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30 Cards in this Set
- Front
- Back
helix
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outer, distal ridge of the pinna
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scaphoid fossa
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trough between the helix and antihelix
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antihelix
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second ridge in, moving proximally
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tragus
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cartilaginous projection extending posteriorly from the middle of the anterior margin of the pinna. Extends over the entrance to the EAM, can shut the eam with it.
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antitragus
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projection of cartilage, posterior and inferior to the tragus.
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lobule (lobe)
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no cartilage, just fat and connective tissue. serves no purpose.
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crura
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fork or extension. antihelix has two with the triangular fossa in between.
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crus of helix
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separates the concha into two sections.
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conch cymba
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part of concha, superior to crus of helix
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concha cava
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part of concha, inferior to crus, entryway to EAM
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microtia and anotia
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small underdeveloped pinna/ no pinna (can also indicate problem in EAM). often seen with stenosis or atresia of the ear canal, which does affect hearing.
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protrusion
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ears that stick out. can be fixed with otoplasty, but seldom done in children because scar tissue does not grow in the same way that normal tissue does.
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auricular deformities
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pinna is deformed due to failure to develop at various stages of gestation
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EAM
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(ear canal) 7 mm in diameter, 25-35 mm in length. elongated S on it's side that provides insulation and protection.
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lateral 1/3 of EAM
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lateral 1/3 cartilaginous, covered with epithelial cells, many having cilia. sabecous glands produce oil and ceruminous glands produce cerumen. innervated by CN V: trigeminal.
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medial 2/3 of EAM
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osseus portion, surrounded by temporal bone, canal narrows at juncture of cartilagious and osseus portions
no cilia, or glands innervated by CN X (vagus) also controls cough reflex. no lining btw epithelium and bone. skin is thin 1/10mm, and has many pain sensors and blood vessels. |
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cerumen
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production is normal, natural and necessary. traps insects and dirt, which is swept outward by cilia to entrance of EAM where it dries and falls out. impacted cerumen occurs when you produce it faster than cilia can sweep it out.
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tympanic membrane
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concave medial boundary of the outer ear. converts acoustic to mechanical energy.
55mm2 in area, 10mm in diameter, 55 degree angle to floor of EAM three layers:external (continuation of epithelial lining of the EAM), middle (fibrous and tough, circular and radial fibers), inner (continuation of mucous membrane lining of the middle ear). very rich in blood supply, size of pinky nail |
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TM landmarks
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pars tensa: 4/5 of TM, held taut and pulled medially by manubrium of malleus (points to left in left ear, and right in right ear)
umbo: inferior end of the manubrium, most medial pt. cone of light: always in anterior/inferior quadrant in normal TM pars flaccida: sup/ant 1/5 of TM, no fibrous layer. inferior border is the lateral process of malleus and the ant/post malleolar folds |
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atresia
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lack of EAM. Treacher-Collins syndrome. not always treatable with surgery, requires a bc hearing aid or a bone anchored hearing aid (BAHA)
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stenosis
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narrowing of the EAM. does not cause HL by itself, but can cause ear wax build up and is common in down syndrome.
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foreign bodies
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disorder of EAM. canal narrows at the osseocartilaginous junction. HL is not the main problem, infection is.
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external otitis
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swimmer's ear. may be bacterial or fungal. can cause extreme pain and foul smelling discharge. can spread to the TM (myringitis) causing some HL.
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osteomas
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bony tumors that grow in the medial, osseus portion of the EAM. may be benign or malignant. cause hearing problems only is they completely close the canal.
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exostoses
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grow in the EAM. look like osteomas but are bony projections, not tumors. surfer's ear-swimming in cold water. completely benign.
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cerumen occlusion
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overactive cerumenous glands and/or incorrect removal techniques that push the wax into the bony portion (o.c. junction) of the canal where cilia can't remove it.
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ways to remove ear wax
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cerumenolytic, such as peroxide, dissolves it.
irrigation suction curettage degree of HL depends on the amount of occlusion. |
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manubrium
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the handle of the malleus is imbedded in the fibrous middle layer of the TM, inclining toward the front of the head.
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TM perforation
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caused by pressure build up (from fluid in the ME bulging the TM out), infection eating through TM, insertion of objects into ear canal, sudden pressure change. when healed can leave a weak spot in the firous layer. almost always causes HL but HL is not proportional to the size of the perforation.
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tympanoslerosis
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thickening of TM, calcium plaques. result of frequent perforations or ear surgeries and may or may not cause HL.
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