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26 Cards in this Set

  • Front
  • Back
What are differentials for pruritis
infectious
ectoparasites
allergic
What are differentials for rash / pustules
pyoderma
eosinophil pustulosis
contact allergy/irritation
trauma
auto immune
What indicates high level of pruritis
Scratching in the exam room
what is diagnosis approach to pruritis
Derm data base:
skin scraping
trichogram
impression smear
fungal culture
cytology of pustule
What are primary factors of pruritis
-Allergic disease: atopy, food adversion, contact allergy, flea allergy
-Ectoparasites
-Inflammatory disease: autoimmune, infectious
-Disorders of cornification: seborrhea
What are predisposing factors to pruritis
Breed
age
endocrine disorder
environment temp and humidity
husbandry
What are perpetuating factors to pruritis
Bacterial infection (staph)
Malassezia
dermatophytes / fungus
Inappropriate treatment - excessive bathing
What type of skin is itchy
dry skin
heat
What is the threshold theory
-every animal has a certain amount of normal itch
-When factors are added above the threshold it becomes clinical
-Threshold is moveable
What causes CS to get worse
problems accumulate
more blocks
How do you manage pruritis
-Id and control perpetuating factors
-ID and treat primary factor
What are diagnostic keys
-Organized diagnostic and therapeutic approach to ID:
perpetuating factors
primary factors
predisposing factors
-good client ed
-keep accurate records
-Follow up exams are crucial
What do you do on intitial visit (Step 1)
Signalment
History
PE
What are keys to pick up in history
-Chronology of condition, age of onset, seasonal or non seasonal, progressive or static
-Severity
-effect of previous treatment
What do you look for on PE
-Evidence of pruritis
Alopecia
Excoriations
saliva staining
broken hairs
-look for causes of pruritis:
pyoderma
ectoparasites
-Inflammatory vs non inflammatory diseases
What are components of Derm data base
Skin scraping
impression smear - yeast
Trichogram
Fungal evaluation - culture preferred
What is treatment strategy
-ID perpetuating (2nd) factors
-Treat perpetuating (2nd) factors
-ID underlying (1) factors
What are optional tests
Fungal culture
fecal float
ear cytology
flea control (cats)
hypoallergenic diet - takes time to work
skin biopsy
What are possible perpetuating factors
-Malassezia
-Bacterial dermatitis - Staph
What are superantigens
Staph
activate CD4 T cells
act as virulence factors
not processed by APCs
What is the effect of SA in atopic dermatitis
trigger ready response in atopic dermatitis animals
What are key historical features for Malassezia
-severe, progressive pruritis
-partial response to steroids
-lick feet / rub face / perianal pruritis / otitis externa / wt loss
-Hx of allergy patient that was managed well but now relapsing
-Allergy patient that cannot be controlled
What are differentials for Intense pruritis
Scabies
Malassezia dermatitis
Flea allergy dermatitis
Familial seborrhea
combintions of severely mild-moderate pruritic conditions
What are PE findings with Malassezia
-severe pruritis
-erythema / scale / crust dermatitis
-progression to lichenified / hyperplastic / hyperpigmented skin
-alopecia due to pruritis
-lymphadenomegaly
-yeast otitis (60-80% cases)
Where are the Malassezia problem areas
-Interdigital - dorsal, ventral
-ventral neck
-Perianal / perivulvar
-axillary / inguinal
-lip folds
-base of claws
What are Malassezia diagnostics
-Derm data base
-Cytology
-Find underlying cause - alergy test, food trial