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109 Cards in this Set
- Front
- Back
- 3rd side (hint)
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CHARACTERISTIC OF HGB H
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BETA 4
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HGB H DSS IS A SEVERE CLINICAL EXPRESSION OF
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ALPHA THALASSEMIA IN WHICH ONLY ONE ALPHA GENE OUT OF FOUR IS FUNCTIONING
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THE ANEMIA SEEN IN SICKLE CELL DSS IS CHARACTERIZED BY
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NORMOCYTIC, NORMOCHROMIC
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WHICH IS THE MAJOR HGB FOUND IN THE RBCS OF PT WITH SICKLE CELL TRAIT
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HGB A
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PASSIVE IMMUNITY IS WHERE
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AB ARE TRANSFERRED. TEMPORARY NO MEMORY
GAMMA GLOBULIN |
PASSIVE-PASS
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CRITERIA FOR DONATING BLOOD
TEMP HCT |
TEMP 99.5 OR 37.5 C
HCT 38% |
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DEFERRAL TIME FOR SOMEONE WHO HAS RECIEVED BLOOD TRANSFUSION PRODUCTS
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1 YEAR
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DEFERRAL FOR SOMEONE WHO HAS HAD EXPOSURE TO HEPATITIS, HIV, MALARIA
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1 YEAR
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PASSIVE IMMUNITY IS ASSOCIATED WITH WHAT
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IMMUNIZATIONS
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ACTIVE IMMUNITY IS WHERE
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AN INDIVIDUAL PRODUCES THE AB, FOLLOWS INFECTION
LASTING |
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DEFERRAL TIME FOR POS SYPHILIS
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1 YR AFTER COMPLETION OF THERAPY
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DEFERRAL FOR POS HEP AFTER 11 YEARS OLD
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INDEFINITE
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POS CONFIRMATORY FOR HBSAG IS DEFERRED FOR....
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INDEFININTE
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POS FOR ANI-HBC
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INDEFINITE
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PRESENT, PAST INFECTION OF HEP C, HIV, HTLV1/11
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INDEFINITE
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HISTORY OF CHAGAS DSS, BABESIOSIS
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INDEFINITE
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FORMULA TO DETERMINE COAGULANT WHEN DRAWING BLOOD.
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PT WT 1BS
---------- X 450 ML X 14% 110 1BS |
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AMOUNT OF ANTICOAGULANT IN BLOOD DONOR BAG
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63 ML
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CPDA-1 EXPIRATION
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35 DAYS
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CPDA-1 MAKEUP
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ADENINE, PHOSPHATE
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ADDITIVE SOLUTIONS
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ADSOL, NUTRICEL, OPTISOL
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ADSOL,NUTRICEL,OPTISOL EXP DATE
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42 DAYS ADDED W/N 72 HRS OF PHLEBOTOMY
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REJUVENATING SOLUTIONS
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PYRUVATE,PHOSPHATE,INOSINE,ADENINE
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WHEN ARE REJUVENATING SOL. ADDED
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DURING STORAGE OR UP TO 3 DAYS AFTER EXP.
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AUTOLOGOUS DONATION AGE,HGB,HCT CRITERIA
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AGE:NONE
HGB:11 hct:33 |
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donating blood for oneself is called a ____ unit
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autologous
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THIS IS A COMPONENT THAT IS GIVEN WHEN A PT IS IN SEVERE SHOCK AND BLD LOSS > 25% BLD VOL.
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WHOLE BLOOD
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PACKED RBCS PROVIDE
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O2 BUT LESS VOLUME THAN WHOLE BLOOD
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HCT LEVEL THAT INDICATES SUFFICIENT PLASMA REMOVAL
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80%
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1 UNIT OF BLOOD RAISES THE HGB AND HCT...
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HBG 1 G
HCT 3% |
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THIS COMPONENT PREVENTS ALLERGIC RESPONSE TO PLASMA PROTEIN AND ANAPHYLACTIC SHOCK IN IGA DEFICIENT PT C ANTI-IGA
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WASHED RBC
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CYTOMEGALO VIRUS AB IS FOUND
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TYPE OF HERPES FOUND IN ALL BODY SECRETIONS
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INTERNATIONAL NORMALIZED RATIO FORMULA IS
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PT RESULT
----------------- MEAN OF REF RANGE |
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COAGULATION SAMPLES SHOULD NOT BE DRAWN FIRST WHEN USING VACUTAINERS B/C
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CONTAMINATION OF TSS THROMBOPLASTIN WHICH ACTIVATES COAGULATION AS NEEDLE PIERCES SKIN
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PROTHROMBIN TIME MEASURES
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EXTRINSIC AND COMMON PWAY
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PT MONITORS
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WARFARIN
COUMARIN DICOUMAROL |
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FIRST CARDIAC MARKERS TO EVALUATE POSSIBLE AMI
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CK-MB FOLLOWED BY CK-MM
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CK-MB AND CK-MM INCREASE WITHIN--- AND RETURN TO NORMAL
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INCREASE IN 24 HRS
RETURN TO NML 2-3 DAYS LATER |
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PRODUCED BY MUSCLES. INCREASES IN MUSCLE DMG AND AMI, RISES IN 30 MIN
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MYOGLOBIN
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CARDIAC MARKERS THAT ELEVATE WITH AMI
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CK-MB
CK-MM AST LD |
REMEMBER MICAL HAD AN MI
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AST RISES AND RETURNS TO NML IN
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AST RISES IN 24-48 HRS
NML 4-6 DAYS |
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LD RISES AND RETURNS TO NML IN
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LD RISES IN 48-72 HRS
NML 7-12 DAYS LATER |
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TROPONIN IS USED FOR
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DETECTION OF AMI
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DIFFERENCE BTWN CK-MB AND TROPONIN
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TROPONIN STAYS ELEVATED LONGER THAN CK-MB
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DETECTS SMALL AMT OF PROTEIN IN UA OF DIABETIC PT TO ASSESS RENAL DMG
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MICROALBUMIN
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APTT STANDS FOR
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ACTIVATED PARTIAL THROMBOPLASTIN TIME
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APTT MEASURES AND MONITORS
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HEPARIN
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2,3 DIPHOSPHOGLYCERATE IS
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ORGANIC PHOSPHATE IN RBCS THAT ALTERS THEIR AFFINITY FOR O2
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ELEVATED LEVELS OF 2,3 DPG ARE FOUND IN THESE DSS AND CONDITIONS
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HYPOXIA, UREMIA, ANEMIA, THYROTOXICOSIS, PYRUVATE KINASE DEFICIENCY
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INCREASE IN 2,3 DPG
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DECREASES O2 BINDING CAPACITY OF HGB SO THAT THE INCREASED AMT OF 02 ARE RELEASED AND AVAILABLE TO TSS.
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DECREASE LEVELS OF 2,3 DPG ARE FOUND IN
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ALKALOSIS, POLYCTHEMIA, HEXOKINASE DEFICIENCY, RESPIRATORY DISTRESS SYNDROME
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KB TEST INDICATES
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MODERATE TO GREAT FETOMATERNAL HEMORRHAGE
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KB TEST IS PERFORMED
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AFTER FULL TERM DELIVERY, IF NEWBORN ANEMIA IS PRESENT WHEN MOTHER IS RH NEG AND AFTER INVASIVE PROCEDURE SUCH AS AMNIOCENTESIS
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KB TEST DETERMINES
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AMT OF FETAL HEMORRHAGING IN AN RH NEG MOTHER AND THE AMT OF RHOGHAM NECESSARY TO PREVENT AB PRODUCTION
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LYMES DSS IS TRANSMITTED BY
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DEER TICK BITE
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S/S OF LYME DSS
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RASH, FLU-LIKE S/S AFTER 1 WK FROM BITE
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HOW TO DETECT LYMES DSS
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IGM AB DETECTED BY ELIZA IN EARLY STAGES.
WESTERN BLOT IN LATER STAGES TSS BIOPSY CSF EXAM DNA ID |
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LYME DSS FALSE POSITIVES
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INCREASED RHEUMATOID FACTOR TITERS
TREPONEMAL DSS ANIBIOTICS INCREASED LIPID |
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LYMBE DSS IS CAUSED BY
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BORRELIA BURGDORFERI
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ACETYLCHOLINE RECEPTOR BINDING AB IS DX FOR
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MYASTHERIA GRAVIS
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WHAT IS MYASTHERIA GRAVIS
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NEUROLOGICAL DEGENERATIVE DSS THAT DESTROYS ACETYLCHOLINE RECEPTORS BOUND BY ANTIBODIES AT THE MUSCLE END PLATE
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S/S OF MYASTHERIA GRAVIS
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FATIGUE, MUSCLE WEAKNESS
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MPV MEAN PLATELET VOLUME NML RANGES
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8.6-11.7 FL
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MEAN PLATELET VOLUME INDICATES
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PT UNIFORMITY OF PLATELET DISTRIBUTION.
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CLINICAL IMPLICATIONS OF MYOGLOBIN
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MUSCLE ENZYME DEFICIENCY,INJURY DYSTROPHY
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MYOBLOBIN IN RELATION TO MI
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DX MI IF BTWN 2-12 HRS AFTER, WILL NOT INCR UNTIL AFTER 2 HRS AND RETURN AFTER 12HRS
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INCREASED LEVELS OF MYOGLOBIN SUGGEST
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RENAL FAILURE, MALIGNANT HYPERTHERMIA, SEVERE BURNS, LE, SEIZURES, TRAUMA
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MYOGLOBIN NML RANGES FOR UA AND BLOOD
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UA 0-2
BLD <70 NG |
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ACANTHOCYTES ARE SEEN IN
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ABETALIPOPROTEINEMIA, SEVERE LIVER DSS
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ESR WESTERGREN IS A TEST WHICH MEASURES
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THE FALL RATE OF RBC THROUGH PLASMA
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BURR CELLS SEEN IN
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uremia, artifact, alkaline glass effect
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another name for burr cells
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echinocyte
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elliptocyte seen in
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hereditary elliptocytosis
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macroovalacyte seen in
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megaloblastic anemia
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keratocyte (helmet) cell seen in
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hemolytic process
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rbc fragments aka
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schistocyte
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schistocytes are seen in
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dic and hemolytic process
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sickle cell aka
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depranocyte
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depranocytes seen in
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hbss
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spherocytes seen in
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hereditary spherocytosis, abo hdn
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mchc in spherocytes
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increased
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stomatocytes (mouth cell) seen in
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hereditary stomatocytosis and liver dss
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target cell aka
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codocyte
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target cells seen in
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liver dss
hb c hgbopathies |
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tear drop cell aka
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dacrocyte
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tear drop cells seen in
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extramedullary hematopoiesis
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diseases caused by hemochromatosis
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diabetes mellitus, irr heart beat, heart attack, arthritis, cirrhosis, liver cancer, infertility, hypothyroidism
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thalassemia aka
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mediterranean anemia
von jaksch anemia cooleys anemia |
named after physicians who dx it.
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hemochromatosis is
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inherited condition of abn fe metabolism. absorb too much fe from diet and cannot be excreted
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fe overload can cause
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cirrhosis, liver cancer, osteoarthritis, oseopenia, diabetes, gallstones, heart attack, hypothyroidsm, depression, infertility
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idiopathic sideroblatic anemia referred to as ... b/c
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myelodyplasia syndrom (mds)
unknown cause |
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acquired sideroblastic anemia dss causes
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rheumatoid arthritis
leukemia lymphoma uremia hyperthyroidism |
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acquired sideroblastic anemia is due to
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prolonged exposure to toxins like alcohol, pb, drugs, decreased folic acid, decr cu and zn
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3 types of sideroblastic anemia
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hereditary, acquired, idiopathic
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sideroblastic anemia is an enzyme disorder in which
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the body has adequate fe but is not able to incorporate it into hgb.
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nml range of hgb for female
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14 +/- 2 g/dL
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nml range for hct for female
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42 +/- 5%
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rbc nml range for females
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4.5 +/- 0.7 x 10 6th/uL
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wbc nml range for females
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5000-10,000/Ul
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nml range for platelet in females
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150k-400k/uL
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nml range for hgb in males
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16 +/- 2 g/dL
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nml range for hct in males
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47 +/- 5%
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rbc range for males
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5.3 +/- 0.8 X 106th
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wbc range for males
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5k-10k
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same as females
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platelet range for males
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150k-400k
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same as females
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sideroblastic anemias are usually assoc. with what cell type
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microcytic, hypochromic
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If you have an increased mcv and mchc and a decreased rbc count what is this assoc with
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cold agglut. warm are rerun
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if the rule of 3 doesn't fit consider...
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clotted sample, cold agglutinin, lipemic or icteric sample
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rule of 3 for rbc
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hgb +/- 0.5
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