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57 Cards in this Set
- Front
- Back
circulation in molluscs |
open, all have hearts some have bv some sinuses |
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crculation in arthropods |
crustacenas mostly open exc cephalopods insects open |
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what is unique about crustacean circulation |
some control of their hemolymph distribution (ostia) |
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what is unique about insect circulation |
separate tracheal system and multiple contractile hearts |
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movement of flagella circulation to pump water |
porifera |
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ciliated cells move institial flow by bulk flow |
platyhelminthes |
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use mmusclular contraction to move water |
cnidarians |
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what makes up the tunica intima |
endothelial cells (vascular) |
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what makes up tunica media |
smooth muscle and elastic connective tissue |
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what makes up tunica externa |
collagen |
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What are the 3 types of capillaries |
continuous, fenestrated and sinusoidal |
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Where are continuous capillaries found |
skin, muscle, CNS (BBB) |
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where are fenestrated capillaries found |
kidneys, endocrine, intestine |
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where are sinusoidal capillaries found |
liver and bone marrow (large proteins) |
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Arthropod Heart |
heart pumps hemoplymph out of arteries, comes back thru ostia suspended with ligaments neurogenic |
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cardiac cycle of arthropods |
neurons of cardiac ganglion spontaneos, vocontraction as normal, closing ostia valves, stretched ligaments pull open heart walls as blood leaves to increase volume |
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4 parts of the vertebrate heart wall |
pericardium, epicardium, myocardium, endocardium |
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Where do the para and sympa innervate the heart (what layer) |
epicardium |
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what are the 4 chambers of the heart |
sinus venosus, atrium, ventricle, bulbus arteriosus |
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contraction of fish heart |
sinus venosus atrium contracts ventricles contract (ventricle) into bulbus arteriosus, flows out |
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Amphibian heart |
3 chambers with trabeculae and spiral fold |
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mechanism of amphibian heart |
deoxy blood thru sinus venosus, goes into ventricle, up the to the conus arteriosus by picking the proper side of sspiral fold (right for pulomnary) - comes back in the pulmonary vein left atrium - back ventricle - up left side of fold |
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reptile hearts |
2 atria 3 ventricles (cavum venosum, cavum pulmonale, cavum arteriosum) |
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mechanism of reptile heart contraction |
pulmonary vein to cavum arteriosum, ridge shuts off cavum pulmonale (for deoxy), it must then go up the cavum venosumthen it goes up to the aortas. |
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what's weird about the reptile heart order of ventricles |
cavum venosum pumps into the aortas |
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right to left shunt |
deoxy bypasses reenters during breath holding |
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left to right shunt |
oxy blood reenters, aids oxygen delivery to myocardium |
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isovolumetric contraction |
both AV and aortic valves losed, occurs during the RS of the pqrs |
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atrial systole part of wave |
P wave |
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ventricular ejection part of wve |
T wave |
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What is the law of bulk flow |
Q=pressure change over resistance |
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Resistance equation |
8Ln/pie R4 |
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puiseuille's equn |
Q=change in P pi r^4/8Ln |
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What is fick's law of diffusion |
Diffusion rate equals concentration gradient times surface area times diffusion coefficient over distance travel |
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how does increasing heart rate work |
via the phosphorylating of Funny and T type Ca+ channels on autorhythmic cells (pacemakers) |
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What causes the plateau phase in extended action potentials |
Calcium entry via L type channels |
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what part of the EEG is the plateau phase |
the ST segment |
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wjat is the eqn for cardiac output |
heart rate times stroke volume |
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what is the frank starling effect |
the distance of overlap between thick and thin filaments has a greater rate of contraction at normal biological level. sympa activity shifts the graph up |
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what is chronotopy |
HR regulation by the rate of SA node depolarization (pae maker rate regulation) |
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what is inotropy |
nervous and endo contractile modulation by EC coupling (stroke volume regulation) |
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decreasing heart rate pathway |
medulla->para->Ach->muscarinic->Gi->deactivates T-type Ca++ because not phosphorylated, leaves K channels open (doesnt touch them) -- increases time for dep, decreases heart rate |
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NE on cardiomyocyte mechanism |
bind to b1->ac->camp->pka->->opens ca++ on SR ->phosphorylates myosin->phosphorylates ca++atpase (removal quickly)-> opens L type Ca++ which stimulates contraction (this is how there is a plateau phase |
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if norepenephrine increases Ca++atpase on the SR why is this stimulatory |
it speeds up removal from cytoplasm which means that there is less relaxation time (relaxing is the removal of it) |
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NE on cardiomyocyte RESULT |
increase in stroke volume |
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Mean arterial pressure equation |
CO x TPR |
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What is myogenic autoregulation? |
stretch sensitive smooth muscle cells in arterioles contract with an increase in BP (neg feedback) |
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what is the purpose of cntraction as an autoregulation in arterioles |
prevents excessive flow of blood into tissues |
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NE effect on arterioles |
vaso constriction |
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effect of decreased sympathetic tone on arterioles |
vasodilation |
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Vasopressin is released from and causes what |
posterior pituitary, vasoconstriction (generalized) (first order) |
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angiotensin II produced where and in response to what? |
in liver, in response to a decrease in blood pressure (constriction) |
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atrial natriuretic peptide |
from atria stretch cells, released during exercise to compensate for increase BP by causing vasodilation (natural big) |
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does para affect HR or SV or both and does Sympa affect one or the other or both? |
para only HR, sympa HR and SV |
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what are baroreceptors |
stretch sensitive mechanoreceptors esp in carotid arteries and aorta |
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where do baroreceptors send signals to |
medulla oblongota cardiovascular control center |
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baro receptor reflex regulates MAP by...? |
high map causes baro to fire more, which go to medulle, which decrease sympa output and NE release, which casuses vasodilation of muscles (decrease in peripheral resistance) and decrease force of contraction (myocardium-SV) and decrease cardiac output which is hrxsv and also decrease HR by SA node (less stimulated by epi) |