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109 Cards in this Set

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CHARACTERISTIC OF HGB H
BETA 4
HGB H DSS IS A SEVERE CLINICAL EXPRESSION OF
ALPHA THALASSEMIA IN WHICH ONLY ONE ALPHA GENE OUT OF FOUR IS FUNCTIONING
THE ANEMIA SEEN IN SICKLE CELL DSS IS CHARACTERIZED BY
NORMOCYTIC, NORMOCHROMIC
WHICH IS THE MAJOR HGB FOUND IN THE RBCS OF PT WITH SICKLE CELL TRAIT
HGB A
PASSIVE IMMUNITY IS WHERE
AB ARE TRANSFERRED. TEMPORARY NO MEMORY
GAMMA GLOBULIN
PASSIVE-PASS
CRITERIA FOR DONATING BLOOD
TEMP
HCT
TEMP 99.5 OR 37.5 C
HCT 38%
DEFERRAL TIME FOR SOMEONE WHO HAS RECIEVED BLOOD TRANSFUSION PRODUCTS
1 YEAR
DEFERRAL FOR SOMEONE WHO HAS HAD EXPOSURE TO HEPATITIS, HIV, MALARIA
1 YEAR
PASSIVE IMMUNITY IS ASSOCIATED WITH WHAT
IMMUNIZATIONS
ACTIVE IMMUNITY IS WHERE
AN INDIVIDUAL PRODUCES THE AB, FOLLOWS INFECTION
LASTING
DEFERRAL TIME FOR POS SYPHILIS
1 YR AFTER COMPLETION OF THERAPY
DEFERRAL FOR POS HEP AFTER 11 YEARS OLD
INDEFINITE
POS CONFIRMATORY FOR HBSAG IS DEFERRED FOR....
INDEFININTE
POS FOR ANI-HBC
INDEFINITE
PRESENT, PAST INFECTION OF HEP C, HIV, HTLV1/11
INDEFINITE
HISTORY OF CHAGAS DSS, BABESIOSIS
INDEFINITE
FORMULA TO DETERMINE COAGULANT WHEN DRAWING BLOOD.
PT WT 1BS
---------- X 450 ML X 14%
110 1BS
AMOUNT OF ANTICOAGULANT IN BLOOD DONOR BAG
63 ML
CPDA-1 EXPIRATION
35 DAYS
CPDA-1 MAKEUP
ADENINE, PHOSPHATE
ADDITIVE SOLUTIONS
ADSOL, NUTRICEL, OPTISOL
ADSOL,NUTRICEL,OPTISOL EXP DATE
42 DAYS ADDED W/N 72 HRS OF PHLEBOTOMY
REJUVENATING SOLUTIONS
PYRUVATE,PHOSPHATE,INOSINE,ADENINE
WHEN ARE REJUVENATING SOL. ADDED
DURING STORAGE OR UP TO 3 DAYS AFTER EXP.
AUTOLOGOUS DONATION AGE,HGB,HCT CRITERIA
AGE:NONE
HGB:11
hct:33

donating blood for oneself is called a ____ unit
autologous
THIS IS A COMPONENT THAT IS GIVEN WHEN A PT IS IN SEVERE SHOCK AND BLD LOSS > 25% BLD VOL.
WHOLE BLOOD
PACKED RBCS PROVIDE
O2 BUT LESS VOLUME THAN WHOLE BLOOD
HCT LEVEL THAT INDICATES SUFFICIENT PLASMA REMOVAL
80%
1 UNIT OF BLOOD RAISES THE HGB AND HCT...
HBG 1 G
HCT 3%
THIS COMPONENT PREVENTS ALLERGIC RESPONSE TO PLASMA PROTEIN AND ANAPHYLACTIC SHOCK IN IGA DEFICIENT PT C ANTI-IGA
WASHED RBC
CYTOMEGALO VIRUS AB IS FOUND
TYPE OF HERPES FOUND IN ALL BODY SECRETIONS
INTERNATIONAL NORMALIZED RATIO FORMULA IS
PT RESULT
-----------------
MEAN OF REF RANGE
COAGULATION SAMPLES SHOULD NOT BE DRAWN FIRST WHEN USING VACUTAINERS B/C
CONTAMINATION OF TSS THROMBOPLASTIN WHICH ACTIVATES COAGULATION AS NEEDLE PIERCES SKIN
PROTHROMBIN TIME MEASURES
EXTRINSIC AND COMMON PWAY
PT MONITORS
WARFARIN
COUMARIN
DICOUMAROL
FIRST CARDIAC MARKERS TO EVALUATE POSSIBLE AMI
CK-MB FOLLOWED BY CK-MM
CK-MB AND CK-MM INCREASE WITHIN--- AND RETURN TO NORMAL
INCREASE IN 24 HRS
RETURN TO NML 2-3 DAYS LATER
PRODUCED BY MUSCLES. INCREASES IN MUSCLE DMG AND AMI, RISES IN 30 MIN
MYOGLOBIN
CARDIAC MARKERS THAT ELEVATE WITH AMI
CK-MB
CK-MM
AST
LD
REMEMBER MICAL HAD AN MI
AST RISES AND RETURNS TO NML IN
AST RISES IN 24-48 HRS
NML 4-6 DAYS
LD RISES AND RETURNS TO NML IN
LD RISES IN 48-72 HRS
NML 7-12 DAYS LATER
TROPONIN IS USED FOR
DETECTION OF AMI
DIFFERENCE BTWN CK-MB AND TROPONIN
TROPONIN STAYS ELEVATED LONGER THAN CK-MB
DETECTS SMALL AMT OF PROTEIN IN UA OF DIABETIC PT TO ASSESS RENAL DMG
MICROALBUMIN
APTT STANDS FOR
ACTIVATED PARTIAL THROMBOPLASTIN TIME
APTT MEASURES AND MONITORS
HEPARIN
2,3 DIPHOSPHOGLYCERATE IS
ORGANIC PHOSPHATE IN RBCS THAT ALTERS THEIR AFFINITY FOR O2
ELEVATED LEVELS OF 2,3 DPG ARE FOUND IN THESE DSS AND CONDITIONS
HYPOXIA, UREMIA, ANEMIA, THYROTOXICOSIS, PYRUVATE KINASE DEFICIENCY
INCREASE IN 2,3 DPG
DECREASES O2 BINDING CAPACITY OF HGB SO THAT THE INCREASED AMT OF 02 ARE RELEASED AND AVAILABLE TO TSS.
DECREASE LEVELS OF 2,3 DPG ARE FOUND IN
ALKALOSIS, POLYCTHEMIA, HEXOKINASE DEFICIENCY, RESPIRATORY DISTRESS SYNDROME
KB TEST INDICATES
MODERATE TO GREAT FETOMATERNAL HEMORRHAGE
KB TEST IS PERFORMED
AFTER FULL TERM DELIVERY, IF NEWBORN ANEMIA IS PRESENT WHEN MOTHER IS RH NEG AND AFTER INVASIVE PROCEDURE SUCH AS AMNIOCENTESIS
KB TEST DETERMINES
AMT OF FETAL HEMORRHAGING IN AN RH NEG MOTHER AND THE AMT OF RHOGHAM NECESSARY TO PREVENT AB PRODUCTION
LYMES DSS IS TRANSMITTED BY
DEER TICK BITE
S/S OF LYME DSS
RASH, FLU-LIKE S/S AFTER 1 WK FROM BITE
HOW TO DETECT LYMES DSS
IGM AB DETECTED BY ELIZA IN EARLY STAGES.
WESTERN BLOT IN LATER STAGES
TSS BIOPSY
CSF EXAM
DNA ID
LYME DSS FALSE POSITIVES
INCREASED RHEUMATOID FACTOR TITERS
TREPONEMAL DSS
ANIBIOTICS
INCREASED LIPID
LYMBE DSS IS CAUSED BY
BORRELIA BURGDORFERI
ACETYLCHOLINE RECEPTOR BINDING AB IS DX FOR
MYASTHERIA GRAVIS
WHAT IS MYASTHERIA GRAVIS
NEUROLOGICAL DEGENERATIVE DSS THAT DESTROYS ACETYLCHOLINE RECEPTORS BOUND BY ANTIBODIES AT THE MUSCLE END PLATE
S/S OF MYASTHERIA GRAVIS
FATIGUE, MUSCLE WEAKNESS
MPV MEAN PLATELET VOLUME NML RANGES
8.6-11.7 FL
MEAN PLATELET VOLUME INDICATES
PT UNIFORMITY OF PLATELET DISTRIBUTION.
CLINICAL IMPLICATIONS OF MYOGLOBIN
MUSCLE ENZYME DEFICIENCY,INJURY DYSTROPHY
MYOBLOBIN IN RELATION TO MI
DX MI IF BTWN 2-12 HRS AFTER, WILL NOT INCR UNTIL AFTER 2 HRS AND RETURN AFTER 12HRS
INCREASED LEVELS OF MYOGLOBIN SUGGEST
RENAL FAILURE, MALIGNANT HYPERTHERMIA, SEVERE BURNS, LE, SEIZURES, TRAUMA
MYOGLOBIN NML RANGES FOR UA AND BLOOD
UA 0-2
BLD <70 NG
ACANTHOCYTES ARE SEEN IN
ABETALIPOPROTEINEMIA, SEVERE LIVER DSS
ESR WESTERGREN IS A TEST WHICH MEASURES
THE FALL RATE OF RBC THROUGH PLASMA
BURR CELLS SEEN IN
uremia, artifact, alkaline glass effect
another name for burr cells
echinocyte
elliptocyte seen in
hereditary elliptocytosis
macroovalacyte seen in
megaloblastic anemia
keratocyte (helmet) cell seen in
hemolytic process
rbc fragments aka
schistocyte
schistocytes are seen in
dic and hemolytic process
sickle cell aka
depranocyte
depranocytes seen in
hbss
spherocytes seen in
hereditary spherocytosis, abo hdn
mchc in spherocytes
increased
stomatocytes (mouth cell) seen in
hereditary stomatocytosis and liver dss
target cell aka
codocyte
target cells seen in
liver dss
hb c
hgbopathies
tear drop cell aka
dacrocyte
tear drop cells seen in
extramedullary hematopoiesis
diseases caused by hemochromatosis
diabetes mellitus, irr heart beat, heart attack, arthritis, cirrhosis, liver cancer, infertility, hypothyroidism
thalassemia aka
mediterranean anemia
von jaksch anemia
cooleys anemia
named after physicians who dx it.
hemochromatosis is
inherited condition of abn fe metabolism. absorb too much fe from diet and cannot be excreted
fe overload can cause
cirrhosis, liver cancer, osteoarthritis, oseopenia, diabetes, gallstones, heart attack, hypothyroidsm, depression, infertility
idiopathic sideroblatic anemia referred to as ... b/c
myelodyplasia syndrom (mds)
unknown cause
acquired sideroblastic anemia dss causes
rheumatoid arthritis
leukemia
lymphoma
uremia
hyperthyroidism
acquired sideroblastic anemia is due to
prolonged exposure to toxins like alcohol, pb, drugs, decreased folic acid, decr cu and zn
3 types of sideroblastic anemia
hereditary, acquired, idiopathic
sideroblastic anemia is an enzyme disorder in which
the body has adequate fe but is not able to incorporate it into hgb.
nml range of hgb for female
14 +/- 2 g/dL
nml range for hct for female
42 +/- 5%
rbc nml range for females
4.5 +/- 0.7 x 10 6th/uL
wbc nml range for females
5000-10,000/Ul
nml range for platelet in females
150k-400k/uL
nml range for hgb in males
16 +/- 2 g/dL
nml range for hct in males
47 +/- 5%
rbc range for males
5.3 +/- 0.8 X 106th
wbc range for males
5k-10k
same as females
platelet range for males
150k-400k
same as females
sideroblastic anemias are usually assoc. with what cell type
microcytic, hypochromic
If you have an increased mcv and mchc and a decreased rbc count what is this assoc with
cold agglut. warm are rerun
if the rule of 3 doesn't fit consider...
clotted sample, cold agglutinin, lipemic or icteric sample
rule of 3 for rbc
hgb +/- 0.5