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22 Cards in this Set
- Front
- Back
What is Parkinson's Disease?
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Most common basal ganglia motor disorders
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What are the 3 subtypes?
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-Akinetic/Rigid
-Tremor-Dominant -Mixed |
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What is Akinetic/Rigid Parkinson's Disease?
-Prevalence -Characterized -Interferes w/ -Difficulty -Gait |
-Prevalance: apprx 50% of all cases
-Characterized: mm rigidity, shuffling, gait, dropping posture, rhythmic muscular tremors and mask-like facial expression -Interferes w/ voluntary and automatic movements -Difficulty sitting to standing -Gait: flexes posture, shuffling of feet, decreased/absent arm swing |
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What are the distinctive signs of Akinetic/Rigid Parkinson's?
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-Akinesia/hypokinesia
-Rigidity -"Freezing" during movement -Postural instability -Resting tremor -Nonmotor signs: depression, psychosis, Parkinson's dementia, autonomic dysfunction |
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What are some characteristics of rigidity?
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-Increased resistance to movement in all mm
-Output from nervous sys causes active mm contraction, directly increasing resistance to movement -Present during sleep |
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What is Akinesia?
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-Absence of movement
-Opposite from hypokinesia described as decreased movement -Parkinson's pts have less control over amt of force mm produce |
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What is Hypokinesia?
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-Decreased AROM and lack of automatic movements (facial expression, normal arm swing during walking)
-Related to decreased ability to control force output of mm |
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Parkinson's:
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-Prone to falls bc inability to generate adequate mm force quickly
-Postural corrections too slow to fix -Pts may abruptly ceases (freezing gait) |
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What are the stages of Ankinetic/Rigid Parkinson's Disease?
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Stage 1: Unilat signs and symp, typically mild tremor of 1 limb
Stage 2: Bilat signs, posture & gait affected, minimal disability Stage 3: Moderately severe generalized dysfunction, significant slowing of body movements, early impairment of equilibrium on walking or standing Stage 4: Severe signs, able to walk to limited extent, rigidity, bradykinesia (slowness of movement), unable to live alone Stage 5: Extreme weight loss, cannot stand or walk, requires constant nursing care |
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What are Visuoperceptive Impairments?
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-Pt difficulty moving past visual movement blocks EX. door way
-Markers placed on floor as cue to get to places -Associated w/ abnormal processing in termporal lobe visual identification pathway |
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What is postural Instability?
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-Extreme stiffness of postural flexors and extensors (severe orob)
-Clinical diag: hypokinesia affectign upper body combined w/ rigidity and/or resting tremor |
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What are the hyperkinetic signs for Parkinson's?
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-Resting tremor: involuntary, rhythmic shaking movements by contractions of antagonism
-Resting tremor of hands: rhythmic movement like thumb rolling pill along fingertips while rest and diminishes during voluntary mvmt (freq 3-6 tremor/sec) -May persist during sleep - |
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What are some disorders that Akinetic/Rigid Parkinson's associated w/ psychopathology?
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-Depression, psychosis (usually visual hallucinations), Parkinson's dementia, autonomic dysfunction (constipation, orthostatic hypotension) further decrease the person's independence
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What is dementia? What is the difference btwn Alzheimer's dementia and Parkinson's dementia?
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-Dementia: deterioration of intellectual function
-Alzeimer's: primarily affects memory -Parkinson's: interferes with the ability to plan, to maintain goal orientation, and to make decisions. |
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Tremor-dominant Parkinson's Disease:
Prevalence? Most disabling feat? Sign/Symp? |
-Prevalence: apprx 40% of all cases
-Most disabling feat: presence of both action (occur during voluntary movements EX. eating) and resting tremors -Sign/Symp: rigidity slowing of movement mild, tremors primary factor interfering w/ ADL |
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Mixed Parkinson's Disease:
Prevalence? Described as? |
-Prevalence: 10% of all cases
-Combo of both akinetic/rigid and tremor-dominant |
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What is the pathology of Parkinson's?
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-Death of dopamine-producing cells by oxidative stress, mitochondrial dysfunction and programmed cell death
-Cell death occurs long before clinical presentation of disease (80% of dopamine-producing cells die before signs of the disease appear) -Loss of dopamine reduce activity in motor areas of cerebral cortex (decreases voluntary movements) |
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Tx of Parkinson's?
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-Medications (L-dopa), invasive procedures, physical therapy and OT
-Dyskinesia (involuntary movement that resembles chorea – brisk, jerky movements), and/or dystonia (involuntary sustained postures or repetitive movements) even with L-dopa patient will experience bouts of normal mobility and bouts of immobility (ON-OFF PHENOMENON) -Deep Brain Stimulation (DBS) to treat dykinesia, tremors and hypokinesia, used as adjunt to meds, require surgical implantation of stimulator and electodes (stim: inf to clavicle, electrodes: thalamus to treat tremors, Continuous high frequency electrical stimulation inhibits the firing of overactive thalamic neurons) |
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DBS:
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-To treat dykinesia, tremors and hypokinesia
-Used as adjunt to meds -Require surgical implantation of stimulator and electodes (stim: inf to clavicle, electrodes: thalamus to treat tremors) -Continuous high frequency electrical stimulation inhibits the firing of overactive thalamic neurons) -Bilat DBS improves speed of movement, rigidity, gait, postural stability and decreases L-dopa dosage -Can only be used/administered to relatively healthy, young and cognitively intact pts |
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Assessment of Parkinson's Pt?
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HH #:
-ADL’s -Present symptoms and previous -Any other interventions? Physiotherapy? -The role they would like massage therapy to play? OBS: -Gait -Posture -Coordination of movements (gross and fine) -Balance -Facial expression -Spasticity OBJECTIVE: -AROM of affected jts (how move thru ROM) -PROM of affected jts --Mild spasticity – allows for gross movements and fine coordination will be clumsy or unable to perform --Moderate spasticity – allows for slow movements and abnormal coordination --Severe spasticity – limited spastic movement and active movements are impossible |
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Signs and Symp of Parkinson's Pt?
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-Often insidiously- slow movement, weakness and resting tremor
-Resting tremor affects distal segments of the limbs – mainly hands, feet, head, neck, face, lips and tongue/jaw -Rhythmic, alternating flexion and contraction movements (4-6/minute) -Usually unilateral at first and progresses to both sides of the body -Difficulty initiating walking and difficulty turning -Walking they lean forward to maintain center of gravity and take small steps without arm swing -Postural changes and gait disturbances continue to become more pronounced |
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MT Tx:
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-Contraindications: Deep & painful techniques may increase symptoms – start with lighter techniques & progress with depth of treatment
-Hydrotherapy: Deep moist heat work well (caution with the first application, testing sensation is important) -Remex: PROM to affected jts and compensatory jts, Diaphragmatic breathing to maintain thoracic mobility -Homecare: Relaxation exercises and diaphragmatic breathing, Appropriate hydrotherapy, Moderate and regular exercise program |