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80 Cards in this Set

  • Front
  • Back
Nature & Nurture
Why are we all the same?

Why are we all different?
The Active Child
Children shape their own development

EX: Difficult temperament elicits harsh parenting
Continuity/Discontinuity
Children seem qualitatively different at different ages
Mechanisms of Change
How does cognitive development happen?
Sociocultural Context
How does culture influence development
Individual differences
What causes them?
Research & Children's Welfare
Applying Research, public policy
3 Contexts for Gathering data
Interview, Naturalistic Observation, Structured Observation
Interview
Pro: cheap, reveal subjective experience
Con:Biased reports, bad memory, prediction inaccurate
Naturalistic Observation
Pro:Useful for describing behaviors, illuminate social interaction processes
Con: Infrequent behaviors
Structured Observation
Pro:Same context, controlled comparison
Con:less natural, reveals less than interviews
Cross-Sectional
Different aged children studied at a single time
Longitudinal
Examined repeatedly over long time
Microgenetic
Observe intensively over short period of time
Correlational
Comparison of existing groups of children or an examination of relations among each child's scores on diff. variables
Experimental
Random assignment of children to groups and experimental control of procedures presented to each group
Correlation Coefficient
Small - .1 to .3
Moderate - .3 to .5
Large - >.5
Correlations and causality
Association between two things. Correlation does not imply causality
Zygote/Germinal
0-2 weeks
Implantation...7-9 days
Placenta by 14 days
Embryo
3-8 weeks
3 layers
Most rapid development
Cell migration & differentiation
Movement, facial features,heart beating
Fetus
9-38 weeks

recognize mother's voice, flavors, smells
Ectoderm
nervous system, skin, nails, teeth, inner ear, lens
Mesoderm
muscles, skeleton, circulatory system, internal organs
Endoderm
digestive system, lungs, urinary tract, glands
Habituation
decrease in response due to repeated presentation of stimulus
32 weeks
Dishabituation/Recovery
rebound in response due to subsequent change in stimulus
Teratogens
agents that cause harm during prenatal development
Factors: dose, genetic predispositions of mother and baby, combinations, age/timing
Eyes Vulnerable
3.5-7.5 weeks
Ears Vulnerable
4-8.5 weeks
Heart Vulnerable
3-6
Limbs vulnerable
4-5 weeks
Cigarette Smoking
Retarded growth/low birth weight
Nicotine constricts blood vessels and increases carbon monoxide in blood, delivering less oxygen to mom and baby
Fetal Alchohol Syndrome
Facial abnormalities
Slow growth; prone to infections
Other deformities: eyes and heart; ears and genitals; nose, throat, urinary tract
The brain and behavior:
Small head, seizures
Mental retardation, deficits in attention
Hyperactivity
Maternal Factors
Nutrition, Disease, Stress
APGAR Score
7-10 normal
4-6 borderline
0-3 needs immediate attention
Low birth weight
2500 grams/5.5lbs or less
Preterm
birth beginning of 36th week
36-40 is normal
Preterm infants tend to be...
More passive, less responsive
Fussier; harder to soothe
More trouble falling asleep, rousing, staying alert
Less regular schedule
Delayed milestones
At risk for abuse/attachment difficulties
Multiple risk model
Risks tend to co-occur
Multiple teratogens
Hazards that continue after birth
Poor health care
Rutter study on the multiple risk model

Poverty itself as a hazard?
Associated with more risk factors
Parent's genotype->Child's genotype
inherited chromosomes
DNA
base pairs:code for amino acids or instructions
Child's genotype->child's phenotype
Not a straightforward mapping from genotype to phenotype(dominance)
Child's environment->child's phenotype
Gene-environment correlation("passive")
Range of Reaction - any given genotype has a range of potential phenotypes depending on environment
Child's phenotype->child's environment
"The active child" - child creates its own environment:
Evocative & niche-picking
Leads to "active" G-E correlation
Gene-environment correlation
genes & environment are not independent since genetically related parents create the environment
Passive G-E correlation
parents pass on genes & environment(these are correlated)
Mom is pianist and has piano @ home
Active G-E correlation
child(because of genetic dispositions) changes environment in ways that affect expression of those very genes(evocative)
Behavior(al) Genetics
Teasing apart effects of genes & environment

Individuals who are genotypically similar should be phenotypically similar
Individuals who have been reared together should be more similar than those who have not
heritability
Proportion of variance in individuals that is due to genetic differences among those individuals
Neuron Parts
Dendrites, Axon, Synapse, cell body & nucleus
Dendrites
Receive input and conduct it toward cell body
Axon
conducts electrical signal from cell body to next neuron, myelin sheath
Synapse
Trillions, a single neuron may have up to 15,000 synapses
Cerebral cortex
80% of the brain in humans
Four lubes:occipital,temporal,parietal,frontal
Occipital
Vision
Temporal
Auditory processing & language;
declarative memory;
emotional processing;
visual recognition
Parietal
Spatial processing;
cross-modal integration
Frontal
Planning,inhibition,
"the executive
Neurogenesis
Complete 18 weeks gestation
Synaptogenesis
Formation of synapses
Time course, variation in areas
Decline
Synapse elimination
Synaptic pruning - Axon retreats, dendritic spine pruned away
Surplus - 6 mo. 2x as many synapses in visual cortex as adult
Continues through adolescence
Use it or lose it
Lateralization
Cerebral Hemispheres - crossed wires
Brain remains plastic for many years
Left Cerebral Hemisphere
language, logical analysis, sequential tasks
Right Cerebral Hemisphere
Spatial abilities, holistic tasks
Plasticity
Flexibility
EX:recover from brain dmg by recruiting other areas to take over function
Experience-Expectant processes
Brain cannot develop normally w/o these experiences
EX: language, Strabismus
Sensitive periods/critical pds
Experience-Dependent processes
Greenough's rats - enriched environments:bigger brains, smarter
Learning - happens throughout life
Myth of 1st 3 years
Expectant VS. Dependent
EE required for normal development to take place(visual stimulation, language exposure, emotional attachment)
ED NOT required for normal development to take place, but nonetheless change brain(2nd language, instrument)
Sensation
receiving basic info from the external world through the sensory receptors
Perception
process of organizing and interpreting sensory information
Visual acuity
sharpness of vision & clarity with which details can be discerned
Sinusoidal gratings
sine wave from above
higher freq. = lines closer together
Techniques for studying infants' visual abilities
Preferential looking
Habituation(color example)
Newborn Visual Acuity
20/400
Rapidly improves(nearly 20/20 by 8 months)
Why is vision so poor at birth?
Acuity is determined by packing of cones on fovea. Newborns' cones are less densely packed, not as efficient.
Object scanning of newborn
Focus on outside boundaries(higher contrast w/ background)
Depth cues
Optical expansion @ 3-4 weeks
Binocular cues & stereopsis @ 4 months(3D movies)
Pictorial/monocular cues @ 5-7 months
Acuity (nature vs nurture)
Probably not based on experience
Depth cues (nature vs nurture)
optical expansion very early(innate?)
Stereo vision is later but has critical period
Pictorial cues likely to be learned from experience
The Active Child (acuity)
poor early visual abilities restrict experience
depth perception related to crawling