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61 Cards in this Set

  • Front
  • Back

Function of Sodium

-major extracellular ion


-transmits nerve impulses


-acid base balance


-maintains osmotic pressure

Sources of Sodium

salt and processed food

Causes of hyponatremia (low sodium)

-hypervolemia (gains of water)


*liver, heart, kidney failure


*inappropriate sodium release


-excessive sodium loss


*diuretics


*V/D/GI suction and excessive sweating

causes of hypernatremia (high sodium)

dehydration, insufficient water intake, cushings disease, diabetes insipidus, primary aldosteronism

regulation of sodium

eat/food and intestines


-minimum requirements


-excreted


symptoms of hyponatremia

-serum sodium of <135mEq/L


-muscle cramps, weakness (falls in elderly), fatigue


-anorexia, N/V


-altered mental status (confusion)

symptoms of hypernatremia

-serum sodium of >145 mEq/L


-thirst


-weakness, muscle twitching


-seizures, coma and death

tests results for hyponatremia

-serum osmolarity less than 280


-serum sodium less than 135


-urine specific gravity less than 1.010


-elevated hematocrit and plasma protein levels

tests results for hypernatremia

-urine specific gravity greater than 1.030


-serum osmolarity greater than 300


-serum sodium level greater than 145

treatment for hyponatremia

-when both sodium and water have been lost= IV NS, hypertonic solutions such as 3% or 5% NaCl solutions given cautiously for very low sodium levels


-when sodium is low but water is normal or excess = give diuretics and initiate fluid restrictions

treatment for hypernatremia

water replacement (o.45% NaCl or D5)


-diuretics

potassium function

-major cation


-supports many metabolic functions


-2% in extracellular, 98% in intracellular fluid


*maintains rate and force of heart conduction, nerve conduction, muscle function, acid base balance, osmotic pressure


-K+ has a "K"ICK effect on heart (cardiaK), GI tract, skeletal muscle

potassium sources

bananas, apricots, raisins, spinach, milk, chicken, tuna

why is potassium important?

-maintains cells electrical neutrality and osmolarity


-aiding neuromuscular transmission of nerve impulsives


-assisting skeletal and cardiac muscle contraction and electrical conductivity


-affects acid base balance in relationship to hydrogen

regulation of potassium

-normal range is 3.5 to 5mEq/L


-ingest daily


-recommendation


-80% of K is excreted


-sources

hypoKalemia

-level drops below 3.5


-moderate 2.5-3


severe less than 2.5


-not enough in


-too much out


-drugs

what to look for with hypoKalemia

-skeletal muscle weakness,


-paresthesia


-leg cramps


-deep tendon reflexes decrease


-tachycardia


-arrhythmias


-cardiac arrest


test results for HypoKalemia

-serum level less than 3.5


-increased 24 hour urine level


-elevated pH and bicarb levels


-charecteristic ECG changes

treatment for hypoKalemia

-restore normal potassium balance


-high potassium low sodium diet


-oral vs IV


*may give runs of 10mEq of K in 100ml of fluid to run over 1-2 hours (never any faster)

HyperKalemia

-potassium levels rises above 5


-moderate 6.1-7


-severe greater than 7


-less common


-kidneys


-acidosis


-cell injury

what to look for with hyperKalemia

-effects of neuromuscular and cardiac functioning


-muscle weakness to flaccid paralysis


-hyperactivity in GI track


-Decreased heart rate, cardiac output, B/P


-ECG changes

tests for hyperKalemia

-serum potassium levels higher than 5


-decreased arterial pH


-ECG abnormalities

how hyperKalemia is treated

-loop diuretics


-potasssium restricted diet


-kayexalate

Calcium function

needed for bone and tooth formation, blood clotting, muscle contraction and nerve impulse conduction


-calcium: "ca"lms vessels and heart

sources of calcium

dairy products, spinach, broccoli, tofu

regulation of calcium

-dietary intake


-vary per patient


-dietary products


green leafy vegetable


-absorbed by the small intestine


-excreted in urine and feces

causes of hypocalcemia

-thyroidectomy with accidental removal of parathryroids; hypoparathyroidism; decreased albumin (liver disease); inadequate intake/absorption; severe respiratory alkalosis; acute pancreatitis, renal failure

causes of hypercalcemia

-hyperparathyroidism


-cancer (bone or PTH producing tremors)


-pagets disease


-prolonged immobilization (breakdown of bone)


-hypophospatemia

symptoms of hypocalcemia

-calcium level of <8.5


-tetany (positive chvostek's sign, Trousseaus sign)


-laryngospasm (difficulty speaking and breathing)
-dysrthymias, cardiac arrest


-diarrhea


symptoms of hypercalcemia

calcium level of >10mg/dl


-muscle weakness


-decreased deep tendon reflexes


-constipation


-EKG changes/hypertension

treatment of hypocalcemia

-oral supplements (TUMS/diet)


-IV calcium preparation

treatment of hypercalcemia

-IV fluids (saline) (to promote renal excretion)


-diuretics


-calcitonin (promotes uptake of calcium into the bone)


-Biphosphonates (prevent bone breakdown)

Magnesium function

-regulates muscular contraction and neuromuscular control (calming effect also)


-regulates blood clotting


-promotes carb metabolism


sources of magnesiunm

-green veg, grains, nuts, meats and seafood


-about 25mEq daily intake


-40% will be absorbed in small intestine

causes of hypomagnesemia

-ETOH (most common cause)


-inadequate absorption/intake


-renal disease


-diuretic abuse


causes of hypermagnesemia

renal failure with intake of magnesium containing antacids or laxatives

symptoms of hypomagnesemia

-serum magnesium < than 1.6mg/dl


-tremors, hyperreactive reflexes, positive chvosteks sign and Troussseaus sign, tetany


-GI- N/V/D, anorexia and abdominal distention

symptoms of hypermagnesemia

-serum magnesium >2.6 mg/dl


-weakness, lethargy, loss of deep tendon reflexes


-respiratory depression


-ECG changes/cardiac arrest

treatment of hypomagnesemia

-dietary/oral replenishment


-IV magnesium sulfate


-if taking TPN, add magnesium

treatment of hypermagnesemia

-treat underlying cause


-withhold drugs with magnesium


-dialysis


-calcium gluconate to reverse neuromusclular and cardiac effects

Phosphorus function

necessary for use of CHO, proteins and fats


-necessary for ATP production


-vital for RBC, nervous and muscle cell function

sources of phosphorus

milk, meat, fish, eggs, poultry, legumes, nuts, cereal, bread

causes of hypophosphatemia

-refeeding syndrome


-antacids bind with phosphate and lower levels


-alcohol intake

causes of hyperphospatemia

renal insufficiency/failure


-disease process that lead to hypocalcemia (hypoparathyroidism, hyperhyroidism)

symptoms of hypophosphatemia

-serum phosphorus level <2.5 mg/dl


-irritability, weakness, numbness, confusion


-hemolytic anemia


-respiratory failure


-chest pain, dysrhythmias


-anorexia, n/v, ileus

symptoms of hyperphosphatemia

-serum phosphorus level of >4.5 mg/dl


-symptoms result more from decrease in calcium levels assoc with elevated phosphare


-muscle cramps, tetany, tingling around mouth

treatment of hypophosphatemia

increase phosphate in the diet (milk, meat, fish, eggs, poultry, cereal, bread)


-oral or IV supplement

treatment of hyperphosphatemia

limit dietary intake,


bind phosphorus in the GI tract (TUMS)


-dialysis

Giving IV electrolytes

always dilute electolytes


-give in large vein or use central line if available


-give slowly and use a pump


-slow down if causing pain or can place warm pack over IV site

fluid volume

-fluids act as a medium for transport of nutrients and wastes


-medium for metabolic reactions within the cells


-helps regulate body temp


-acts as lubrication and insulation

causes of fluid volume deficit

-loss from GI tract


-diuretic therapy, abuse of laxatives


-sweating, increased enviornmental temp


-hemorrhage


-inadequate intake (NPO)


-third spacing (loss of vascular fluid into spaces where it is not useful to do its job)


--losses into the abdomen, bowel, pleural space

symptoms of fluid volume deficit

-drop in body weight, dry mucous membranes, decreased urine output, altered mental status, diminished skin turgor, decreased capillary refill, increased pulse, decreased BP

treatment of fluid volume deficit

vital signs, administer ordered fluids, assess skin turgor/mucous membranes, daily weight, I/O

Causes of fluid volume excess

conditions that cause retention of sodium and water


-heart failure, cirrhosis, renal failure


-steroid admin


-admin of IV fluids


-post op fluid mobilization in patients with heart and kidney issues

symptoms of fluid volume excess

increased body weight, cough, dyspnea, crackles in lungs, edema, full bounding pulse, increased blood pressure

treatment/nursing care for fluid volume excess

-diuretics, fluid restrictions, sodium restricted diet, intake and output, daily weight, lung assessment, vital signs, assess extremities for edema

When C02 is elevated think...

ACIDOSIS

when C02 is decreased think...

ALKALOSIS

when HC03 is elevated think..

ALKALOSIS

when HC03 is decreased think...

ACIDOIS

Compensation

buffers, lungs, kidneys