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24 Cards in this Set

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  • Back
What pharynx muscle is obstructive while sleeping in OSA (obstructive sleep apnea)
-Velopharynx
-Oropharynx
-Hypopharynx
-Occur when pharyngeal dilator muscle can't keep airwway open when sleeping
What type of Polysomnograph is used to DX OSA?
-EEG (ElectroENCEphalography)
-EOG (Electro-OCUlography)
-Nasal & Roal Thermistors
-ElectroMYOgraphy (EMG)
What is the purpose of Pulse Oximetry?
-Measure Hg saturation w/ O2 dinstinguish changes in blood O2 sutation during an apneic or hypopneic event
What is the differences Apnea and Hypopnea?
-Apnea: Complete Obstruction for > 10s
-Hypopnea: Partial obstruction for > 10s fulfilling other criteria
Describe the severity of Obstructive Sleep Disordered Breathing
AHI = # Apneas + # hypopneas/ hours of sleep
-Normal (<5)
-Mild (5-15)
-Moderate (15-30)
-Severe (>30)
What is OSAS (Syndrome)
-Clinical symptoms of EX daytime sleepiness, hypersomnolence or cogntive impariment
Describe OSA vs. Hypopnea vs. UARS
-OSA & Hypopnea: Decrease Airflow, O2 Desaturation, Increase Respiratory Effort, EEG Arousal, AHI
-UARS: Increase Respiratory Effort, EEG Arousal. No change in Air flow, O2 Desaturation, AHI
Why UARS difficult to DX?
-No changes in Air flow, O2 Desaturation, AHI
-Require Effort (PES) to DX
Natural course of OSA
-Incrase with age, weight, alcohol, sedatives, physical exhausation in the evening and at bedtime, smoking.
Describe UARS
Young Adult females, normal or below wieght, profile w/ anxiety/distress, XS day time sleepiness/snoring, normal pharygenal mucosal mechanosensitivity,
-Co-morbid w/ functional somatic disorders, partically fibromyalgia
OSA can decrease the average life span of the general population by 20 years (T/F)
True
Significane of a __ mm Hg drop in mean artial blood pressure: means what
-10 mm Hg
-37% reduction in risk of coronary heart disease
-56% reduction in risk of stroke
What is the GOLD standard TX:
Positive Airwya PRessure (CPAP, BiPAP)
What are the types of PAP?
-Manual CPAP Mode
-Bilevel Mode
-Auto CPAP Mode
Indications for TOngue Retaining Devices?
-No teeth or insufficient tooth support
-Patients w/ TMJ dysfunction/pain or who can't tolerate jaw advancement or who can't protrude the jaw
Indications for Tongue Restraining Devices (FBS)?
1. Patients with TMJ dysfunction/pain or who can't tolerate jaw advancement or who can't protrude the jaw
2. To minimize risk of long-term changes in the bite or tooth position
3. Failure of, or in place of therapy with MAS
Indications for Combination of Oral Appliance & PAP?
-Typically reserved for most severe cases of OSA. Enables reduction of PAP pressure
-Oral vs. nasal delivery of PAP (OPAP)
-Reduce PAP "claustrophobia (eliminate head and chin straps)
Mechanism of Action of Mandibular Advancement Splints?
-Pull the tongue and its base forward increasing the ML dimension of oropharynx
-Place tension across the soft palate via palatoglossus muslces, moving it forward, making it broader & more stable in position; increasing the AP and ML dimension of velopharynx
-Increase activity and reflex response of pharyngeal dilator muslces
-Reduce collapsibility of airway
-Maintain jaw in more close position = less posterior movement
The Efficiency of MAS therapy may equal or exceed that of PAP therapy due to much better patient compliance w/ MAS therapy (T/F)
-True
What is Complex Sleep apnea?
-Obstructive and Central Apnea
-Untreated pt often exhibit OSA but upon TX of OSA, CSA emerges
3 methods to Monitor Tooth Movement During the Course of Treatment?
-Measurements from digital images of teeth
-Cephalographic evaluation (Orthodontists Standard)
-Intraoral Photograph
3 Screening Tools in Dental Clinic?
-STOP (Snore, Tired, Observed stop breathing, Pressure BP)
-Adjusted Neck Size
-STOP-Bang
Muscle tone Inversely related to Upper Airway Obstruction (T/F)
True
Problem: Airway Obstruction at Base of Tongue
Body's Solution: Brux to activate the tongue to bring forward