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22 Cards in this Set

  • Front
  • Back
What is Parkinson's Disease?
Most common basal ganglia motor disorders
What are the 3 subtypes?
-Akinetic/Rigid
-Tremor-Dominant
-Mixed
What is Akinetic/Rigid Parkinson's Disease?
-Prevalence
-Characterized
-Interferes w/
-Difficulty
-Gait
-Prevalance: apprx 50% of all cases
-Characterized: mm rigidity, shuffling, gait, dropping posture, rhythmic muscular tremors and mask-like facial expression
-Interferes w/ voluntary and automatic movements
-Difficulty sitting to standing
-Gait: flexes posture, shuffling of feet, decreased/absent arm swing
What are the distinctive signs of Akinetic/Rigid Parkinson's?
-Akinesia/hypokinesia
-Rigidity
-"Freezing" during movement
-Postural instability
-Resting tremor
-Nonmotor signs: depression, psychosis, Parkinson's dementia, autonomic dysfunction
What are some characteristics of rigidity?
-Increased resistance to movement in all mm
-Output from nervous sys causes active mm contraction, directly increasing resistance to movement
-Present during sleep
What is Akinesia?
-Absence of movement
-Opposite from hypokinesia described as decreased movement
-Parkinson's pts have less control over amt of force mm produce
What is Hypokinesia?
-Decreased AROM and lack of automatic movements (facial expression, normal arm swing during walking)
-Related to decreased ability to control force output of mm
Parkinson's:
-Prone to falls bc inability to generate adequate mm force quickly
-Postural corrections too slow to fix
-Pts may abruptly ceases (freezing gait)
What are the stages of Ankinetic/Rigid Parkinson's Disease?
Stage 1: Unilat signs and symp, typically mild tremor of 1 limb
Stage 2: Bilat signs, posture & gait affected, minimal disability
Stage 3: Moderately severe generalized dysfunction, significant slowing of body movements, early impairment of equilibrium on walking or standing
Stage 4: Severe signs, able to walk to limited extent, rigidity, bradykinesia (slowness of movement), unable to live alone
Stage 5: Extreme weight loss, cannot stand or walk, requires constant nursing care
What are Visuoperceptive Impairments?
-Pt difficulty moving past visual movement blocks EX. door way
-Markers placed on floor as cue to get to places
-Associated w/ abnormal processing in termporal lobe visual identification pathway
What is postural Instability?
-Extreme stiffness of postural flexors and extensors (severe orob)
-Clinical diag: hypokinesia affectign upper body combined w/ rigidity and/or resting tremor
What are the hyperkinetic signs for Parkinson's?
-Resting tremor: involuntary, rhythmic shaking movements by contractions of antagonism
-Resting tremor of hands: rhythmic movement like thumb rolling pill along fingertips while rest and diminishes during voluntary mvmt (freq 3-6 tremor/sec)
-May persist during sleep
-
What are some disorders that Akinetic/Rigid Parkinson's associated w/ psychopathology?
-Depression, psychosis (usually visual hallucinations), Parkinson's dementia, autonomic dysfunction (constipation, orthostatic hypotension) further decrease the person's independence
What is dementia? What is the difference btwn Alzheimer's dementia and Parkinson's dementia?
-Dementia: deterioration of intellectual function
-Alzeimer's: primarily affects memory
-Parkinson's: interferes with the ability to plan, to maintain goal orientation, and to make decisions.
Tremor-dominant Parkinson's Disease:
Prevalence?
Most disabling feat?
Sign/Symp?
-Prevalence: apprx 40% of all cases
-Most disabling feat: presence of both action (occur during voluntary movements EX. eating) and resting tremors
-Sign/Symp: rigidity slowing of movement mild, tremors primary factor interfering w/ ADL
Mixed Parkinson's Disease:
Prevalence?
Described as?
-Prevalence: 10% of all cases
-Combo of both akinetic/rigid and tremor-dominant
What is the pathology of Parkinson's?
-Death of dopamine-producing cells by oxidative stress, mitochondrial dysfunction and programmed cell death
-Cell death occurs long before clinical presentation of disease (80% of dopamine-producing cells die before signs of the disease appear)
-Loss of dopamine reduce activity in motor areas of cerebral cortex (decreases voluntary movements)
Tx of Parkinson's?
-Medications (L-dopa), invasive procedures, physical therapy and OT
-Dyskinesia (involuntary movement that resembles chorea – brisk, jerky movements), and/or dystonia (involuntary sustained postures or repetitive movements) even with L-dopa patient will experience bouts of normal mobility and bouts of immobility (ON-OFF PHENOMENON)
-Deep Brain Stimulation (DBS) to treat dykinesia, tremors and hypokinesia, used as adjunt to meds, require surgical implantation of stimulator and electodes (stim: inf to clavicle, electrodes: thalamus to treat tremors, Continuous high frequency electrical stimulation inhibits the firing of overactive thalamic neurons)
DBS:
-To treat dykinesia, tremors and hypokinesia
-Used as adjunt to meds
-Require surgical implantation of stimulator and electodes (stim: inf to clavicle, electrodes: thalamus to treat tremors)
-Continuous high frequency electrical stimulation inhibits the firing of overactive thalamic neurons)
-Bilat DBS improves speed of movement, rigidity, gait, postural stability and decreases L-dopa dosage
-Can only be used/administered to relatively healthy, young and cognitively intact pts
Assessment of Parkinson's Pt?
HH #:
-ADL’s
-Present symptoms and previous
-Any other interventions? Physiotherapy?
-The role they would like massage therapy to play?
OBS:
-Gait
-Posture
-Coordination of movements (gross and fine)
-Balance
-Facial expression
-Spasticity
OBJECTIVE:
-AROM of affected jts (how move thru ROM)
-PROM of affected jts
--Mild spasticity – allows for gross movements and fine coordination will be clumsy or unable to perform
--Moderate spasticity – allows for slow movements and abnormal coordination
--Severe spasticity – limited spastic movement and active movements are impossible
Signs and Symp of Parkinson's Pt?
-Often insidiously- slow movement, weakness and resting tremor
-Resting tremor affects distal segments of the limbs – mainly hands, feet, head, neck, face, lips and tongue/jaw
-Rhythmic, alternating flexion and contraction movements (4-6/minute)
-Usually unilateral at first and progresses to both sides of the body
-Difficulty initiating walking and difficulty turning
-Walking they lean forward to maintain center of gravity and take small steps without arm swing
-Postural changes and gait disturbances continue to become more pronounced
MT Tx:
-Contraindications: Deep & painful techniques may increase symptoms – start with lighter techniques & progress with depth of treatment
-Hydrotherapy: Deep moist heat work well (caution with the first application, testing sensation is important)
-Remex: PROM to affected jts and compensatory jts, Diaphragmatic breathing to maintain thoracic mobility
-Homecare: Relaxation exercises and diaphragmatic breathing, Appropriate hydrotherapy, Moderate and regular exercise program