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57 Cards in this Set

  • Front
  • Back

circulation in molluscs

open, all have hearts some have bv some sinuses

crculation in arthropods

crustacenas mostly open exc cephalopods


insects open

what is unique about crustacean circulation

some control of their hemolymph distribution (ostia)

what is unique about insect circulation

separate tracheal system and multiple contractile hearts

movement of flagella circulation to pump water

porifera

ciliated cells move institial flow by bulk flow

platyhelminthes

use mmusclular contraction to move water

cnidarians

what makes up the tunica intima

endothelial cells (vascular)

what makes up tunica media

smooth muscle and elastic connective tissue

what makes up tunica externa

collagen

What are the 3 types of capillaries

continuous, fenestrated and sinusoidal

Where are continuous capillaries found

skin, muscle, CNS (BBB)

where are fenestrated capillaries found

kidneys, endocrine, intestine

where are sinusoidal capillaries found

liver and bone marrow (large proteins)

Arthropod Heart

heart pumps hemoplymph out of arteries, comes back thru ostia


suspended with ligaments


neurogenic

cardiac cycle of arthropods

neurons of cardiac ganglion spontaneos, vocontraction as normal, closing ostia valves, stretched ligaments pull open heart walls as blood leaves to increase volume

4 parts of the vertebrate heart wall

pericardium, epicardium, myocardium, endocardium

Where do the para and sympa innervate the heart (what layer)

epicardium

what are the 4 chambers of the heart

sinus venosus, atrium, ventricle, bulbus arteriosus

contraction of fish heart

sinus venosus atrium contracts ventricles contract (ventricle) into bulbus arteriosus, flows out

Amphibian heart

3 chambers with trabeculae and spiral fold

mechanism of amphibian heart

deoxy blood thru sinus venosus, goes into ventricle, up the to the conus arteriosus by picking the proper side of sspiral fold (right for pulomnary) - comes back in the pulmonary vein left atrium - back ventricle - up left side of fold

reptile hearts

2 atria 3 ventricles (cavum venosum, cavum pulmonale, cavum arteriosum)

mechanism of reptile heart contraction

pulmonary vein to cavum arteriosum, ridge shuts off cavum pulmonale (for deoxy), it must then go up the cavum venosumthen it goes up to the aortas.

what's weird about the reptile heart order of ventricles

cavum venosum pumps into the aortas

right to left shunt

deoxy bypasses reenters during breath holding

left to right shunt

oxy blood reenters, aids oxygen delivery to myocardium

isovolumetric contraction

both AV and aortic valves losed, occurs during the RS of the pqrs

atrial systole part of wave

P wave

ventricular ejection part of wve

T wave

What is the law of bulk flow

Q=pressure change over resistance

Resistance equation

8Ln/pie R4

puiseuille's equn

Q=change in P pi r^4/8Ln

What is fick's law of diffusion

Diffusion rate equals concentration gradient times surface area times diffusion coefficient over distance travel

how does increasing heart rate work

via the phosphorylating of Funny and T type Ca+ channels on autorhythmic cells (pacemakers)

What causes the plateau phase in extended action potentials

Calcium entry via L type channels

what part of the EEG is the plateau phase

the ST segment

wjat is the eqn for cardiac output

heart rate times stroke volume

what is the frank starling effect

the distance of overlap between thick and thin filaments has a greater rate of contraction at normal biological level. sympa activity shifts the graph up

what is chronotopy

HR regulation by the rate of SA node depolarization (pae maker rate regulation)

what is inotropy

nervous and endo contractile modulation by EC coupling (stroke volume regulation)

decreasing heart rate pathway

medulla->para->Ach->muscarinic->Gi->deactivates T-type Ca++ because not phosphorylated, leaves K channels open (doesnt touch them) -- increases time for dep, decreases heart rate

NE on cardiomyocyte mechanism

bind to b1->ac->camp->pka->->opens ca++ on SR ->phosphorylates myosin->phosphorylates ca++atpase (removal quickly)-> opens L type Ca++ which stimulates contraction (this is how there is a plateau phase

if norepenephrine increases Ca++atpase on the SR why is this stimulatory

it speeds up removal from cytoplasm which means that there is less relaxation time (relaxing is the removal of it)

NE on cardiomyocyte RESULT

increase in stroke volume

Mean arterial pressure equation

CO x TPR

What is myogenic autoregulation?

stretch sensitive smooth muscle cells in arterioles contract with an increase in BP (neg feedback)

what is the purpose of cntraction as an autoregulation in arterioles

prevents excessive flow of blood into tissues

NE effect on arterioles

vaso constriction

effect of decreased sympathetic tone on arterioles

vasodilation

Vasopressin is released from and causes what

posterior pituitary, vasoconstriction (generalized) (first order)

angiotensin II produced where and in response to what?

in liver, in response to a decrease in blood pressure (constriction)

atrial natriuretic peptide

from atria stretch cells, released during exercise to compensate for increase BP by causing vasodilation (natural big)

does para affect HR or SV or both and does Sympa affect one or the other or both?

para only HR, sympa HR and SV

what are baroreceptors

stretch sensitive mechanoreceptors esp in carotid arteries and aorta

where do baroreceptors send signals to

medulla oblongota cardiovascular control center

baro receptor reflex regulates MAP by...?

high map causes baro to fire more, which go to medulle, which decrease sympa output and NE release, which casuses vasodilation of muscles (decrease in peripheral resistance) and decrease force of contraction (myocardium-SV) and decrease cardiac output which is hrxsv and also decrease HR by SA node (less stimulated by epi)