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30 Cards in this Set

  • Front
  • Back
chronic airflow limitation, destruction of alveoli, alveolar walls, non-reversible
COPD
Pt PW:
anorexia
well-defined accessory resp ms,
barrel-shaped chest,
flat diaphragm
BS: decreased, crackles, wheezes
RR: increased at rest
COPD
cor pulmonale
-when present?
-what is it?
hypertrophy of right ventricle-overworking to pump blood into lungs.

present in COPD
chronic bronchitis=
inflammation of bronchi for 3 mos over 2 consecutive years
emphysema:
destruction of air spaces at ends of terminal bronchioles, resulting in changes in alveoli and reduction in their numbers.
COPD =

_____+______
chronic bronchitis+ emphysema
chest tightness
weight loss
anorexia
hemoptysis
chest wall changes
clubbing
cyanosis
postures
BS decreased
BS crackles
BS wheezes
increased RR at rest
prolonged exhalation w/pursed lips
COPD
COPD trends compared to normal:

TLC:
FRC (ERV + RV)
RV
VC
FEV1
FEV1/FVC
TLC incr
FRC: incr
RV-incr
VC-usually decreases
FEV1 decreases
FEV1/FVC decreases
treatment for COPD:
-medication
anticholinergic (bronchodilator by mimicking parasympathetic NS), antibiotics, rescue drug for breakthrough symptoms, supplemental O2
treatment of COPD:
removal of exposure:
smoking cessation
treatment of copd
secretion removal:
Active Cycle Breathing/flutter/PEP
manual techniques
treatment of copd:
breathing exercises:
pacing techniques, IMT
treatment of copd:
aerobic conditioning
mode, intensity, duration, frequency
a chronic obstructive pulmonary disease with respiratory disturbance (most severe), pancreatic insufficiency, and GI disturbance. diagnosed by sweat test
CF
medication for CF:
antibiotics, mucolytics, rescue drugs, pancreatic enzyme replacements, supp o2
Pt PW:
resp disturbance,
frequent resp infections
increased RR, effort, prolonged exh.
meconium ileus
failure to thrive
fatty stools
glucose intolerance
flat diaphragm,
cor pulmonale
Disease present in entire lung
CF
chronic inflammatory disorder of the airways:
eosinophilic inflammation chronically present.

increased reactivity of the trachea and bronchi to various stimuli (incl. dust, mold, perfume, animal dander, cold, URI)
asthma
bronchial smooth muscle constriction + increased airway secretion + airway inflammation
asthma
patient PW:
wheezing
breathlessness
chest tightness
cough
particularly at night and early morning
particularly in response to antigen
reversible in nature (measure per PFTs)
asthma
physical findings include:
BS: adventitious, decreased
HR incr
RR incr
accessory ms use
Pa02 decr
paCo2: decr then incr
asthma
asthma tx:
medication
rescue meds

inhaled steroids for stage 2+
long acting b2 for stage 3+
use of stabilizing drug for EIB
step 4: inhaled steroid, long acting beta 2 and leukotriene
asthma tx:
secretion removal techniques:
autogenic drainage
ACBT

DO NOT AVOID PHYSICAL ACTIVITY
bronchiectasis
bronchi have abnormal dilation: action of cilia decreased; aren't able to mobilize secretions out of bronchi
tx of bronchiectasis
secretion removal:
flutter/acapella, PEP, ACBT, autogenic, manual techniques
caused by loss of heat, water, or both from lungs from hyperventilation of cooler drier air during exercise
EIB
lung immaturity, alveolar collapse, surfactant deficiency
grunting, nasal flaring,
xray: ground glass appearance
Respiratory distress syndrome
results from intervention of RDS (ventilation support, etc)
bronchopulmonary dysplasia
lung tissue fibrosed down; don't have same respiratory capacity.
pt pw: crackles, dry cough, decr excursion, decr o2
restrictive parenchymal disease
PT intervention for restr parenchymal disease:
support o2 needs during exercise
IMT
energy saving techniques
rote breathing ex for times of distress
restrictive disease, resp ms don't work
ALS