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48 Cards in this Set

  • Front
  • Back
pre-term labor
uterine contractions and cervical changes between 20-37 weeks
cause most neontal deaths
preterm birth
before 37 weeks gestation
low birth weight
2500 grams or less
risks for pre-term labor
minorities
low socioeconomic status
unmarried
low levels of education
late/no prenatal care
age
multifetal pregnancy
* at least 50% of women who experience pre-term birth have no identifiable risk factors
fetal fibronectin
glucoproteins in plasma and cervical canal
appearance between 24-34 weeks predicts labor
salivary estriol
form of estrogen
levels increase with pre-term birth
endocervical length
shortened cervix - 30 mm or less at 34 weeks
symptoms of PTL
uterine activity
discomfort
vaginal discharge
dx of PTL
20-37 weeks
contractions
progressive cervical change (dilation of greater than 4 cm)
tocolytics
suppress uterine activity - relaxes smooth uterine muscles
brethine/terbutaline, MgSO4, ritodrine, indocin, procardia
antenatal gluccocorticoids
accelerate fetal lung maturity
24-34 weeks if preterm birth is a threat
bethamethasone, dexamethasone
24 hours to be effective
premature rupture of membranes
at least 1 hr before onset of labor
any gestational age
sudden gush or slow leakage
preterm premature rupture of membranes
before 37 weeks
25% of all PTL cases
infection often precedes
test for ROM
nitrazine, ferning tests
sterile vaginal exam only when necessary
complications of ROM
cord compression and prolapse
monitor for signs of infection/fetal distress
dystocia
long, difficult, abnormal labor
primary cause of c-section
factors leading to dystocia
dysfunctional labor
alterations in pelvic structure
fetal causes
maternal positioning
psychologic response
primary powers (Uterine contractions) - dystocia
abnormal UC
hypertonic uterine dysfunction(frequent, painful, ineffective UC)
hypotonic uterine dysfunction (weak and ineffective UC)
secondary powers - dystocia
abnormal pushing efforts
decreased effectiveness of bearing down
abnormal labor patterns
prolonged latent phase
protracted active phase dilation
secondary arrest/no change
protracted descent
arrest of descent
failure of descent
causes of abnormal labor patterns
ineffective UC, pelvic contractions, cephalopelvic disproprotion, abnormal fetal presentation or position, early use of analgesics, nerve block, anxiety, stress
risks of abnormal labor patterns
maternal - morbidity and death from uterine rupture, infection, severe dehydration, postpartum hemorrhage
fetal - increased risk of hypoxia
precipitous labor
rapid or sudden labor - less than 3 hours
complications of precipitous labor
maternal - uterine rupture, lacerations of the birth canal, amniotic fluid embolism, postpartum hemorrhage
fetal - hypoxia, intercranial trauma
pelvic dystocia
decreased capacity of bony pelvis
soft tissue dystocia
obstruction of birth passage with anatomic abnormality, other than pelivs (placenta previa, tumor, full bladder/rectum)
fetal causes -dystocia
anomalies
cephalopelvic disproportion
malposition (occiput posterior)
malpresentation (breech)
multifetal pregnancy
complications
maternal position
alter functional relationship of UC, fetus, pelvis
"effects of gravity" - find a position that facilitates fetal descent (sit, stand, squat)
psychologic response
fear, anxiety, stress can have negative impact
hormones and nuerotransmitters released with stress and anxiety can negativity impact labor
external version
after 37 weeks - can cause pre-term labor
tocolytics to relax uterus
US and NST
induction of labor
any condition putting mother/baby at risk
sucess rate is better when cervix is favorable
bishop score - 9 if multipara, 5 if nullipara
cervical ripening
prostagladins
mechanical dilators
amniotomy
when cervix is ripe and infant engaged
must deliver afterward
oxytocin
hormone to stimulate UC - risk of hyperstimulation
augmentation of labor
stimulating UC that are weak or ineffective
common management for hypotonic uterine dysfunction (protracted active phase)
methods of augmenting labor
noninvasive - empty bladder, ambulation, relaxation, hydrotherapy
invasive - oxytocin, amniotomy, nipple stimulation
operative delivery
forceps
vacuum assisted
both to aid in descent and shorten 2nd stage of labor
higher rate of c-section due to
repeat c-section
EFM
epidurals
first time labor + advanced maternal age
decline VBAC rates
uterine incisions
classic, vertical
low-uterine transverse
VBAC
depends on reason for first c-section
contraindications- fundal scar, scar from uterine surgery, evidence of CPD
post-term pregnancy
beyond 42 weeks
risks - large baby, induction, aging placenta, hypoxia, decreased AFV, meconium
shoulder dystocia - what and risks
emergency
related to macrosomia , pelvic abnormalities
risks - asphyxia, brachial plexus damage, fracture + maternal blood loss, uterine rupture, laceration
shoulder dystocia interventions
suprapubic (not fundal) pressure
maternal position change (hands/knees, squat)
mcroberts maneuver - legs flexed apart with knees on abdomen
umbilical cord prolapse
occult (visible), frank (not visible)
occlusion of blood flow for +5 minutes = fetal CNS damage, death
causes - AROM, small baby, polyhdramnion
umbilical cord prolapse interventions
manually hold presenting part off cord
modified sims, trendelenberg, knee chest
c-birth
uterine rupture causes
scar seperation of classic c-birth incision, uterine trauma, congenital uterine anomaly, hypertonic stimulation, multiple gestation, malpresentation, external/internal version
complete or incomplete
uterine rupture complications
loss of FHR, internal bleeding = shock
amniotic fluid embolism
amniotic fluid + debris enter maternal circulation, obstruct pulmonary vessels
respiratory distress/circulatory collapse
clotting and DIC