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53 Cards in this Set
- Front
- Back
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liquid part of blood, contains fibrigen 45-55% blood volume
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Plasma
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3.6-5.4
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RBC
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5000-10000
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WBC
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protective fx
life span 1-2 days |
leukocytes
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150000-350000
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platelets
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neutrophils, basophils, eosinophils
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granulocytes
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increas in cancers and autoimmune disorders
decreass with use of steroidss, hiv, hodgings |
agranulocytes
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life span of 7.5 days, most are in blood-others in spleen
release glycoprotein IIb/IIIa |
thrombocytes
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most abundent protein in plasma. helps maintain osmotic pressure.
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albumin
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12-17.4
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hemoglobin
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40-54% male
37-47% female |
hct
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diagnosis pernicious anemia, macrocytic anemia, and malabsobtion syndromes
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schillings test
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symptoms common to all blood dyscrasias
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fatigue
dyspnea bleeding chills&fever |
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used for absorbtion of iron
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vitamin c
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abnormally low circulating volume of blood caused by sudden loss of small amounts of blood over a prolonged period.
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hypovolemic anemia
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intrinsic factor determines with age
normally present in stomach secretion needed for absorbtion of vit b 12 |
pernicious anemia
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not enough rbc, wbc, and plt
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aplastic anemia
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not enough iron to produce hgb caused by increase blood loss, not enough Fe in system
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iron deficiency anemia
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crescent shaped rbc, chronic and incurable, most common inherited disorder in us
must get 2 defective genes 40 % of hgb is affected |
sickle cell anemia
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increase in rbc, wbc, and plt
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polycythemia vera
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herediatary hemolytic anemia
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thallassemias
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immature
rbc, rellated to intake of b9 |
folic acid anemia
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s/s: weakness, dyspnea, tachycardia, hypotension, and decreased uo, changes to loc
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hypovolemic anemia
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s/s: stomatitis, glossitis, digestive problems, diarrhea, if severe jaundice
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pernicious anemia
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have to take b12 for life
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pernicious anemia
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s/s: normal s/s of anemia,
spleen increase in size, and increase # of infections, clotting abnormalties |
aplastic anemia
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s/s tired, cold, dyspnea, tachycardia
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iron deficiency anemia
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don't take antacids with this med
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iron
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will stain teeth
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iron
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sickle shapes cells get stuck in sm bld vessels
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sickle cell crisis
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don't give this med with sickle cell anemia. will provoke grand mal seizures
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demerol
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s/s jaundice, infection, block bld vessels, leg ulcers from blockage
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sickle cell anemia
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s/s face/lips reddish-purple possibility of thrombis formationedema of feet and legs
enlarged spleen increased bd |
poly vera
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chronic premature destruction of RBC causes cardio pulmonary bypass, poison arsenic, malaria, and hazaedous chemicals.
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hemolytic anemia
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asymptomatic frequent transfusions
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thalassemias
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s/s jaundice, enlarged spleen
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hemolytic anemia
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alcoholics, intestinal disorders, drugs
beefy red tongue, weak/fatigue |
folic acid anemia
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potentially fatal blood condition charcaterized by severe reducton of granulocytes caused by meds such as sulfonamides
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agranulocytosis
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s/s infection, ulcers on mm
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agranulocytes
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increased number of leukocyte, don't have protctive function
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leukocytosis
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abrupt begins with infect, blding,s/s anemia, wt loss, fever, bone, and joint pain.
50 % may be cured |
acute leukemia
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onset gradual, life expected is longer, not usually cured, can have remissions
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chronic leukemias
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decrease in platelets from bone marrow
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thrombocytopenia
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s/s bleeding disorders, petechiae,
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thrombocytopenia
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inherited disorder, mom carries it, males have it,
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hemophilia
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inflam of lympha channelscaused by acute strp ofr staph
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lymphangitis
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s/s fine red streaksfrom affected area to groin or armpit
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lymphagitis
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edema of extrem cused by blockage by tumor, growth, adnesions, or inflam
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lymph edema
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s/s massive edema, ant tightness, causes pressure and pain to afected area
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lymph edema
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s/s: fatigue, fever, st, faint red rash on hands
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mono
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malignant disease orgin lymph sytem, usually young men
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hodgkins disease
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neoplastic disorder of lyph tissue 1 out of 30
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non hodgkins's disease
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protimine sulfate
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heprain
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