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62 Cards in this Set
- Front
- Back
If a pts rhythm strip shows the R waves are 4 large boxes between complexes, what is the heart rate?
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75
(300, 150, 100, 75, 60, 50...) |
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The rhythm strip on the bottom of a EKG is how long?
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12 sec
(3 sec each lead on other leads) |
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What is the rate range for normal sinus rhythm?
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60-100
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Define sinus bradycardia.
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Heart rate less than 60, regular, with beats having P-QRS-T
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What is the rate range for ventricular focus induced rhythms?
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20-40 beats per minute
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The malfunction of what organ system is the usual cause of multifocal atrial tachycardia?
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pulmonary
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The difference between multifocal atrial tachycardia and wandering atrial pacemaker is decided by:
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WAP rate 100
MAP rate greater 100 |
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What is the maximal normal QRS width?
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0.12
normal 0.06-0.11 |
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Which atrial dysrhythmia usually occurs between an atrial rate of 250-350?
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Flutter 250-350
Firbrillation 350-450 |
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In atrial fltutter, what is the most common form of AV block?
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AV node block (usually 2:1)
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Whick of the irregularly irregular rhythms is due to many causes and is the most common to occur?
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atrial fibrillation
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What electrical finding on the EKG signifies pacer induced rhythm?
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electrical spike that is too narrow to be physiologically normal
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What signs help separate ventricular tachycardia from supraventricular with bundle branch block?
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Brugada's sign: Vtach
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What signifies first degree heart block?
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PR greater than 0.2 sec
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What signifies that the second degree block is Mobitz 1?
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progressive lengthing of the PR intervals until a ventricular beat is dropped (**99+% of the time)
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Describe the atrial and ventricular rates in third degree AV block.
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atrial rate:
ventricular rate: 20-40 |
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What happens to the timing of the following beat, post PVC?
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compensatory pause
(PACs do not have a compensatory pause) |
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In what leads do you expect the P waves to always be positive?
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+ P in leads 1, 2, V4-6
(-P in lead aVR) |
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What is the difference between WPW pattern WPW syndrome?
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WPW Pattern
short PR interval wide QRS delta wave ST-T wave changes WPW Syndrom: all above + proxysmal tachycardia |
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How does the PR interval change in Wenkebach block versus Mobitz 2?
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Wenkebach (Mobitz 1) AV node has long refractory period- progressive legnthing until dropped QRS
Mobitz 2 (more dangerous) PR intervals constant until sudden dropped beat |
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Define complete heart block.
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complete dissociation of Ps and QRSs
always more Ps than QRSs |
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Define a Q wave.
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first negative deflection after the P wave
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Define a significant Q wave.
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0.03 sec or wider
depth equal to or greater than 1/3 R wave |
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What do Q waves in two or more adjacent leads mean?
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MI
(need ST changes to dx ACUTE MI) |
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What is the normal QRS duration?
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0.06-0.11
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In which leads does one expect the usual transition zone to occur?
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transition zone= where precordial leads change from neg to pos.
usually between V3 and V4 |
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Extremely low voltage of every wave in every lead could indicate which 2 possible conditions?
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MI (scar tissue is electrically inert)
marked increase body fat huge pleural EFFUSION pericardial EFFUSION recording at half standard |
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What condition precludes the diagnosis of left ventricular hypertophy?
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LBBB
only dx with normal QRS morphology |
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What conditions can cause a QRS duration of great than .11 sec width?
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LBBB
RBBB IV conduction delay (don't look like LBBB or RBBB) WPW |
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What metabloic emergency can cause IVCD?
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hyperkalemia
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How wide does the QRS have to be before you can diagnose at bundle branch block?
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at least one lead with QRS > 0.12
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If the axis of an EKG is "normal," which 2 EKG leads would be expected to be positive?
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lead 1 and aVF
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Which bundle by definition is always involved in bifascular blocks?
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RBBB
(+ one of the hemi's) |
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What diagnosis can never be made in the presence of a Left Bundle Branck Block?
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LVH
(left ventricular hypertrophy) |
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What is the typical QRS appearance in RBBB?
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RSR`
"rabbit ears" |
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What is the typical QRS appearance in LBBB?
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discordant T waves (opposite direction)
small R in V1 "uglygram" |
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What axis would you expect with a left anterior hemiblock?
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vector is superior an leftward
axis: -30 to -90 deg lead 1: qR or R lead 3: rS |
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In determining the vector of the ORS, what is the characteristic of the isoelelctric lead even if R wave size is a different amplitude than the S wave?
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the axis is +/- 20deg from the axis of the isoelectric lead
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What is the hallmark of acute transmural MI?
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pathologic Q wave (wider than 0.03, or greater than 1/3 depth of R wave)
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What is the hallmark sign of acute ischemia?
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ST depression and T wave opposite to QRS
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What is the EKG clue for Arythmogenic Right Ventricular Cardiomyopathy?
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R ventricular inherited desmosome problem= SUDDEN DEATH
epsilon wave-small waves at the J point |
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What is the QTC?
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QT interval corrected for heart rate
QTC = QT + 1.75X normal up to 0.419sec |
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In a normal EKG what can be stated about the T and U waves?
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normal: T and U always in same direction
bad (ischemia til proven otherwise): T and U opposite |
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Why do normal T waves occur in the same direction as the QRS?
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Perkinje system is endocardial-> depolarization is endo- to epi-. Repolarization is opposite because ap duration is longest in the epicardium. Depolarization going toward electrode and repolarization going away from electrode both are positive!
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T wave shape should always be:
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asymetrical
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What causes should you consider in significant T wave elevation?
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acute MI
hyperkalemia (greater than 6mm in limb leads or 12 mm in precordial) (T 2/3 height of the R) |
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what is the difference on EKG between LVH and LVH with strain?
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LVH with Strain- asymmetrical ST depression
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What ST changes are expected in an EKG of a patient with pericarditis?
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PR depression
diffuse ST changes without reciprocal ST depression scooping- upward slope of ST Notching at end of QRS |
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What is the relationship of the T wave to the QRS in a Bundle Branck Block?
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LBBB- discordant T waves (opposite QRS
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What is meant by the term "reciprocal changes" in an EKG and in what pathological state is it relevant?
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lead opposite the damaged area show opposite changes.
ST elevation in one side, ST depression in the opposite |
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Which ion essentially sets the non-pacemaker myocytes resting membrane potential?
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potassium
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What happens to the cell membrane electrically to cause an action potential?
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influx of Na+
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What is differnct about phase 4 action potentials in pacemaker cells and what ion and channel is chiefly responsible?
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Na+ and Ca++ leak channels allow electrical potential to slowly increase during phase 4
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What is the name for the potential difference across the membrane reqquired to maintain the concentraiton gradient?
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equilibrium potential
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What phase of the action potential is responsible for repolarization and what ion is most responsible?
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phase 1
transient K channel (K+) also Ca++ entering thru slow Ca++, fast Na+ channels are inactivated |
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Which ion is predominantly responsible for the plateau phase of an action potential?
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phase 2
slow Ca++ chanels stay open- creates electroneutrality |
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What phase/s of the action potential found in myocardial muscle cells is missing from pacemaker cells?
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NOT phase 1 and 2
Pacemaker cell phases: 0, 3, 4 |
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What is the funciton of the fast Na+ channel in phase 0 of pacemaker cells?
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depolarization
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What two mechanisms are responsible for Phase 0 in pacemaker cells?
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L type Ca++ channels
funny Na channel (If) |
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What EKG abnormality would you expect to demonstrate with severe aortic stenosis?
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LVH or LBBB
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What EKG abnormality would be most likely in severe mitral stenosis?
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A-fib or P mitrale (P>0.12 and notched)
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What EKG abnormalities would you expect in severe chronic mitral regurgitation?
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P mitrale (P>0.12 and notched)
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