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53 Cards in this Set

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liquid part of blood, contains fibrigen 45-55% blood volume
Plasma
3.6-5.4
RBC
5000-10000
WBC
protective fx
life span 1-2 days
leukocytes
150000-350000
platelets
neutrophils, basophils, eosinophils
granulocytes
increas in cancers and autoimmune disorders
decreass with use of steroidss, hiv, hodgings
agranulocytes
life span of 7.5 days, most are in blood-others in spleen
release glycoprotein IIb/IIIa
thrombocytes
most abundent protein in plasma. helps maintain osmotic pressure.
albumin
12-17.4
hemoglobin
40-54% male
37-47% female
hct
diagnosis pernicious anemia, macrocytic anemia, and malabsobtion syndromes
schillings test
symptoms common to all blood dyscrasias
fatigue
dyspnea
bleeding
chills&fever
used for absorbtion of iron
vitamin c
abnormally low circulating volume of blood caused by sudden loss of small amounts of blood over a prolonged period.
hypovolemic anemia
intrinsic factor determines with age
normally present in stomach secretion
needed for absorbtion of vit b 12
pernicious anemia
not enough rbc, wbc, and plt
aplastic anemia
not enough iron to produce hgb caused by increase blood loss, not enough Fe in system
iron deficiency anemia
crescent shaped rbc, chronic and incurable, most common inherited disorder in us
must get 2 defective genes
40 % of hgb is affected
sickle cell anemia
increase in rbc, wbc, and plt
polycythemia vera
herediatary hemolytic anemia
thallassemias
immature
rbc, rellated to intake of b9
folic acid anemia
s/s: weakness, dyspnea, tachycardia, hypotension, and decreased uo, changes to loc
hypovolemic anemia
s/s: stomatitis, glossitis, digestive problems, diarrhea, if severe jaundice
pernicious anemia
have to take b12 for life
pernicious anemia
s/s: normal s/s of anemia,
spleen increase in size, and increase # of infections, clotting abnormalties
aplastic anemia
s/s tired, cold, dyspnea, tachycardia
iron deficiency anemia
don't take antacids with this med
iron
will stain teeth
iron
sickle shapes cells get stuck in sm bld vessels
sickle cell crisis
don't give this med with sickle cell anemia. will provoke grand mal seizures
demerol
s/s jaundice, infection, block bld vessels, leg ulcers from blockage
sickle cell anemia
s/s face/lips reddish-purple possibility of thrombis formationedema of feet and legs
enlarged spleen
increased bd
poly vera
chronic premature destruction of RBC causes cardio pulmonary bypass, poison arsenic, malaria, and hazaedous chemicals.
hemolytic anemia
asymptomatic frequent transfusions
thalassemias
s/s jaundice, enlarged spleen
hemolytic anemia
alcoholics, intestinal disorders, drugs
beefy red tongue, weak/fatigue
folic acid anemia
potentially fatal blood condition charcaterized by severe reducton of granulocytes caused by meds such as sulfonamides
agranulocytosis
s/s infection, ulcers on mm
agranulocytes
increased number of leukocyte, don't have protctive function
leukocytosis
abrupt begins with infect, blding,s/s anemia, wt loss, fever, bone, and joint pain.
50 % may be cured
acute leukemia
onset gradual, life expected is longer, not usually cured, can have remissions
chronic leukemias
decrease in platelets from bone marrow
thrombocytopenia
s/s bleeding disorders, petechiae,
thrombocytopenia
inherited disorder, mom carries it, males have it,
hemophilia
inflam of lympha channelscaused by acute strp ofr staph
lymphangitis
s/s fine red streaksfrom affected area to groin or armpit
lymphagitis
edema of extrem cused by blockage by tumor, growth, adnesions, or inflam
lymph edema
s/s massive edema, ant tightness, causes pressure and pain to afected area
lymph edema
s/s: fatigue, fever, st, faint red rash on hands
mono
malignant disease orgin lymph sytem, usually young men
hodgkins disease
neoplastic disorder of lyph tissue 1 out of 30
non hodgkins's disease
protimine sulfate
heprain