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44 Cards in this Set
- Front
- Back
Factors that cause decease |
Genetic Environmental Anatomic |
|
Male chromosome |
XX |
|
Female chromosome |
XY |
|
Causal risk factors |
Direct causes the decease |
|
Non casual |
Does not cause decease directly but facilitates it |
|
Prevelence |
Number of cases in a period |
|
Morbidoty |
The presence of decease |
|
Mortality |
Number of deaths |
|
Familiar tendency |
Cluster of deceases in family groups despite lack of evidence |
|
Autosomal recessesive |
Must inherit two copies of a particular gene |
|
Autosomal dominant: |
Needs to only inherit one copy of a particular gene |
|
Allergies |
Acquired following intial exposure ( second exposure ) |
|
Asthma |
Chronic inflamatory condition |
|
Cancer |
-Large number of malignant growths Prognosis depends on extent of spread and effectiveness of treatment. -lung Leading cause of death due to camcer |
|
Colorectal |
Secon most comon type of cancer |
|
Endocrine dossorder. |
Ketoacidosis (type 1 diabetics) No Ketoacidocis ( type 2 |
|
Hemophelia can only be gotten by |
Males |
|
Hemochromatosis |
Body absorbs more iron than needed |
|
Long qt sundrome |
Cardiac consuction system abmormality |
|
Cardiomyopathy |
Deceases of the myocardium . Leads to heart failure, heart attack, death |
|
coronary heart decease |
Atherosclerosis Arteriosclerosis. Caused by impaired circulation to the heart |
|
Gout |
Abnormal acumulation of uric acid. (Causes destructive tissue change ) |
|
Kidney stone |
Masses of uric acid or calcium salts. Form in urinary system |
|
Lactose intolerance |
Defficiecy in Enzymes that break down lactose. (Lactase) |
|
Ulcerative colitis |
Chronic inflamatory disease |
|
Pelptic ulcers deceace |
Circumscribe erosions in the lining of the gi track |
|
Gallstones |
Stone like masses in the gallbladder |
|
Obesity |
Unhealthy accumulation of body fat |
|
Huntington decease |
Characterized by progresive chorea and progresive decease |
|
Alzheimers deceace |
Cortical atrophy |
|
Schizophrenia: |
Group of mental disorders |
|
Hypoperfusion |
Release of catecholamines Activation of RAAS Raise blood pressure. Release inti diuretic hormone Fluid shifts from intestinal |
|
Shock |
Hypoperfusion |
|
Central shock |
Cardiogenic-pump,container, contents. Obstructive- cardiac tamponade -tensions pneumo -chf |
|
Peropheral shock |
-Hemoragic (Exogenous) -Non Hemoragic (Endogenous) |
|
Peripheral-Distributive |
Anaphykactic Septoc Neurogenic |
|
Myltiple organ dysfunction system |
Primary MODS- direct injury Secondery MODS- progresive organ dysfunction |
|
MODS occur when |
Occurw when injury or infection triggers massive systemic immune response |
|
Two tyoes of anatomic barriers |
Mechanical and chemical Mech-skin ,hair ect Skin- barriers |
|
. |
. |
|
Leukocites |
Page 373 |
|
For the body to react what must happen first |
Primary exposure |
|
Secondary response is followed by |
First exposure to antigen |
|
Page 374 |
Figure 31 |