Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
150 Cards in this Set
- Front
- Back
What is one of the most common diseases in humans? |
Dental caries |
|
What does dental caries cause? |
pain and disability |
|
What do caries lead to? |
infection, tooth loss, and edentulism at any age |
|
What is one of the oldest theories about caries? |
a tooth worm lived in the center of the tooth. |
|
What was a theory about caries in the 1700s? |
caused by erosion that would be smoothed by a file |
|
What was a theory about caries in 1881? |
caused by organisms |
|
Who published the chemicoparisitic theory of caries? |
W.D. Miller |
|
Who is G.V. Black? |
founder of modern dentistry |
|
What did Dr. Black add to the belief about caries? |
microbic plaque was the source of the acids |
|
Why have disease patterns changed? |
fluoride has been introduced |
|
What must there be in order for caries to develop? |
a susceptible tooth and host cariogenic microorganisms frequent excessive consumption of refined carbohydrates |
|
When must the development of caries process occur? |
over a long period of time |
|
How many categories of the disease process need to occur for caries to happen? |
all 4 |
|
What is the first step in the development of caries? |
a tooth covered in a film of cariogenic bacteria is exposed to carb and metabolizes it. |
|
What is produced from the metabolism of the carb by cariogenic bacteria? |
weak acid byproducts |
|
What does the acidogenesis create? |
an environment that demineralizes the tooth enamel and dentin |
|
Where are the general areas of the tooth that carious lesions occur? |
pit and fissure smooth surface root surface secondary/recurrent carries |
|
Where are secondary/recurrent caries found? |
on tooth surface adjacent to an existing restoration |
|
What are the 3 stages of caries? |
Incipient lesion demineralization toward dentinoenamel juction and pulp overt or frank lesion |
|
What is an incipient lesion? |
initial stage of tooth decay that hasty penetrated the outer surface of the tooth |
|
What does an incipient lesion look like? |
a white spot on enamel |
|
What is lost in incipient lesions? |
calcium, phosphorus, ions of enamel |
|
What is an overt or frank lesion? |
actual cavitation (loss of enamel integrity) |
|
When does rampant decay occur? |
when the development of overt lesions is rapid or extensive and after frequent intake of sucrose and xerostomia |
|
What is an example of when rampant decay would occur? |
baby bottle decay |
|
How is there a direct connection of the bacterial biofilm to the lesion? |
demineralization remineralization enamel rods |
|
What does demineralization create? |
a raged profile |
|
What does progression of a lesion depend on? |
pH Saliva flow rate buffering capacity |
|
What happens when caries reaches the dentin? |
it spreads to a larger area |
|
In what shape does caries develop on the enamel? |
a triangle or arrow |
|
What is an effective means of preventing caries in children? |
reduce the number of MS in parents and siblings mouths before a child's birth |
|
What are two main bacteria species that cause caries? |
mutans streptococcus lactobacillus |
|
What is the main bacteria species that causes caries? |
mutans streptococcus |
|
How many species of bacteria in plaque? |
300 |
|
Where and when does mutans streptococci form? |
immediately over developing smooth surface lesions |
|
What does mutans streptococci produce? |
extracellular glucans and acids |
|
How does mutans streptococci cause damage? |
through lactic acid and other acids |
|
At what pH does enamel get damaged? |
5.5 or lower |
|
What are lactobacilli? |
cariogenic acidogenic aciduric |
|
Are lactobacilli required for caries development? |
no |
|
When are lactobacilli common? |
after irradiation therapy for head and neck cancer |
|
What are the sources of acid? |
dietary bacterial environmental intrinsic (bulimia) |
|
How long does it take to neutralize the pH from solid/sticky sugars?
|
40 minutes
|
|
How long does it take to neutralize the pH from liquid sugars? |
20 minutes |
|
What are the most susceptible teeth to caries? |
1st and 2nd molars on mandible and maxilla mandibular premolars |
|
What teeth have a lower incidence rate of caries? |
anterior teeth |
|
Why do anterior teeth have a lower incidence rate of caries? |
they are smooth saliva |
|
What teeth are the least susceptible to caries? |
lower anterior |
|
What are the major salivary glands? |
parotid submandibular sublingual |
|
What role does saliva play with caries? |
helps prevent caries |
|
How does saliva help prevent caries? |
repaires damage by neutralizing acids and replacing lost minerals |
|
Which gland secretes sodium bicarbonate and initiates carbohydrate digestion? |
parotid |
|
What is sodium bicarbonate? |
baking soda
|
|
What is the function of saliva? |
lubrication clearance of food buffers pH antibacterial remineralizes with its contents |
|
What contents of saliva help with remineralization? |
calcium phosphates fluoride saturation |
|
What ph does coronal dental caries start? |
5.5 and lower |
|
What pH does root caries start? |
6 and lower |
|
What does enamel protect? |
underlying dentin |
|
What does dentin protect? |
pulp |
|
What features on coronal caries give in to caries? |
pits and fissures interproximal smooth surface |
|
What exposes root to caries? |
gingival recession |
|
what can create xerostomia? |
medication, cancer and age |
|
What are the risk factors that lead to caries? |
age gender fluoride exposure illness oral hygiene diet |
|
What are secondary or recurrent caries due to? |
imperfection in or around restoration |
|
How can secondary or recurrent caries be lessened? |
by preventing the imperfections applying fluoride materials to bone the tissue |
|
What is the Stephan Curve? |
the immediate drop in pH when sugary snacks are eaten, followed by a longer recovery period when other foods are eaten. |
|
What is another name for the Stephan Curve? |
drop and recovery curve |
|
What is demineralization caused by? |
plaque acid that dissolves the tooth minerals |
|
What does remineralization require? |
ions with fluoride with a catalyst |
|
What are the minerals that make up the tooth? |
calcium, phosphate, and hydroxyl crystals |
|
What does continued exposure to low concentrations of fluoride result in? |
gradual incorporation of fluoride into existing hydroxyapatite crystals |
|
what does fluoride and hydroxyapatite crystals create? |
fluorohydroxyapatite |
|
What is special about fluorohydroxyapatite? |
its more resistant to acid damage |
|
What is successful in stimulating surface remineralization? |
topical procedures such as gels, dentifrices and varnish |
|
What do some researchers believe? |
that remineralization is a reasonable objective for caries that reach the dentin |
|
What are some protective measures against caries? |
daily oral hygiene diet fluoride sugarfree gum or mints antibacterial rinse |
|
What helps with dry mouth? |
saliva substitute products bakingsoda xylitol gum/mints sip on water throughout day |
|
What helps with caries management? |
risk assessment early detection fluoride/antibacterial therapy minimally invasive restorations |
|
What is CAMBRA? |
validated evidence-based approach to preventing, reversing, and treating caries |
|
what does CAMBRA emphasize? |
whole disease process |
|
how long does caries take to form? |
months to years |
|
How do you diagnose caries? |
drying tooth visually transillumination radiographs lasers |
|
What is dental calculus? |
hard calcified dental plaque |
|
where does dental calculus occur? |
teeth implants dentures appliances |
|
What is calculus always covered with? |
plaque |
|
Where doesn't calculus grow? |
gingiva |
|
What is found in calculus? |
things found in plaque and calcifying salts |
|
Does calculus by itself initiate periodontal disease or caries? |
no |
|
What are some physical characteristics of calculus? |
porous attracts and harbors plaque |
|
how does calculus perpetuate periodontal disease? |
by being physically and chemically irritating |
|
What is the first step in non-surgical periodontal therapy? |
removal of calculus to eliminate gingival inflammation |
|
Is there pain associated with calculus? |
no |
|
How is calculus classified? |
by its location on the tooth surface related to the gingival margin |
|
what is supragingival calculus? |
above the gingival margin |
|
what are other names for supragingival ?
|
supramarginal
extragingival |
|
what is the source of minerals for supra gingival
calculus? |
saliva |
|
Where is supra gingival calculus most abundant? |
near openings of major salivary glands buccal side of maxillary molars lingual of mandibular anteriors |
|
What is the recurrence like after removal of supra gingival calculus? |
may be rapid |
|
what is the texture and color of supra gingival calculus? |
hard covered in plaque white,yellow |
|
how is supra gingival calculus recognized? |
clinical inspection by drying the tooth to see a chalky appearance |
|
Where is subgingival calculus? |
under the gingival margin |
|
What are other names for subgingival calculus? |
submarginal serumal |
|
Where does subgingival calculus get its minerals? |
the serum in blood |
|
Where is subgingival calculus commonly found? |
interproximal proximal and lingual surfaces |
|
Which is harder to remove supra gingival or subgingival calculus? |
subgingival |
|
What is the consistency, texture, and color of subgingival calculus? |
brittle harder and denser light to dark brown or black |
|
which type of calculus has a wider distribution? |
subgingival calculus |
|
How is subgingival calculus detected? |
tactile visual (color change or blowing air) radiographs periscopy |
|
what are the structures of subgingival calculus?
|
spicules ledge ring veneer |
|
What is calculus made of? |
inorganic and organic componants and water |
|
Is it made of mainly organic or inorganic componants? What percent?
|
inorganic 70-90% |
|
What are the inorganic componants? |
calcium phosphorus carbonate sodium magnesium potassium |
|
Is there flouride in calculus? |
yes, but it varies on amount of fluoride from H2O, topical Fl, toothpaste and rinses
|
|
What is 2/3 of the inorganic matter? |
crystals |
|
is calculus alive or dead? |
dead |
|
how long does mineralization of calculus take? |
3-4days to week |
|
how does calculus appear on a radiograph? |
radioopaque, in interproximals |
|
What forms in 12 hours? |
mineral elements from plaque |
|
what forms in 3-4 days? |
heavy calculus more calcium and phosphate |
|
what is the average time it takes for calculus to mineralize? |
12 days |
|
Why is there an increase in calculus for people on dialysis and tube feeding?
|
they aren't chewing to produce saliva |
|
what color is swimmers calculus? |
yellow to brown |
|
What is the mineralization theory? |
saliva is super saturated because of the sales and is able to support crystal growth but can't be used because of the pellicle |
|
What is the carbon dioxide theory? |
saliva leaving the duct has CO2 tension... but not accepted |
|
What is the ammonia theory? |
ammonia breaks down from urea that results in pH increase in plaque.... but not accepted |
|
what is the initial layer of crystal formation? |
brushite paralel to tooth |
|
what does brushite calcify into? |
whitlockite |
|
What happens to the older layers of whitlockite? |
become hard to remove |
|
What do crystals contain? |
ions |
|
How does calculus form? |
in layers |
|
how long does calculus form in heavy formers? |
3-4 days |
|
Where does mineralization begin? |
intercellular plaque matrix |
|
What are the modes of calculus attachment? |
acquired pellicle irregularities on tooth surfaces direct contact (braces) |
|
Which mode of attachment is most common? |
irregularities on tooth surfaces |
|
where does calculus form? |
areas closest to salivary ducts areas hard to clean |
|
What type of calculus do kids have? |
supra |
|
Why should calculus be removed? |
to prevent inflammation of periodontal tissue |
|
What does the tenacity of calculus depend on? |
the length of time present attachment mechanism supra vs sub the individual |
|
What are the means of attachment of calculus?
|
acquire pellicle mechanical locking direct contact |
|
Which attachment is easiest to remove?
|
pellicle |
|
which attachment is hardest to remove? |
mechanical locking |
|
what is direct contact attachment? |
between calculus and exposed roots and strongly attached to cementum |
|
how do you prevent calculus? |
homecare instruction professional supervision |
|
What does the professional removal of calculus benefit? |
makes environment conducive to healing easier maintenance |
|
what is the significance of radiographs for calculus? |
aids in detection guides during scaling patient education |
|
What is the objective of anticalculus dentifrices and mouthrinses? |
inhibits calculus growth |
|
what should the patient be taught about calculus? |
person oral hygiene how it forms what it is effects on peridontium |
|
how long does it take for supra gingival calculus to form? |
3-20 days |
|
how early can mineralization of subgingival calculus begin? |
24 - 48 hours |
|
what do anti calculus agents have that prevents calculus?
|
pyrophosphates
|