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26 Cards in this Set

  • Front
  • Back
Sinus Node Dysrhythmias include
SInus bradycardia and SInus Tachycardia
Sinus bradycardia is a hr of
less than 60 bpm shown in p wave before qrs
Sinus tachycardia is a hr of
more than 100 bpm and is show in p wave before QRS
Atrial dyrhythmias include
PAC and A FIB
PAC atrial dyrhythmia
Premature Atrial Contraction that is a contraction that originates from somewhere else in the atria not in the SA node where it should be
in healthy people PAC's result from
stress, caffeinem and tobacco and slim people
A fib atrial dyrhythmia is caused by
the SA losing the ability to control the atrium so chaotic impulses arise from other areas of the atria and result in disorganized atrial depolarization
With A fib is the atrial contraction is not
effective
A fib can cause
impulses to not get through the AV node, so they are very irregular ventricular response
There is an increased risk for _____ with a fibs
embolism in the atria
due to fear or mural thrombus' forming as a result of atrium emboli risk
Must give patients anticoagulants
The most common dysrhythmias are
atrial dysrhythmias and they increase with age
Ventricular dysrhythmias include
PVC, Ventricular tachycardia, Ventricular fibrillation
Ventricular dysrhythmias are a big problem because
they have greater volume effects
PVC ventricular dysrhythmia is
premature ventricular contraction cause by a ventricular ectopic pacemaker (prob with av node and can be caused by premenopause hormones)
Ventricular tachycardia (ventricular dysrhythmia) originates in ___ at what rate
in the ventricle at a rate of 70-250 bpm and is sudden or insidious
Ventricular tachycardia (ventricular dysrhythmias) are charecterized by
wide (>.10 seconds) tall QRS complexes.
Ventricular tachycardias are a big problem because
Cardiac output is diminshed (ventricle cant contract, recive or empty volume)
Ventricular fibrilliation (ventricular dysrhythmia) is a
quivering of the ventricle but there is no contraction.
Ventricular fibrillation is fatal without intervention because
there is no cardiac output and is fatal without intervention
Av block ventricular dyrhythmias include
1st degree, 2nd degree, third degree
Av block first degree is shown by a
prolonged PR interval and slowing through the AV node but it usually asymptomatic
Av block seond degree is
intermittent failure of one or more impulse conductions being delayed from the atria to the ventricles.
Two types of second degree av blocks include
Type 1- wenckebach or type 2 which is more serious
Third degree av block is
a complete block and there is no connection between the atria and ventricles, both are firing seperately by seperate pacemakers.
Third degree av blocks must be treated with
a pace maker