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101 Cards in this Set

  • Front
  • Back
Axis I - V
I Clinical
II Personality/MR
III Medical
IV Psychosocial/Environmental
V GAF
MR Criteria
IQ less than or equal to 70
Impaired adaptive functioning
Onset before 18
Degrees of MR
Mild: 50-55 to 70
Moderate: 34-40 to 50-55
Severe: 20-25 to 35-40
Profound: < 20-25

As adults, individuals with Moderate MR can contribute to their own support by performing unskilled or semi-skilled tasks under supervision -- in sheltered workshops or in the competitive job market. They constitute 10% of the mentally retarded population, they are unlikely to progress beyond the second grade level in academic skills, and they can talk or learn to communicate during the preschool years.
Etiology
5% - Heredity
30% - Early alt in embroyonic dev
10% - Pregancy and perinatal problems
5% - General med problems
15-20% - Environmental factors
30-40% Unknown
PKU
Inability to metabolize PKU, which causes MR if untreated/improper diet.
DS/Trisomy 21
5th finger
Large protruding tongue
Heart lesions
Respirator and intestinal defects
Risk for AD
Borderline IQ
IQ of 71-84
MR; 71-75 with poor adaptive functioning
LD
20-30% of children w/ LD have ADHD
Greater risk of antisocial beh
More males
Comm DIs
Exp, Exp-Rec, Phono, Stuttering (stress reduction)
PDD
Impairments in:
1. Comm
2. Social Int
3. Stereotyped Beh
Autism
Prior to 3
1. Comm
2. Social Int
3. Stereotyped Beh
70% have MR
Atrophic cerebellum and enlarged ventricles
Rett's
Normal until 5+ months
Deceleration of head growth
Loss of motor
Language impairment
Less social interest
ONLY IN FEMALES
Childhood Disintegrative Disorder
After 2 normal years, regression in 2 areas.
Asperger's
Social imp & restriced interests w/o comm diff
ADHD
Onset before 7
Duration of 6 months
Lower IQ, yet ave/high ave
25-30; 50% have LD
30-90% have Conduct Dis
Inattentive = equal gender
Hyper = more males
60% of children cont as adults w/ CONDUCT PROBLEMS being a good predictor of issues.
ADHD and Beh Disinhibition Hypothesis
Core ADHD feature is the inability to regulate behavior to fit situational demands.
ADHD Treatment
Methylphenidate (Ritalin)
Atomoxetine (Strattera; non stimulant)
Classroom management
Parental involvement
Conduct Disorder
W/in the past 12 months:
1. Agg to people or animals
2. Destruction of property
3. Deceitfulness or theft
4. Violation of rules
CD
Childhood onset = prior to 10
Adolescent onset = after 10
Moffitt's CD Types
Life-course-persistent
Adolescence-limited
CD Treatment
Target preadolescents + family intervention
Pica
12-24 months
Tourette's Disorder Treatment
Antipsychotics = Haloperidol and pimozide (80% effective)
Enuresis
Clinical after 5 years of age
Night alarm
Separation Anxiety
Lasts 4 weeks
Onset before 18
RAD
Inappropriate social relatedness before 5.
Parental neglect or family chaos
Inhibited and Disinhibited
Behavioral Pediatrics (Pediatric Psy)
In response to increased mental disorders in children w/ medical conditions.
Behavioral Ped Recommendations
Disclose asap
Cog-Beh anxiety reduction for med procedures
Allow family to visit often
Reduced compliance
School adjustment
Delirium
Disturbance in consciousness
Change in cog and/or perceptual abnormalities
Dementia
Memory + 1
Pseudodementia = intact rec w/ poor procedual memory.
AD
Course until death = 8-10 years
> Females
HIV Dementia
Motor slowness, absence of aphasia, severe dep/anx.
Substance Dependence
12 months of 3 criteria:
Tolerance
Withdrawal
Larger amounts
Can't stop
Time spent
Impacts activities
Continued use despite issues
Substance Dependence Treatment
Covert sensitization
Aversion therapy
Social skills training
Stress management
AA
Most common precipitant of relapse...
...anxiety, frustration, and depression.
Marlatt & Gordon (1985)
Dep is an "overlearned habit pattern" w/ a "abstinence violation effect" that invokes anxiety and self-blame.

Decrease relapse when relapse is blamed on external factors.

Marlatt's model of substance dependence holds that addicts learn to associate substance use with relief of self-criticism and guilt through a variety of cues and reinforcers, such as advertisements depicting people feeling cheerful when drinking and social occasions in which a carefree attitude is reinforced. In other words, there are a variety of cues and mediators that serve to encourage and reinforce use. Contrary to the person's expectations, however, excessive substance use only exacerbates problems, such as interpersonal or work-related problems. This leads to more self-criticism and guilt, which the person again attempts to relieve by using. In other words, substance use is self-reinforcing -- it is the cause of and the expected solution to the same problems. And it is "over-learned" in that, due to the multiplicity of its antecedents, it becomes a strongly ingrained behavior.

According to Marlatt, a slip is likely to lead to a full-blown relapse when the person makes dispositional attributions for it, such as when the person blames him or herself. Relapse prevention involves teaching the person to make non-dispositional attributions, such as blaming the situation or the nature of the disease.
Relapse Prevention Program
Id environmental cues and coping with them.
Nicotine Dependence
Only 7.5% quit long term
91% of them do it alone
Treatment:
Nicotine replacement
Beh therapy
Clinical support
Substance Abuse
1 criteria in the last 12 months:
Impacts life
Use of sub
Recurrent legal issues
Continued use despite issues
ETOH withdrawal
Autonomic hyperactivity (e.g., sweating tachycardia), tremor, insomnia, nausea, hall, anxiety, seizures.
Schizophrenia
6 months or more w/ 2 or more:
Delusions
Hall
Dis Speech
Disorganized or catatonic beh
Neg symptoms
Schizophrenia Subtypes
Paranoid - most fav prognosis
Catatoic
Disorganized
Undifferentiated
Residual
Type I & II Schizophrenia (Crow; 1985)
Type I = Positive symptoms; responds well to antipsychotics; neurotransmitter based
Type II = Negative symptoms; poor premorbid functioning; brain structure based
Schizophrenia Prevalence
Males>Females
Male onset (18-25)
Female onset (25-35)
A-A>Majority
Patients from developing countries more often exhibit an acute onset of symptoms, a shorter clinical course, and a complete remission of symptoms.
Etiology
Gen pop = .5%-1.5%
Concordance rate
Bio sib = 10%
Fraternal twin = 17%
Identical twin = 48%
Child (both parents schiz) = 46%
Dopamine Hypothesis
Schizo is the result of elevated dopamine.
Antipsychotics
Reduce positive symptoms
Tardive dyskenesia
Atypical Antipsychotic
Clozapine = less likely to cause TD and help neg symptoms
Expressive Emotion
High EE is associated with relapse
Schizophreniform Disorder
1 month, but less than 6 for impairment
2/3 convert to Schizo
Delusional Disorder
"Nonbizarre"
Poisioned
Betrayed
Followed
Delusional Subtypes
Erotomanic
Grandiose
Jealous
Persecutory
Somatic
Brief Psychotic Disorder
1 day, but less than 1 month
MDE
2 weeks
5 criteria
Manic Episode
1 week or longer
3 criteria
Hosp, impairment, psychotic
Hypomanic
4 days
3 symptoms of manic
increased productivity, increased efficiency, and increased creativity
Mixed Episode
1 week
MDD
10-20% after childbirth
2x more likely for females than males beginning in adolescence
Onset = mid 20's
W/ psychotic features (higest suicide rate)

A rapid onset of REM sleep
Decreased percentage of slow wave sleep
Increased REM sleep

No prior history of depression but rapid REM onset have an increased risk of developing depression
MDD Course
May last 6 months or longer, but usually w/ remission
20-30% w/ residual
50% remission rate
Catecholamind Hypothesis
Depression = low norepinephirne levels
Indolamine Hypothesis
Depression = low dopamine levels
Antidepressants
1. Tricyclics (TCA's) - "classic" depression w/ vegative symptoms
2. SSRI - fewer SE/melancholic dep
3. Monoamine oxidase inhibitors (MAOI's) - phobia
Dysthymic Disorder
Chronic low grade depression over 2 years for adults and 1 year for children.
No symptom free periods
Bipolar I
Manic is focus
Bipolar II
Depression w/ Hypomania (no mania)
Bipolar Demographics
BP I = M/F
BP II F>M
Onset in early 20's
BP Genitic Links
65% Mono twins
14% Di twins
Cyclothymic Disorder
Hypomania and depressive symptoms
Dep must be at least 2 years in adults and 1 year in children
Suicide
60-80% who complete have made previous attempts
80% warning signs
Attempts 24-44
Complete 65+
Highest for Whites
Divorce, separation, widowed
Hopelessness
Socially-prescribed perfectionism
Life stress
3 months after MDD improves
Low serotonin
Panic Disorder
2+ PA's w/in 1 month
Panic Disorder Demographics
Females > Males
Onset adolescence - mid 30's
Treatment for PA
Flooding (60-70% effective)
Drugs alone have a high relapse rate (30-70%)
Types of Specific Phobia
Animal
Natural Environment
Situational - flying, elevators, bridges; most common
Blood-Injection-Injury
Other
Social Phobia
Fear of social performance or sit that may cause embarrassment or humiliation as the result of scrutiny or evaluation by others.
OCD
Males=Females
Ave onset is earlier in males (@ 6 more males)
Low serotonin
Due to hyperactive right caudate
Treated with tricyclic/SSRI and exposure/prevention
PTSD
Experience and react to stress
Symptoms last > 1 month
Treatment = Cog-Beh, exposure, anx man w/ SSRI
Acute Stress Disorder
Onset w/in 4 weeks and last for 2-3 weeks
Emotional detachment
Derealization
Diss amnesia
GAD
6 months
3 criteria:
Keyed up
Easily fatigued
Diff concentrating
Irritability
Muscle tension
Sleep distrubance

Treated with Cog-Beh SSRI or anxiolytic
Somatization Disorder
Prior to age 30 and persist for several years
4 pain
2 gastrointestinal
1 sexual
1 pseudoneurological

Research indicates that 61% of Somatization Disorder patients have one or more co-occurring personality disorders. The next most frequent co-diagnoses are Major Depression (55%), Generalized Anxiety Disorder (34%), and panic disorder (26%).
Conversion Disorder
Voluntary motor or sensory functioning that suggests a serious condition. Symptoms reduce under hypnosis or amytal interview.
Primary gain = psychological conflict remains intact
Secondary gain = avoid activity or gain support
Fictitious Disorder
Intentional feigning to be in a "sick role"

Internal reward (Malngering is external reward)
Dissociative Amnesia Subtypes
1. Localized - all events; specific time
2. Selective - some events; specific time
3. Generalized - entire life
4. Continuous
5. Systematized - category specific
Dissociative Fugue
Travel
Depersonalization Disorder
"Out of body"
Dyspareunia
Genital pain w/ intercourse
Vaginismus
Involuntary spasms that interferes w/ intercourse
Paraphilias
Intense sexual urge or fantasy involving obj, suffering/humiliation, or children
Gender Id Dis
Discomfort w/ sex and desire to be the opp.
Dyssomnia
Disturbance in amount, quality, and timing of sleep
Parasomnias
Beh or psychiological abn during sleep
Bulimia
Binging and compensation 2x/week for 3 months
Genetic
Low levels of opioid beta-endorphins, serotonin, and norepinephrine
Paranoid Personality Disorder
Distrust
4 criteria
Schizoid PD
Lack of interpersonal relationships
4 criteria
Schizotypal PD
Low social and odd behaviors
5+ criteria
Antisocial PD
Must be 18
Conduct disorder by 15
3 criteria
Borderline PD
5 characteristics
Treated w/ Dialectical Behavior Therapy
Histrionic PD
Attention-seeking
5 criteria
Narcissistic PD
Grandiosity
Lack of empathy
Need for admiration
5 criteria
Avoidant PD
Social ingibition
Feelings of inadequacy
Hypersensitivity to neg eval
4 criteria
Dependent PD
Excessive need to be taken care of which leads to submissive, clinging beh, and fear of separation.
5 Criteria
OC PD
Order!
4 criteria