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15 Cards in this Set
- Front
- Back
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WHAT ARE FACTORS AFFECTING WOUND HEALING
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*AGE-HEALTHY KIDS AND ADULTS HEAL QUICKER THAN OLDER PEOPLE
*NUTRITIONAL STATUS-MALNOURISHED CLIENTS REQUIRE MORE TIME *LIFESTYLE-PEOPLE WHO EXCERCISE REGURALLY HAVE GOOD CIRCULATION *MEDICATION=ANTIINFLAMMATORY DRUGS AND ANTINEOPLACTIC AGENTS INTERFERE WITH HEALING AND PROLONG USE OF ANTIBIOTICS |
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RISK FACTORS CONTRIBUTING TO THE FORMATION OF PRESSURE ULCERS
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IMMOBILITY-REDUCTION IN THE AMOUNT AND CONTROL OF MOVEMENT A PERSON HAS\
INADEQUATE NUTRITION-CAN CAUSE WEIGHTLOSS MUSCLE ATROPHY AND LOSS OF SUB Q TISSUE FECAL AND URINARY INCONTINENCE DECREASED MENTAL STATUS DIMINSHED SENSATION-STROKE, PARALYSIS EXCESSIVE BODY HEAT ADVANCED AGE CHRONIC MEDICAL CONDITIONS-DIABETES CARDIO etC |
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STAGES OF PRESSURE ULCER FORMATION
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STAGE 1=NONBLANCHABLE INTACT SKIN RED
STAGE 2=PARTIAL THICKNESS SKIN LOSS, ABASION, BLISTER, CRATER STAGE 3-FULL SKIN LOSS INVOLVING DAMAGE OR NECROSIS OF SUB Q TISSUE EXTENDED DOWN BUT NOT THROUGH STAGE 4-FULL THICKNESS SKIN LOSS WITH TISSUE NECROSIS OR DAMAGE TO MUSCLE BONE OR SUPPORTING STRUCTURES, LIKE A TENDON OR JOINT CAPSULE |
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COMPLICATIONS OF WOUND HEALING
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HEMMORAGE-MASSIVE BLEEDING
INFECTION-WHEN MICROORGANISMS COLONIZING THE WOUND MULTIPLY EXCESSIVELY OR INVADE TISSUES, INFECTIONS OCCUR DEHISCENSE-PARTIAL OR TOTAL RUPTURE OF A SUTURED WOUND EVISCERATION-PROTRUSION OF THE INTERNAL VISCERA THROUGH AN INCISION |
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IMPAIRED SKIN INTEGRITY IS A THREAT TO WHT TYPE OF PEOPLE
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*CIENTS WITH RESTRICTED MOBILITY
*CHRONIC ILLNESS OR TRAUMA INVASIVE HEALTH CARE PROCEDURES |
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WHAT ARE PRESSURE ULCERS
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BEDSORES, DECUBITUS ULCERS OVER BONY PROMINENCES
A PRESSURE ULCER IS ONLY A LESION CAUSED BY UNRELIEVED PRESSURE THAT RESLUT IN DAMAGE TO THE UNDERLYING TISSUE |
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ETIOLOGY OF PRESSURE ULCERS
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ISCHEMIA(LOCALIZED)-DEFICIENCY IN BLOOD SUPPLY TO TISSUE CELLS ARE DEPRIVED OF O2 AND NUTRIENTS TISSUE MAY DIE BECAUSE OF ANOXIA
REACTIVE HYPEREMIA-WHEN PRESSURE IS RELIEVED AND SKIN TURNS BRIGHT RED FLUSH FRICTION-A FORCE ACTING PARALLEL TO SKIN SURFACE , SHEETS RUBBING SHEARING FORCE-A COMBO OF FRICTION AND PRESSURE OCCURS COMMONLY IN FOWLERS POSITION |
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INTACT SKIN IS
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PRESENCE OF NORMAL SKIN LAYERS UNINTERRUPTED BY WOUNDS
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APPEARANCE OF SKIN AND SKIN INTEGRITY ARE INFLUENCED BY WHAT EXTERNAL FACTORS
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GENETICS
AGE UNDERLYING HEALTH OF INDIVIDUAL AS WELL AS EXTERNAL FACTORS LIKE ACTIVITY |
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WHAT IS REGENERATION
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RENEWAL OF TISSUES
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WHAT ARE 2 TYPES OF WOUND HEALING
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PRIMARY INTENTION HEALING-OCCURS WHERE THE TISSUE SURFACES HAVE BEEN APPROXIMATED(CLOSED) AND THERE IS MINIMAL OR NO TISSUE LOSS(SURGICAL INCISION)
SECONDARY INTENTION HEALING=A WOUND THAT IS EXTENSIVE AND INVOLVES CONSIDERABLE TISSUE LOSS, EDGES CANNOT BE APPROXIMATED (PRESSURE ULCERS |
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NAME THE 3 PHASES OF WOUND HEALING
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INFLAMMATORY
PROLIFERATIVE MATURATION/REMODELING |
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DESCRIBE THE INFLAMMATORY PHASE OF WOUND HEALING
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INFLAMMATORY PHASE IS INITIATED IMMEDIATELY ATER INJURY AND LAST 3-6 DAYS 2 MAJOR PROCESSES OCCUR
HEMOSTASIS-CESSATION OF BLEEDING PHAGOCYTOSIS-MACROPHAGES ENGULF MICROORGANISMS AND CELLULAR DEBRIS |
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DESCRIBE THE PROLIFERATIVE PHASE OF WOUND HEALING
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THE SECOND PHASE IN HEALING EXTENDS FROM DAY 3 OR 4 TO ABOUT DAY 21 POST INJURY
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DESCRIBE THE MATURATION PHASE OF WOUND HEALING
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BEGINS ABOUT DAY 21 AND CAN EXTEND 1 OR 2 YEARS AFTER INJURY FIBRBLAST CONTINUE TO SYNTHESES COLLAGEN DURING THIS STAGE THE WOUND IS REMODELED AND CONTRACTERD
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