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17 Cards in this Set

  • Front
  • Back

dysphagia

sensation of food being hindered in passage from mouth to stomach



oropharyngeal; stoke, enlarged thyroid, skeletal muscle disorder, decreased saliva



esophageal; obstruction, motility disorder (both solids and liquids)

3 zones of esophagus

upper esophageal sphincter


body


lower esophageal sphincter



between swallows tone in UES and LES protect against reflux

Achalasia

failure of complete LES relaxation with swallows, hypertensive LES



aperistalsis of smooth muscle of esophagous = none, zilch, zero peristaltic propagations

Type 1 achlasia

high res vs regular, high res has more sensors so better pictures and straight line measure the contractions all the way down



in normal we expect a smooth change in pressure



no change in the pressure in the LES,



ALL have aperistalsis and no LES opening

achalasia clinical symptoms

dysphagis for solids in all and liquids in 2.3


chest pain


weight loss


regurg


increased risk for squamous cell carcinoma

Achalasia in X-Ray

movement image



dilated esophagus



bird beack distal esophagus due to hypertensive poorly relaxing LES



chest x ray of gastric bubble

Endoscopy of LES

dilated esophagous with poor motility, retention of food



puckered closed LES



esophagitis due to retained food, candida



can pass a scope thru

achalasia LES

can't do much with the aperistalsis, but can relax he muscles



esophagomyotomy



pneumatic dilatation



botulinum toxin into LES, interfers with aceytlcholine release



drugs to relace smooth muscle, nitrates, cqclium channel blockers

Pseudoachalasia

usually caused by cancer, does not present with other symptoms or causes of normal achalasia



need to always check to make sure its not a tumor

Scleroderma

hypotensive LES



atrophy of the muscle and replacement with fibrous tissue



neural dysfunction may proceed muscle disease



manometry; esophageal body dysfunction, weak LES



clinical featuresl esophagitis from reflux, dysphagia due to dysmotility

difuse esophageal spasm

simultaneous contractions some nl peristlasis



spontaneous repetitive contractioncs

nutcracker eso

high amplitude conractions

GERD

any sympotmatic condition or histopatholigic alteration resulting from GER

Factors that maintain the GE junction

intrinsic LES pressure



extrinsic pressure via diaphragm



intraabdominal location of LES (hiatal hernia)



phrenoesophagela ligament



acute angle of his

pathogenesis of GERD

transient LES relaxation



hypotensive LES, LES affect by hormones, drugs, and food, progesteron decreases LES, LES decerased by beta blockers



LES decreased by fat chocolate alcohol peppermint



only a small numer

Consequences of GERD

heartburn, without endoscopic esophagitis



esophagitis



dysphagia, peptic stricture and a schatzkis ring about a hiatal hernia, peristaltic dysfunction and mucosal inflammation



barretts

Impedance

correlated to the number of ions and if the bolus is present