ with deep inspiration

decreases - inspiration decreases the thoracic pressure and increases blood movement intothe heart (venous return)

causes of rhythm disturbance

hypoxia


ischemia/irritability


drugs


sympathetic stimulation


bradycardia


electrolyte distrubance


stretch

rales

pulmonary edema