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65 Cards in this Set

  • Front
  • Back
concha =
turbinate
concha bullosa =
air-filled cavity within a turbinate
Is having concha bullosa pathological?
normal anatomic variant in <50%
- NOT more prone to have sinus disease
- but a large concha bullosa can obstruct sinus openings leading to recurrent sinusitis
-often associated with deviation of the nasal septum toward the opposite side
Haller cell =
pneumatized infraorbital ethmoid cell
(below the orbit in the roof of the maxillary sinus)
How can a Haller cell be pathological?
occurs in 10% of people
enlarged Haller cells → narrowing of the ethmoidal infundibulum → recurrent sinus disease
Paradise Criteria for Tonsillectomy in children
7 or more episodes in the preceding year, OR
5 or more episodes in each of the preceding 2 yrs
3 or more episodes in each of the preceding 3 yrs
most common tumour of the nose
benign polyposis
OR
SCC
adenoma
Treatment for nasal polyps causing obstructive symptoms
CT ± MRI
prednisone (reducing regimen)
saline nasal douche
± biopsy
symptoms of vasomotor rhinitis
persistent daytime rhinorrhea
laryngopharyngeal reflux only responds to
Gaviscon vs. Nexium
(ie. antacid vs. PPI)
polyp =
benign growth
innervation of nasal glands
Vidian n.
(nerve of the pterygoid canal)
steroids are effective in rhinitis/rhinosinusitis?
rhinosinusitis (sinusitis) only
rhinitis =
inflammation of mucous membranes of the nose
rhinosinusitis (sinusitis) =
inflammation of the sinuses
Nasal Obstruction Hx
Is it a runny or blocked nose? Postnasal drip?
How often? How long?
Has it affected your sense of smell or taste?
Infections?
Epistaxis?
Sore throat?
Any pain?
Headache?
Earache, hearing?
Tonsillitis?
Snoring?

Have you had any previous surgery?
nasopharyngeal carcinoma can be visualised by laryngoscopy of the
postnasal space
Tonsilitis Hx
Have you ever had glandular fever / quinsy before?
What is the risk of bleeding post-tonsillectomy?
10% within 2 weeks
Epistaxis Protocol
Lean forward, pinch nostrils tightly & hold for 15 min. Don't blow the nose.

3 treatments tds for 3 days = 3x3x3
1. wash nose using nasal saline douche
2. topical decongestant (3 sprays each nostril)
3. moisturise & protect with Nasalate cream
venous drainage of tonsils
peritonsillar plexus
lingual veins and pharyngeal veins
internal jugular vein
nerve supply of tonsils
glossopharyngeal (CN 9)
blood supply to tonsils
branches of external carotid a. :
Lingual artery
Facial artery
Ascending pharyngeal artery
Maxillary artery
the tonsil bed is formed by 2 muscles
Superior constrictor
Styloglossus
palatine tonsil arises from which embryological origins
2nd pharyngeal pouch (endoderm)
tonsils =
mucous lymphoid tissue
1st line of defence, forms Waldeyer's ring
components of Waldeyer's ring
palatine tonsils
lingual tonsils
adenoids
openings of eustachian tube
Describe the anatomical location of the palatine tonsils
lateral oropharynx

Deep - Superior constrictor muscle
Anterior - Palatoglossus
Posterior - Palatopharyngeus
Superior - Soft palate
Inferior - Lingual tonsil
contraindications to tonsillectomy
Acute infection
Bleeding predisposition
Anemia
Poor anesthetic risk or uncontrolled medical illness
Conditions that predispose to sinusitis
Allergic/Nonallergic rhinitis
Anatomic (Septal deviation, Paradoxical middle turbinate, Ethmoid bulla hypertrophy, Choanal atresia, Adenoid hypertrophy)
GORD
hormonal factors (eg. pregnancy, hypothyroid)
immune deficiency
microorganisms in acute sinusitis
Streptococcus pneumoniae
Haemophilus influenzae
Moraxella catarrhalis
microorganisms in chronic sinusitis
Streptococcus pneumoniae
Haemophilus influenzae
Moraxella catarrhalis
Staphylococcus spp.
concha bullosa
septal spur
Sx of chronic sinusitis
Facial pain or pressure
Facial congestion or fullness
Nasal obstruction or blockage
Nasal discharge, purulence, or postnasal drip
Hyposmia or anosmia
Headache
Fever
Halitosis
Fatigue
Dental pain
Cough
Ear pain, pressure, fullness
posterior ethmoid & sphenoid sinus drainage to
sphenoethmoidal recess and superior meatus
anterior ethmoid & frontal/maxillary sinus drainage to
middle meatus
Zenker's diverticulum (pharyngeal pouch) classical hx finding
coughing up /spitting undigested food
halitosis
Function of anterior 2/3 vocal cord
phonation
Function of posterior 1/3 vocal cord
respiration
what is the management of vocal cord nodules (benign) ?
conservative mx with voice therapy/rest & PPI
classical hx finding in Laryngospasm
panic attacks
inspiratory stridor
cyanosis
classical exam finding in Laryngospasm
paradoxical vocal cord movement
prognosis of fungal sinusitis
high mortality
Cholesteatoma
a destructive and expanding growth consisting of keratinizing squamous epithelium in the middle ear / mastoid process
acute otitis media common age group
1-6 years old
microoganisms causing acute otitis media
Strep pneumoniae
H. influenzae
Moraxella catarrhalis
S. aureus / pyogenes
viral
Pathophysiology of acute otitis media
eustachian tube obstruction → air absorbed in middle ear (becomes irritant) → oedema of mucosa → infection of exudate from nasopharyngeal secretions
Causes of acute otitis media
URTI
allergies / chronic sinusitis
adenoid hypertrophy
tumour (eg. NPC)
cleft palate
Down's syndrome
immunosuppression
Risk Factors for acute otitis media
bottle feeding
2nd hand smoke
crowded living
male
+ve FH
Sx of acute otitis media
TRIAD:
fever + otalgia + conductive hearing loss

Rarely:
tinnitus, vertigo, CN7 paralysis, otorrhea if TM perforated
Tx of acute otitis media
amoxycillin / cephalosporin for 10-14 days
± paracetamol
± decongestants
Prevention of acute otitis media
vaccination (Hib, pneumococcal)
antibiotic prophylaxis
surgery
Complications of acute otitis media
TM perforation
chronic suppurative otitis media
effusion
brain abscess, meningitis
mastoiditis
labyrinthitis
CN 7 paralysis
most common benign tumour of parotid gland
pleomorphic adenoma (rarely transformation to SCC)
adenolymphoma
most common malignant tumour of parotid gland
SCC
adenocarcinoma
Symptomatically what is the distinguishing feature between acute sinusitis & chronic sinusitis?
acute = FEVER (unwell), pain
How does the middle turbinate separate the blood supply of the nose?
ABOVE middle turbinate = internal carotid a.
BELOW middle turbinate = external carotid a.
blood supply of the superior nasal septum
internal carotid a. → ethmoidal aa.
blood supply of the lower nasal septum
external carotid a. → sphenopalatine + greater palatine + nasopalatine a.
→ anastomose to form Kiesselbach's plexus anteriorly
Investigations for epistaxis
CT
FBC & coags
Tx of epistaxis
ABC
hemostasis
Causes of epistaxis
LOCAL:
trauma - fracture, foreign body
dryness
septal perforation
inflammation - rhinitis, infection
tumors - benign (polyps, inverting papilloma, angiofibroma) & malignant (SCC)
iatrogenic / cocaine

SYSTEMIC:
coagulopathies (liver failure, vWB, hemophilia, haematological malignancy, anticoagulants)
Treatment of benign tumours of the vocal cord
voice rest
speech therapy
Which patients with hoarseness should you refer to an ENT?
voice change > 3 weeks
red flags (neck trauma, hx of malignancy)
px who rely on their voice as professionals