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128 Cards in this Set
- Front
- Back
Electrocardiogram (ECG) |
Monitors PQRST & U waves of heart |
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Why is ECG used? |
Dx dysrythmias, myocardial ischemia, injury, infarction & monitor electrolyte imbalance or med admin |
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P Wave represents? |
Firing of SA node, atrial depolarization |
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QRS represents? |
AV depolarization through ventricles |
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T wave represents? |
Repolarization of ventricles |
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U wave represents? |
Repolarization of purkinje fibers or associated w/ hypokalemia |
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Systole |
Contraction, ventricles eject blood |
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Diastole |
Relaxation, blood flow into ventricles |
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Stroke Volume (SV) |
Amount of blood ejected per beat |
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Cardiac output (CO) |
Amount of blood pumped by each ventricle per min. |
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CO formula |
CO=SV×HR |
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Norm. CO |
4-8 L/min |
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Cardiac index formula? |
CO÷BSA |
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Cardiac index norm. |
2.8-4.2 L/min/m^2 |
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CVD inc. With? |
age |
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Leading cause of death in adults >65yo |
CVD |
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CAD most common cardiovascular problem due to? |
Atherosclerosis |
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Cardiovascular change result of |
Aging, disease, environmental factors, lifetime behaviors |
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Age related changes include |
Dec. Stress response, inc. SBP and dec./no change in DBP, ortho hypotension, postprandial hypotension, inc. collagen, dec. Elastin |
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Subjective CVD Qs |
Hx present illness, past health Hx, past/current meds, past surg. |
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CVD risks factors |
HTN, Sedentarty life, DM, obesity, genetics, tobacco, abn. Lipids |
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CVD objective data (vitals) |
Vitals BP bilateral SBP should not dec. More than 20 mmHg from supine to standing |
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CVD objective data inspect, palate, auscultate |
Inspect: edema, neck vein distention, stasis ulcers, nail clubbing.
Palpate: extremity moisture, temp, pulse, cap. Refill, edema Auscultate: major arteries, bruit |
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Bruit is |
Abn. Flow causing humming or buzzing |
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Cardiac biomarkers/enzymes |
Released into blood when heart suffers ischemia |
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Troponin levels help Dx? |
Acute coronary failure |
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Troponin specific to heart? |
Troponin T (cTnT)/ Troponin I (cTnI) |
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Troponin rise, peak, detected for? |
Rise: 4-6hr Peak: 10-24hr Detected: 10-14 days |
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Creatine kinase (CK) |
Found in heart (specific CK-MB) |
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CK-MB rise, peak, baseline return? |
Rise: 3-6hr Peak: 12-24hr Baseline return: 12-48hr |
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Myoglobin is |
Heme protein found in heart, sensity to early myocardial injury, limited in MI Dx |
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C-reactive protein |
Produced by liver during acute inflammation, inc. Lvl linked to presence of atherosclerosis/1st occurance of heart attack. |
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Serum lipids consist of |
Triglycerides, cholesterol, phospholipids |
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Total cholesterol lvls |
<200 mg/dL |
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LDL (Bad cholesterol) lvl |
Transport cholesterol to cells from liver
<130mg/dL |
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Triglyceride lvls |
M: 40-160 mg/dL F: 35-135 mg/dL |
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HDL (good cholesterol) lvls |
Protect coronary arteries by transporting cholesterol to liver M: >45 mg/dL F: >55 mg/dL |
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Chest X-ray Dx |
Show heart shape, size, anatomical changes. Records enlargement of heart, pericardial effusion, pulmonary congestion. |
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Electrocardiogram (ECG) is used to |
Asses heart activity, monitor resting/ambulatory ECG, stress testing |
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Echocardiogram is |
Ultrasound of heart, with/without contrast, provide info regarding structure/motion of heart, measure ejection fraction |
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Ejection fraction |
65-70% percentage of end diastolic blood volume ejected,can also measure during systole. Provide info on left ventricle during systole |
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Cardiac catheterization |
R Side: measure pressures L Side: evaluate coronary arteries |
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Cardiac catheterization edu, consideration, actions |
NPO 8hrs, consent form, client understand procedure, iodine/shellfish allergy, renal function |
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Coronary angiography is for |
Identifying coronary blockage using contrast medium |
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One of the most modifiable risk factors that lead to CVD is |
HTN |
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Inc. BP inc. Risk for |
MI, Heart attack, Stroke, Renal disease |
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Systemic vascular resistance |
Force opposing the movement of blood within vessels |
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Normal BP range |
SBP: <120 mmHg DBP: <80 mmHg |
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Prehypertension range |
SBP: 120-130 mmHg Or DBP: 80-89 mmHg |
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Stage 1 HTN range |
SBP: 140-159mmHg Or DBP: 90-99 mmHg |
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Stage 2 HTN range |
SBP: > OR = 160 mmHg Or DBP: >OR= 100mmHg |
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Primary HTN is |
AKA essential/idiopathic HTN Inc. Without identified cause Exact cause unknown, several contributing factors |
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Secondary HTN |
inc.BP with specific cause (med, kidney disease), Tx aimed at removing or Tx cause. |
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HTN risk factors |
Age, DM, Obesity, Stress, Excess sodium, ETOH, inc. Serum lipids. |
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HTN clinical manifestations are |
Monitor: Headaches, facial flushing, fatigue, dizziness/fainting, palpation, angina, renal change, dyspnea, visual disturbance |
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HTN target organs are |
Heart, brain, PVD, kidney, eyes |
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Overall HTN goals are |
Control BP, dec. CVD risk factors/target organ disease, lifestyle modifications(weight loss #1) |
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2 main action of drug therapy for HTN |
Dec. Volume circulating blood Reduce SVR |
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Diuretics for HTN |
Dec. Volume, promote Na/H2O excretion, reduce plasma volume/vascular response to dopamine, nor-epinephrine Monitor for hypokalemia: muscle weakness, irregular pulse, dehydration |
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Ace inhibitors function by |
Prevent conversion of angiotensin 1 to angiotensin 2 (BP enzyme) preventing vasoconstriction. |
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Captopril Rx |
Monitor BP, Hypotension (fatigue, dizziness, nausea, blurry vision, lightheadedness) monitor edema (evidence of heart failure) Can cause renal/heart complications Report cough/angioedema severe adverse effect |
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Calcium channel blocker |
Blocks Ca access to cells causing vasodilation, dec. BP/HR |
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Nifedipine Rx (Ca blocker) |
Monitor BP, hypotension, use with caution in pts with heart failure. Educate pt how to take pulse, call HCP if irregular or lower that norm. Avoid grapefruit juice, potentates med, inc. Hypotensive effects. |
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What is atherosclerosis? |
Hardening of arteries |
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What are arthromas? |
Fatty deposits that prefer coronary arteries |
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Stages of atherosclerosis development are |
1. Chronic endothelial injury 2. Fatty streak 3. Fuberous plaque 4. Complicated lesion |
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Non modifiable risk factors for CAD are |
Inc. Age, genetics, ethnicity (more common in white men), gender (more common in men than women until 75yo) |
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Modifiable risk factors of CAD are |
Obesity, DM, physical act., serum lipids, BP <140/90 mmHg, tobacco, psychosocial (stress, anger, depression) |
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What should a CAD assessment include? |
Health/Family/psychosocial Hx, diet, activity, presence of cardio symptoms. |
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What type of nutritional therapy should be considered in CAD pts? |
Lower LDL cholesterol |
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Low LDL cholesterol meals include... |
Dec. Saturated fats and cholesterol (vegetable oil, shellfish, nuts, seeds margarine) |
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What foods are major sources of saturated fats and cholesterol? |
Red meat, egg yolks, whole milk products (butter, sour cream, cream cheese) |
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Initiate lipid lowering drug therapy if... |
Diet and exercise are ineffective |
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The type of med most commonly used that functions by inhibiting synthesis of cholesterol in liver, dec. LDL, and inc. HDL? |
Statins |
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When on statins, why are liver enzymes monitored? |
Liver damage/myopathy |
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A serious side effect of Simvastatin (zocor) and Gemfibrozil (lopid) is... |
Liver failure, rhabdomyolysis (muscle pain, weakness) |
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Antiplatelet therapy functions by |
Dec. Platelet aggregation, inhibit thrombus formation. |
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Some anticoagulants include... |
Aspirin (ASA), Clopidrogrel (plavix) |
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Some considerations before starting AC therapy? |
Hx of bleeding, teach pt of inc. Bleeding and bruising, have blood monitored to check bleeding times (PT, aPTT, INR, CBC) |
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High o2 demand greater than o2 supply can lead to... |
Myocardial ischemia |
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Clinical manifestation of angina is |
Chest pain |
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The most common reason for angina to develop is |
Atherosclerosis (70% or more blockage) |
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Chronic stable angina is... |
Chest pain that occurs intermittently over a long period of time. Usually in similar pattern. |
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What can cause an episode of Chronic stable angina? |
Exercise or emotional stress |
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What can relieve an episode of Chronic stable angina? |
Rest or nitroglycerin |
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Some pt descriptions of Chronic stable angina are... |
Pressure, heaviness, tight suffocating sensation, difficulty breathing, fatigue |
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Overall goal to treat Chronic stable angina? |
Dec. O2 demand and/or inc. O2 supply |
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Shirt acting nitrates work by... |
Dilate peripheral and coronary blood vessels |
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Shirt acting nitrate administration are by |
Sublingual or spray |
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Short acting nitrates in an emergency steps are |
Administer, no relief of pain call 911;if some relief repeat q5 mins for a max of 3 doses. *WE NEED COMPLETE RELIEF NOT SOME* |
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What could be included in pt teaching when giving shirt acting nitrates? |
Headache is the most common side effect, can be take prophylactically before activity know to cause angina |
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Beta blockers work by... |
Blocking receptors on heart by causing dec. HR and force of contraction. |
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Some beta blockers are... |
Atenolol and metoprolol ( end in -lol) |
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Unstable angina.... |
Occurs at rest, pain last >/= 10 min, need immediate Tx |
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What are S/S of unstable angina |
SOB, fatigue (most prominent), anxiety, indigestion |
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Some interventions for Angina are.... |
Upright position, supplemental o2, vitals, 12 lead ECG, a assess heart/lungs. Pt will most likely be anxious have cool, pale, clammy skin. |
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Peripheral Artery Disease (PAD) is |
thickening of artery walls |
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What does PAD AND PVD affect |
Blood flow to (PVD) and from (PAD) heart |
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PVD/PAD is a result of... |
Atherosclerosis |
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Risk factors for PVD/PAD |
Tobacco (most important) ,CKD, DM, HTN, females, age, hypercholesterolemia |
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Clinical manifestations of PAD/PVD include... |
Burning, claudication (pain and/or cramping), numbness in feet. |
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PVD/PAD assessment findings include... |
*appearance is most important regarding blood flow* Dec. Cap refill, atrophy, redness, diminished or absent pedial, femoral, popliteal pulses. |
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Complications of PVD/PAD are |
Atrophy of skin, delayed healing, infection, necrosis, arterial ulcers (occur over boney prominences on toes, legs, feet) |
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What helps promote vasodilation? |
Warm environment (ex. Warm socks) avoid caffeine, cold, stress and nicotine, avoid crossing legs, elevation to reduce swelling. |
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What type of exercise is recommended? |
Walking helps build circulation. Walk until pain, stop, rest, walk further |
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Angioplasty is |
Insertion of catheter through femoral artery |
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Mechanical rotational abrasive artherectomy is |
Scrap plaque from inside peripheral artery |
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Arterial revascularization surgery is |
Rerouting circulation around a blockage, can cause potential limb loss |
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What are some nursing considerations for pts who've had therapeutic procedures? |
Frequent vitals (q15min), observe site for bleeding, monitor VS, peripheral pulse, cap refill, movement and sesation. Bedrest and keep limb straight 2-6hrs |
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How can a nurse provide a pt with PVD/PAD conservative Tx? |
Trauma protection, dec. Ischemic pain, preventing/control infection, improve perfusion. |
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Venous thromboembolism (VTE) risk factors are |
Heart failure, immobility, post OP, pregnancy, oral contraceptives, active cancer |
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VTE expected findings are |
Tenderness,sudden edema, warmth, calf/groin pain, can be asymptomatic |
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What is indicative that a embolism has gone to the lungs from VTE? |
SOB and chest pain |
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What type of care can be offered to those with DVT and thrombophlebitis? |
Encourage rest and elevation, warm compress, no massage, anti embolism stockings, IVC filter |
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What are some risk factors for venous insufficiency? |
Sitting/standing in place for long time, obesity, preg, thrombophlebitis |
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Some of the expected findings in those who may have venous insufficiency are |
Edema, stasis ulcers, brown discoloration. |
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Risk factors for varicose veins are |
Female, 30yo older, fam Hx, obesity, preg, standing |
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Finding that may indicate varicose veins are... |
Distended veins, muscle cramps/spasms, pruritus (itching) |
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Nursing care for venous insufficiency are |
Evelvate legs 20 min 4-5× day., avoid restrictive clothes/crossing legs, wear compression sock after elevation and when swelling is minimal |
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Therapeutic procedures for venous insufficiency could be... |
Sclerotherapy, vein stripping, laser Tx |
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What meds are used for DVT/thrombophlebitis? |
Heparin, enaxoprin, warfarin |
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What med is used to prevent clot formation or enlargement? |
IV heparin |
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What med uses potanin sulfate as it antidote? |
Heparin |
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What to monitor when giving heparin |
PTT, bleeding, platelet count |
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What med uses vitamin K as its antidote? |
Warfarin |
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What labs are monitored when administering warfarin? |
PT/INR |
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How long to reach warfarin therapeutic levels? |
3-4 days |
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What therapy dissolves clots within 5 days of therapy? |
Thrombolytic therapy |