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62 Cards in this Set

  • Front
  • Back
If a pts rhythm strip shows the R waves are 4 large boxes between complexes, what is the heart rate?
75

(300, 150, 100, 75, 60, 50...)
The rhythm strip on the bottom of a EKG is how long?
12 sec

(3 sec each lead on other leads)
What is the rate range for normal sinus rhythm?
60-100
Define sinus bradycardia.
Heart rate less than 60, regular, with beats having P-QRS-T
What is the rate range for ventricular focus induced rhythms?
20-40 beats per minute
The malfunction of what organ system is the usual cause of multifocal atrial tachycardia?
pulmonary
The difference between multifocal atrial tachycardia and wandering atrial pacemaker is decided by:
WAP rate 100

MAP rate greater 100
What is the maximal normal QRS width?
0.12

normal 0.06-0.11
Which atrial dysrhythmia usually occurs between an atrial rate of 250-350?
Flutter 250-350

Firbrillation 350-450
In atrial fltutter, what is the most common form of AV block?
AV node block (usually 2:1)
Whick of the irregularly irregular rhythms is due to many causes and is the most common to occur?
atrial fibrillation
What electrical finding on the EKG signifies pacer induced rhythm?
electrical spike that is too narrow to be physiologically normal
What signs help separate ventricular tachycardia from supraventricular with bundle branch block?
Brugada's sign: Vtach
What signifies first degree heart block?
PR greater than 0.2 sec
What signifies that the second degree block is Mobitz 1?
progressive lengthing of the PR intervals until a ventricular beat is dropped (**99+% of the time)
Describe the atrial and ventricular rates in third degree AV block.
atrial rate: 

ventricular rate: 20-40
What happens to the timing of the following beat, post PVC?
compensatory pause

(PACs do not have a compensatory pause)
In what leads do you expect the P waves to always be positive?
+ P in leads 1, 2, V4-6

(-P in lead aVR)
What is the difference between WPW pattern WPW syndrome?
WPW Pattern
short PR interval
wide QRS
delta wave
ST-T wave changes
     WPW Syndrom: all above + proxysmal tachycardia
How does the PR interval change in Wenkebach block versus Mobitz 2?
Wenkebach (Mobitz 1) AV node has long refractory period- progressive legnthing until dropped QRS

Mobitz 2 (more dangerous) PR intervals constant until sudden dropped beat
Define complete heart block.
complete dissociation of Ps and QRSs

always more Ps than QRSs
Define a Q wave.
first negative deflection after the P wave
Define a significant Q wave.
0.03 sec or wider
depth equal to or greater than 1/3 R wave
What do Q waves in two or more adjacent leads mean?
MI

(need ST changes to dx ACUTE MI)
What is the normal QRS duration?
0.06-0.11
In which leads does one expect the usual transition zone to occur?
transition zone= where precordial leads change from neg to pos.

usually between V3 and V4
Extremely low voltage of every wave in every lead could indicate which 2 possible conditions?
MI (scar tissue is electrically inert)
marked increase body fat
huge pleural EFFUSION
pericardial EFFUSION
recording at half standard
What condition precludes the diagnosis of left ventricular hypertophy?
LBBB

only dx with normal QRS morphology
What conditions can cause a QRS duration of great than .11 sec width?
LBBB
RBBB
IV conduction delay
    (don't look like LBBB or RBBB)
WPW
What metabloic emergency can cause IVCD?
hyperkalemia
How wide does the QRS have to be before you can diagnose at bundle branch block?
at least one lead with QRS > 0.12
If the axis of an EKG is "normal," which 2 EKG leads would be expected to be positive?
lead 1 and aVF
Which bundle by definition is always involved in bifascular blocks?
RBBB

(+ one of the hemi's)
What diagnosis can never be made in the presence of a Left Bundle Branck Block?
LVH

(left ventricular hypertrophy)
What is the typical QRS appearance in RBBB?
RSR`
 "rabbit ears"
What is the typical QRS appearance in LBBB?
discordant T waves (opposite direction)
small R in V1

"uglygram"
What axis would you expect with a left anterior hemiblock?
vector is superior an leftward
axis: -30 to -90 deg

lead 1: qR or R 
lead 3: rS
In determining the vector of the ORS, what is the characteristic of the isoelelctric lead even if R wave size is a different amplitude than the S wave?
the axis is +/- 20deg from the axis of the isoelectric lead
What is the hallmark of acute transmural MI?
pathologic Q wave (wider than 0.03, or greater than 1/3 depth of R wave)
What is the hallmark sign of acute ischemia?
ST depression and T wave opposite to QRS
What is the EKG clue for Arythmogenic Right Ventricular Cardiomyopathy?
R ventricular inherited desmosome problem= SUDDEN DEATH

epsilon wave-small waves at the J point
What is the QTC?
QT interval corrected for heart rate

QTC = QT + 1.75X
normal up to 0.419sec
In a normal EKG what can be stated about the T and U waves?
normal: T and U always in same direction

bad (ischemia til proven otherwise): T and U opposite
Why do normal T waves occur in the same direction as the QRS?
Perkinje system is endocardial-> depolarization is endo- to epi-. Repolarization is opposite because ap duration is longest in the epicardium. Depolarization going toward electrode and repolarization going away from electrode both are positive!
T wave shape should always be:
asymetrical
What causes should you consider in significant T wave elevation?
acute MI
hyperkalemia

(greater than 6mm in limb leads or 12 mm in precordial)
(T 2/3 height of the R)
what is the difference on EKG between LVH and LVH with strain?
LVH with Strain- asymmetrical ST depression
What ST changes are expected in an EKG of a patient with pericarditis?
PR depression
diffuse ST changes without reciprocal ST depression
scooping- upward slope of ST
Notching at end of QRS
What is the relationship of the T wave to the QRS in a Bundle Branck Block?
LBBB- discordant T waves (opposite QRS
What is meant by the term "reciprocal changes" in an EKG and in what pathological state is it relevant?
lead opposite the damaged area show opposite changes.

ST elevation in one side, ST depression in the opposite
Which ion essentially sets the non-pacemaker myocytes resting membrane potential?
potassium
What happens to the cell membrane electrically to cause an action potential?
influx of Na+
What is differnct about phase 4 action potentials in pacemaker cells and what ion and channel is chiefly responsible?
Na+ and Ca++ leak channels allow electrical potential to slowly increase during phase 4
What is the name for the potential difference across the membrane reqquired to maintain the concentraiton gradient?
equilibrium potential
What phase of the action potential is responsible for repolarization and what ion is most responsible?
phase 1

transient K channel (K+)
also Ca++ entering thru slow Ca++, fast Na+ channels are inactivated
Which ion is predominantly responsible for the plateau phase of an action potential?
phase 2

slow Ca++ chanels stay open- creates electroneutrality
What phase/s of the action potential found in myocardial muscle cells is missing from pacemaker cells?
NOT phase 1 and 2

Pacemaker cell phases: 0, 3, 4
What is the funciton of the fast Na+ channel in phase 0 of pacemaker cells?
depolarization
What two mechanisms are responsible for Phase 0 in pacemaker cells?
L type Ca++ channels

funny Na channel (If)
What EKG abnormality would you expect to demonstrate with severe aortic stenosis?
LVH or LBBB
What EKG abnormality would be most likely in severe mitral stenosis?
A-fib or P mitrale (P>0.12 and notched)
What EKG abnormalities would you expect in severe chronic mitral regurgitation?
P mitrale (P>0.12 and notched)