Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
60 Cards in this Set
- Front
- Back
What is the Carotid Sheath made of?
|
3 structures
1. Internal Jugular vein(lateral) 2. Common carotid artery(middle) 3. Vagus nerve (posterior) |
|
In the majority of cases what supplies the SA and AV node is supplied by...
|
RCA (80% of the time)
RCA supplies the inferior part of the left ventricle by the PD artery(rt dominant) |
|
What abt 20% of the time, what supplies the SA and AV node?
|
the PD arises from the CFX
|
|
Where does coronary artery occlusion most commonly occur?
|
LAD
which supplies the anterior interventricular septum |
|
When do the coronary arteries fill?
|
Diastole
|
|
What is the most posterior part of the heart?
|
Left atrium
|
|
What does the enlargement of the left atrium cause?
|
Dysphagia
due to compression of the esophageal nerve or hoarseness due to compression of therecurrent laryngeal nerve, a branch of the vagus |
|
What are the two branches of the root of the atria>
|
Left main and right main
|
|
What does the left main branch of into
|
LAD and the Circumflex
|
|
What does the LAD supply?
|
2/3 of the intervent
ant wall of the LV ant papillary muscle |
|
What does the circumflex supply?
|
lat posterior wall of the LV
(10% of the time its PDA) |
|
What does the Right main divide into?
|
SA and AV node arteries
PDA(85-90% of the time) |
|
What does the PDA supply?
|
inferior wall of the LV, diaphragmatic part of the heart
|
|
What is CO?
|
the amt of blood pumped out of the heart per minute
|
|
How do you calculate CO?
|
CO- SV x HR
|
|
CO=
|
rate of consumption/arterial O2-venous O2 content
|
|
During exercise, CO increases initially bec...
|
increased SV
|
|
After prolonged exercise, CO will remain increased bc of
|
increased HR
|
|
MAP
|
CO x TPR
|
|
IF HR is too high, diastolic filling is...
|
incomplete and the CO is decreased
(ex-ventric tachy) |
|
MAP
|
2/3 diastolic P + 1/3 systolic P
|
|
Pulse Pressure=
|
systolic P- diastolic P
|
|
Pulse P is proportional to
|
SV
|
|
SV=
|
CO/HR
|
|
SV=
|
EDV-ESV
|
|
What is stroke volume affected by?
|
contractility,afterload, and preload
|
|
When does SV increase?
|
increased preload
decreased afterload or decreased contractility |
|
What 4 circumstances increase contractility(and SV)?
|
1. catecholamines(increased activity of Ca2+ pump in sarcoplasmic reticulm)
2. increased intracellular calcium 3. decreased extracellular sodium (decreased activity of Na+/Ca2+ exchanger) 4. Digitalis(increased intracellular Na+ resulting in increased Ca2+) |
|
What does increased sodium do?
|
stops the Na+ Ca2+ exhange
|
|
Contractility( and SV) decreased with...
|
1. B1 blockade
2. Heart failure 3.acidosis 4. hypoxia/hypercapnea 5.nondihydropyridine Ca2+ channel blockers |
|
What causes SV to increase?
|
anxiety
exercise pregnancy |
|
What causes SV to decrease?
|
a failing heart
|
|
myocardial oxygen demand is increased by...
|
1. increased afterload(analalog to arterial P)
2.increase contractility 3.increased HR 4.increased Heart size(increased wall tension) |
|
Preload=
|
ventricular EDV
|
|
Preload increases with
|
1. exercise(slightly)
2.increased blood vol(overtransfusion) 3.excitement(sympathetics) |
|
what does Preload do to the heart?
|
It pumps up the heart
|
|
Afterload-
|
mean arterial pressure(proportional to peripheral resistance)
|
|
venodilators=
|
ex- Nitroglycerin
decreases Preload |
|
vAsodilators=
|
ex- hydrAlazine
decreases Afterload (Arterial) |
|
Starling curve
|
force of contraction is proportional to initial length of cardiac muscle fiber(preload)
|
|
What increases contractile state of myocardium?
|
1 circulating catecholamines
2. digitalis 3. sympathetic stimulation |
|
What decreases the contractile state of myocardium?
|
1.pharmacologic depressants
2.loss of myocardium |
|
change in pressure=
|
Q x R
similar to Ohm's law change in V=IR |
|
What is isovolumetric contraction?
|
period btwn mitral valve closing and the aortic valve opening.
**highest O2 consumption** |
|
What is systolic ejection?
|
period btwn aortic valve opening and closing
|
|
What is isovolumetric relaxation?
|
period btwn aortic valve closing and mitral valve opening
|
|
What is rapid filling?
|
period just after mitral valve opening
|
|
what is reduced filling?
|
period just before mitral valve closure
|
|
what is the S1?
|
when the mitral and tricuspid valve close.
loudest at the mitral area |
|
Whats is the S2?
|
aortic and pulmonic valve closure?
loudest at the left sternal border. |
|
What is S3?
|
in early diastole, during rapid ventricular filling phase
assoc with increased filling pressures and more common in dilated ventricles but normal in kids and preggos |
|
What is S4?
|
atrial kick
in late diastole high atrial P assoc with ventric hypertrophy left atrium must push bs stiff LV wall |
|
what is the a wave
|
atrial contraction
|
|
what is the C wave?
|
RV contraction
(tricuspid valve bulging into the atrium) |
|
v wave
|
increased atrial P due to filling against closed tricuspid valve
|
|
what is S2 splitting?
|
aortic valve closes before pulmonic,
inspiration increases this difference |
|
what is wide splitting?
|
assoc with pulmonic stenosis or rt bundle branch block2
|
|
what is fixed splitting?
|
assoc with ASD
|
|
what is paradoxical splitting?
|
assoc with aortic stenosis or left bundle branch block
|
|
what is normal splitting lungs
a |
inspiration leads to a drop in intrathoracic pressire which increases capacity of pulm circulation
pulm valve closes to later accomodate more blood entering lungs. Aortic valve |