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58 Cards in this Set
- Front
- Back
Normal Axis
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upright QRS Lead I and aVF
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Left Axis Deviation
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Lead I UP aVF DOWN
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Right Axis Deviation
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Lead I DOWN aVF UP
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Irregular Rhythm, ventricular rhythm
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no P before each QRS
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AV block
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PR>200msec or P and NO QRS after
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LBBB
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QRS>120 no R in V1 tall R in I V5 and V6
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RBBB
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QRS>120 RSR' complex WIDE R in V1 QRS with S pattern in I V5 V6
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Long QT syndrom
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QTc>440 msec undiag. cong. disorder predisposes to VTACH
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Wave forms Q > 40 (or >1/3 QRS)
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= Old MI
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Wave forms T inverted
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Ischemia
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Chamber enlargement RAA hypertrophy
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P amp > 2.5 Lead I
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Chamber enlargement LAA
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P width Lead II > 120 msec
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Chamber enlargement LVH
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R in aVL+S in V3 >_24mm Men >_20 Women
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Chamber enlargement RVH
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rt axis dev and R wave V1.7mm
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Treat afib is ABCD
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Anticoag, BBB-rate, Cardioconvert-CCB, Digoxin
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Dilated cardiomyopathy
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Most common cardiomyopathy
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Whats req for Dx of Dilated CM
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LV dilation + Sys dysfx (Low EF)
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Cause of DCM
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most idiopathic, alcohol, wet beri beri, coxsackie, Chagas, parasites, cocaine myocarditis, doxorubicin, HIV AZT
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2 MCC of 2nd DCM
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ischemia and Long standing HTN
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HX/PE DCM
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gradual CHF, Cardiomegaly S3 gallop TVR and MVR
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Dx DCM
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Echocardiograph is Diagnostic
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ECG changes in DCM
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nonspecific ST-T , Low volt. QRS, Sinus Tachy, ectopy, LBBB common
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CXR of DCM
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enlarged, balloon like heart and Pulm congestion
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S3 Gallop signifies
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end of rapid ventricular filling in setting of fluid overload in DCM
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Tx of DCM
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stop OH, treat sx of CHF=diurectic, ACEIs, BB, Anticoags dec risk of thrombus, ICD if EF <35%
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Hypertrophic CM
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LVH cant relax and fill (diastolic dysfx)
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Hypertrophy frequently involves
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IV septum-->LV outflow obstruction and impaired ejection of blood
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IHSS
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idiopathic hypertrophic subaortic stenosis
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IHSS inheritance
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auto dominant 50% risk
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IHSS is MCC of
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sudden death in young, healthy athletes in US
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Other causes of Marked Hypertrophy of heart
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HT, AS
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HX of HCM
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asymptomatic, syncope, dyspnea, palpitation, angina, suddent cardiac death
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PE of HCM
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MR, sustained apical pulse, S4, Systolic ejection crescendo-decrescendo louder with dec preload (valsalva, squatting)
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obstruction in HCM made worse by
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inc. myocardial contractility or dec LV filling (exercise, valsalva, vasodilators, dehydration)
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S4 signifies
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stiff ventricle, and inc "atrial kick" assoc with HCM
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Dx of HCM
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Echocardiography is diagnostic shows THICKENED LV walls and dynamic obstruction of BF
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ECG changes of HCM
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LVH
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CXR changes of HCM
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LA enlargement from MR
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Tx of HCM
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BB first therapy for sx relief, CCB second line
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TX of HCM
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surg option for IHSS=dual chamber pacing, partial excision of myocard, ICD, avoid intense athletics
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Restrictive CM
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Dec elasticity of myocardium-->impair diastolic filling without significant systolic dysfunction (EF nl or slight dec)
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Causes of RCM
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infiltrative disease (sarcoid, hemochrom
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Hx/PE of RCM
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Left and Rt side HF, RHF sym (JVD, Peripheral edema) predominate
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DX of RCM
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CXR and Echo NON diagnostic, Cardiac Bx->show fibrosis or infiltrates
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ECG changes with RCM
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LBBB
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Tx of RCM
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symptoms, diuretics for fluid overload, vasodilators to dec filling pressure, Anticoags of no CI
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CHF is a clinical syndrome caused by
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inability of heart to pump enough blood for O2 needed by hrt and periph tissue
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CHF risk factors
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CAD, HTN, CM, valve disease, DB
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Systolic dysfx leading to HF
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dec EF by 50% and inc LVEDV
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Cause of syst. dysfx HF
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inadequate ventricular contractility or inc afterload
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Mechanism of HF in syst dysfx HF
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hrt compensates for low EF and inc PL through hypertrophy and ventricular dilation (FSlaw) then fail, lead to inc myocard work and worse sys fx
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Hx/PE systol HF
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dyspnea** MC and earliest sx, chronic cough, fatigue, PND, Cheyne-stokes, abd. fullness
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CHF is a
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CLINICAL Syndrome based on Sign and Sxs
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Stages of CHF
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ABCD
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A of CHF
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high risk for CHF NO structural or fx or SS
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B of CHF
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struct heart disease(MI hx, LVSys Dysfx, Valve disease) but NO sym of CHF
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C of CHF
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structure + sx (dyspnea, fatigue, dec exercise tol)
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D of CHF
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Marked sx of CJF despite max treatment
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