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124 Cards in this Set

  • Front
  • Back
Mediastinum
the chest cavity that contains the heart
Pericardium
protective covering surrounding the heart
Epicardium
Outer layer of the heart, continuos with the pericardium
Myocardium
Middle layer of the heart, muscle
Endocardium
Inner layer of the heart, continuous with tunica intima of blood vessels
Atrioventricular (AV) valves
separate atria from ventricles
Bicuspid (mitral) valve
separates left atrium from left ventricle
Tricuspid valve
separates right atrium from ventricle
Chordae tendinease
cords holding valve leaflets
Papillary Muscles
attach to chordae tindineae
Semi lunar valves
go from ventricles to arteries, prevent back flow of blood into the ventricle
Pulmonic Valve
Prevents back flow of blood from pulmonary artery into right ventricle
Aortic Valve
prevents back flow of blood from aorta into left ventricle
Cardiac Cycle
one complete heartbeat
Systole
contraction phase of heartbeat
Diastole
relaxation phase of heartbeat, when atria and ventricles are filling. Lasts longer then the systole phase
Stroke Volume (SV)
amount of blood ejected from either ventricle during one contraction
Cardiac Output (CO)
amount of blood pumped through cardiovascular system in one minute.
CO =
SV x heart rate (HR)
- expressed in liters/min
End diastolic volume
volume in ventricle prior to contraction
End systolic volume
volume in ventricle after contraction
Ejected Fraction (EF)
Stroke volume/end diastolic volume. The % of ventricular blood volume that is pumped out with each contraction
LVEF
left ventricular ejection fraction, a measure of heart functionality
>50%
normal
35%- 50%
indicative of weakened myocardium
<35%
very bad
EF can be determined by
-echocardiography measurements
- gated myocardial perfusion imaging studies
-MUGA (multi gated acquisition scan) the gold standard for determining LVEF
Sinoatrial (SA) node
dominant pacemaker of the heart. Paces at 60-100 bbm
Atrioventricular (AV) node
another pacemaker along the conduction tissue pathway. Paces at 40-60 bbm.
Bundle of HIS
transits impulses to left ventricle
Left bundle branch
conducts impulses to left ventricle
Right bundle branch
conducts impulses to right ventricle
Purkinje fibers
network of fibers that conduct impulses throughout ventricular walls. Also, a pacemaker; paces at 20-40 bbm
Depolarization
electrical charge in the myocyte is changed to positive by a shift in electrolytes on either side of the cell membrane.
- Causes muscles fiver to contract
Re-polarization
Chemical pumps reestablish an internal negative charge in the myocyte
Electrolyte balance
is important for our hearts!
- This involves sodium, potassium, calcium, and magnesium.
Electrocardiogram
(EKG) - a record of the heart's electrical activity
Movement of electrolytes
The depolarization from the SA node through the atria is caused by fast moving sodium ions (NA+)
The depolarization is carried through the AV node by the slower moving
calcium ions (Ca+)
Conduction through the ventricular system, after the
AV node, is by fast sodium ions (Na+)
Re-polarization is caused by
potassium ions (K+) leaving the myocytes
Cardia Cycle in Detail
The wiggly lines commonly seen
Sa node sends a wave of depolarization that spreads outward through the aria, causing them to contract.
This is represented by the P wave on the EKG.
The depolarization wave travels through the
conduction fibers through the AV node.
There is a delay in conduction as the wave moves through the
AV node.
The delay allows the ventricles to fill
before contracting. This delay is represented by the P-R interval on the EKG
Once depolarization reaches the AV node, it is conducted
rapidly through the ventricular conducting system.
It moves through the Bundle of HIS, through the left and right ventricle bundle branches,
and into the Purkinje fibers. THe ventricles contract. This is represented by the QRS complex on he EKG.
After the ventricles contract they begin the process of re polarization, represented by
the plateau on the EKG called the ST segment.
The broad T wave represents the
final, rapid phase of ventricular re polarization.
EKG
A recording of the heart's electrical activity.
Leads ( also called views)
monitor the voltage changes in electrodes placed in different positions of the body
Voltage changes are amplified and displayed on
an oscilloscope and on graph paper.
Each lead looks different because the placement of the
electrode changes, and the electrical activity in each area of the body is different.
Incorrect placement of the electrodes can
turn a normal ECK into a abnormal EKG
EKG
For cardiac monitoring and gating during nuclear medicine procedure, a 3-electrode EKG is used.
The three leads are
black, red and white.
The white is placed on the
patient's right clavicle
The black lead is placed on the
patient's left clavicle
The red lead is placed on the
patient's chest just left of midline and near the 4th intercostal space.
Remember:
White on
right
Smoke (black) over
fire (red)
EKG analyses are performed using a
12 lead EKG
For a 12-lead EKG, four wires are attracted to each limb and six
wire are attached to the chest. The ten wires provide 12 different leads (views)
Electrodes are placed on the right arm RA, left arm LA,.....
right left RL and left leg LL
Six leads are views from these four electrodes :
standard leads I, II, III
Augmented leads =
aVR, aVL, aVF
EKG Lead Placement
Look at Diagram
The four limb electrodes each have a positive and element to them.
Together the four electrodes produce 6 leads, I, II, III, aVR, aVF, and aVL
The 6 chest leads are all unipolar and are all
positive
They are placed on the chest as shown on the diagram, slide 23
They are numbered V1 through V6
The ECG tracing shows progressive changes in the
wave forms coming from leads V1 through V6
A wave of depolarization traveling toward a positive electrode causes
a positive (upward) deflection in the ECK tracing.
A wave of depolarization traveling away from a positive electrode causes
a negative (downward) deflection in the EKG tracing.
P wave
atrial depolarization (contraction)
PR interval
time in which depolarization wave travels from atria to ventricles.
QRS interval
Q is the first negative deflection,
R is the positive deflection
S is the second negative deflection, ventricular depolarization (contraction)
ST segment
time between ventricular depolarization and depolarization
T wave
rapid phase of ventricular re polarization
U wave
small rounded wave following T wave, represents re polarization of Purkinje fibers, not always present see more often with a slow HR.
The EKG tracing is recorded at a constant
speed of 25mm/sec
Each small box on the paper is
1 mm x 1mm
Each large box is
5mm x 5mm
Each small box is
0.04 seconds
Each large box is
0.20 seconds
Calculating heart rate
Method 1: Count the number of large boxes between the 2 R waves and divide into 300.
- 60 sec/min divided by 0.20 sec/large box = 300 large boxes/min
Counting Heart Rate
Method 2: Count the number of small boxes between R waves and divide by 1500
- Used for vert fast heart rates
For irregular heart rates, count the number of R-R intervals
in a six second strip and multiply by 10
Normal values

PR interval
0.12- 0.20 sec
QRS Complex
0.06 - 0.10 sec
QT Interval
0.30 - 0.46 sec
Rate
Pacing from the SA node generates a sinus rhythm.
- Normal sinus rhythm is 60 to 100 beats per minute.
When the Sa node paces at a rate less than 60 bpm, it is called a
sinus brachycardia
When the SA node paces at a rate greater than 100 bpm, it is called a
trachycardia
Rhythm is either
regular or irregular.
Sinus Arrhythmia
The Sa node paces irregularly
The R-R interval is
irregular
P wave is normal, PR interval is normal,
QRS complex is normal
Atrial Arrhythmia's

P Waves
differ in appearance from sinus P waves, indicating irregularity in atrial contractions
Premature Atrial Contraction (PAC)
A single complex occurs earlier than expected, then normal sinus rhythm resumes
Atrial Fibrillation (A-fib)
many sites in the atria are generating depolarization waves
No distinguishable P wave
PR interval not measurable
Rate 100 - 160 bpm
Rhythm - regular
Atrial Flutter
P wave replaced with multiple flutter waves
- Rate around 110 bpm
- Rhythm - regular
First Degree AV Block
Caused by delayed conduction through the AV node
- Characterized by prolonged PR interval (>5 small blocks or 0.20 sec)
Ventricular Tachycardia

Rate 180 - 190 bpm
Rhythm regular
- Abnormal tissues in the ventricular generate rapid contractions.
- Poor cardiac output results.
***This is an emergency situation
Ventricular Fibrillation (V-Fib)

Rate:
300 +
Disorganized electrical signals caused the ventricles to quiver instead of contract
- There is no blood being pumped to the brain or body
- Use the defibrillator ASAP!!
Myocardial Infarct (MI)
Rhythm - Regular
Rate- 80 bpm
P wave- normal, QRS complex, - normal
ST segment does not return to baseline
Asystole
Rhythm - flat
Rate- 0 bpm
Carry out CPR and defibrillation
Coronary Artery Disease (CAD)
Blockage of one of the coronary arteries by plaque and cholesterol deposits
PTCA
Percutaneous transluminal coronary angiography
The coronary arteries are accessed via skin incision, and the procedure is performed within the blood vessels.
Radioplaque dye is used to show coronary arteries
Stents and ballon catheters are
deployed through the incision if needed
Stents
a tube use to prevent flow constriction
Ballon Catheter
A catheter with an inflatable ballon at its tip used to open or enlarge a blocked artery
Stenting
LOOK AT P.P
ACID
Automated implanted cardioverter defibrillator
Pacemaker
Cardo Terminology
CABG
Coronary artery bypass graft
CHF
Congestive heart failure
Dyspnea
difficulty breathing
MI
Myocardial infarction
Angina
Chest Pain