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124 Cards in this Set
- Front
- Back
Mediastinum
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the chest cavity that contains the heart
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Pericardium
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protective covering surrounding the heart
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Epicardium
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Outer layer of the heart, continuos with the pericardium
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Myocardium
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Middle layer of the heart, muscle
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Endocardium
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Inner layer of the heart, continuous with tunica intima of blood vessels
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Atrioventricular (AV) valves
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separate atria from ventricles
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Bicuspid (mitral) valve
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separates left atrium from left ventricle
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Tricuspid valve
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separates right atrium from ventricle
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Chordae tendinease
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cords holding valve leaflets
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Papillary Muscles
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attach to chordae tindineae
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Semi lunar valves
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go from ventricles to arteries, prevent back flow of blood into the ventricle
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Pulmonic Valve
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Prevents back flow of blood from pulmonary artery into right ventricle
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Aortic Valve
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prevents back flow of blood from aorta into left ventricle
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Cardiac Cycle
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one complete heartbeat
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Systole
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contraction phase of heartbeat
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Diastole
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relaxation phase of heartbeat, when atria and ventricles are filling. Lasts longer then the systole phase
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Stroke Volume (SV)
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amount of blood ejected from either ventricle during one contraction
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Cardiac Output (CO)
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amount of blood pumped through cardiovascular system in one minute.
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CO =
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SV x heart rate (HR)
- expressed in liters/min |
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End diastolic volume
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volume in ventricle prior to contraction
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End systolic volume
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volume in ventricle after contraction
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Ejected Fraction (EF)
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Stroke volume/end diastolic volume. The % of ventricular blood volume that is pumped out with each contraction
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LVEF
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left ventricular ejection fraction, a measure of heart functionality
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>50%
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normal
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35%- 50%
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indicative of weakened myocardium
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<35%
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very bad
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EF can be determined by
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-echocardiography measurements
- gated myocardial perfusion imaging studies -MUGA (multi gated acquisition scan) the gold standard for determining LVEF |
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Sinoatrial (SA) node
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dominant pacemaker of the heart. Paces at 60-100 bbm
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Atrioventricular (AV) node
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another pacemaker along the conduction tissue pathway. Paces at 40-60 bbm.
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Bundle of HIS
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transits impulses to left ventricle
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Left bundle branch
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conducts impulses to left ventricle
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Right bundle branch
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conducts impulses to right ventricle
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Purkinje fibers
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network of fibers that conduct impulses throughout ventricular walls. Also, a pacemaker; paces at 20-40 bbm
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Depolarization
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electrical charge in the myocyte is changed to positive by a shift in electrolytes on either side of the cell membrane.
- Causes muscles fiver to contract |
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Re-polarization
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Chemical pumps reestablish an internal negative charge in the myocyte
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Electrolyte balance
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is important for our hearts!
- This involves sodium, potassium, calcium, and magnesium. |
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Electrocardiogram
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(EKG) - a record of the heart's electrical activity
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Movement of electrolytes
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The depolarization from the SA node through the atria is caused by fast moving sodium ions (NA+)
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The depolarization is carried through the AV node by the slower moving
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calcium ions (Ca+)
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Conduction through the ventricular system, after the
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AV node, is by fast sodium ions (Na+)
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Re-polarization is caused by
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potassium ions (K+) leaving the myocytes
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Cardia Cycle in Detail
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The wiggly lines commonly seen
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Sa node sends a wave of depolarization that spreads outward through the aria, causing them to contract.
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This is represented by the P wave on the EKG.
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The depolarization wave travels through the
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conduction fibers through the AV node.
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There is a delay in conduction as the wave moves through the
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AV node.
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The delay allows the ventricles to fill
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before contracting. This delay is represented by the P-R interval on the EKG
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Once depolarization reaches the AV node, it is conducted
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rapidly through the ventricular conducting system.
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It moves through the Bundle of HIS, through the left and right ventricle bundle branches,
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and into the Purkinje fibers. THe ventricles contract. This is represented by the QRS complex on he EKG.
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After the ventricles contract they begin the process of re polarization, represented by
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the plateau on the EKG called the ST segment.
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The broad T wave represents the
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final, rapid phase of ventricular re polarization.
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EKG
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A recording of the heart's electrical activity.
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Leads ( also called views)
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monitor the voltage changes in electrodes placed in different positions of the body
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Voltage changes are amplified and displayed on
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an oscilloscope and on graph paper.
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Each lead looks different because the placement of the
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electrode changes, and the electrical activity in each area of the body is different.
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Incorrect placement of the electrodes can
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turn a normal ECK into a abnormal EKG
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EKG
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For cardiac monitoring and gating during nuclear medicine procedure, a 3-electrode EKG is used.
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The three leads are
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black, red and white.
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The white is placed on the
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patient's right clavicle
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The black lead is placed on the
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patient's left clavicle
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The red lead is placed on the
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patient's chest just left of midline and near the 4th intercostal space.
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Remember:
White on |
right
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Smoke (black) over
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fire (red)
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EKG analyses are performed using a
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12 lead EKG
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For a 12-lead EKG, four wires are attracted to each limb and six
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wire are attached to the chest. The ten wires provide 12 different leads (views)
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Electrodes are placed on the right arm RA, left arm LA,.....
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right left RL and left leg LL
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Six leads are views from these four electrodes :
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standard leads I, II, III
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Augmented leads =
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aVR, aVL, aVF
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EKG Lead Placement
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Look at Diagram
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The four limb electrodes each have a positive and element to them.
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Together the four electrodes produce 6 leads, I, II, III, aVR, aVF, and aVL
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The 6 chest leads are all unipolar and are all
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positive
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They are placed on the chest as shown on the diagram, slide 23
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They are numbered V1 through V6
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The ECG tracing shows progressive changes in the
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wave forms coming from leads V1 through V6
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A wave of depolarization traveling toward a positive electrode causes
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a positive (upward) deflection in the ECK tracing.
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A wave of depolarization traveling away from a positive electrode causes
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a negative (downward) deflection in the EKG tracing.
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P wave
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atrial depolarization (contraction)
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PR interval
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time in which depolarization wave travels from atria to ventricles.
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QRS interval
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Q is the first negative deflection,
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R is the positive deflection
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S is the second negative deflection, ventricular depolarization (contraction)
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ST segment
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time between ventricular depolarization and depolarization
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T wave
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rapid phase of ventricular re polarization
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U wave
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small rounded wave following T wave, represents re polarization of Purkinje fibers, not always present see more often with a slow HR.
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The EKG tracing is recorded at a constant
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speed of 25mm/sec
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Each small box on the paper is
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1 mm x 1mm
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Each large box is
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5mm x 5mm
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Each small box is
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0.04 seconds
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Each large box is
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0.20 seconds
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Calculating heart rate
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Method 1: Count the number of large boxes between the 2 R waves and divide into 300.
- 60 sec/min divided by 0.20 sec/large box = 300 large boxes/min |
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Counting Heart Rate
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Method 2: Count the number of small boxes between R waves and divide by 1500
- Used for vert fast heart rates |
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For irregular heart rates, count the number of R-R intervals
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in a six second strip and multiply by 10
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Normal values
PR interval |
0.12- 0.20 sec
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QRS Complex
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0.06 - 0.10 sec
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QT Interval
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0.30 - 0.46 sec
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Rate
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Pacing from the SA node generates a sinus rhythm.
- Normal sinus rhythm is 60 to 100 beats per minute. |
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When the Sa node paces at a rate less than 60 bpm, it is called a
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sinus brachycardia
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When the SA node paces at a rate greater than 100 bpm, it is called a
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trachycardia
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Rhythm is either
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regular or irregular.
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Sinus Arrhythmia
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The Sa node paces irregularly
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The R-R interval is
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irregular
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P wave is normal, PR interval is normal,
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QRS complex is normal
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Atrial Arrhythmia's
P Waves |
differ in appearance from sinus P waves, indicating irregularity in atrial contractions
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Premature Atrial Contraction (PAC)
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A single complex occurs earlier than expected, then normal sinus rhythm resumes
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Atrial Fibrillation (A-fib)
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many sites in the atria are generating depolarization waves
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No distinguishable P wave
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PR interval not measurable
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Rate 100 - 160 bpm
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Rhythm - regular
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Atrial Flutter
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P wave replaced with multiple flutter waves
- Rate around 110 bpm - Rhythm - regular |
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First Degree AV Block
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Caused by delayed conduction through the AV node
- Characterized by prolonged PR interval (>5 small blocks or 0.20 sec) |
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Ventricular Tachycardia
Rate 180 - 190 bpm |
Rhythm regular
- Abnormal tissues in the ventricular generate rapid contractions. - Poor cardiac output results. ***This is an emergency situation |
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Ventricular Fibrillation (V-Fib)
Rate: |
300 +
Disorganized electrical signals caused the ventricles to quiver instead of contract - There is no blood being pumped to the brain or body - Use the defibrillator ASAP!! |
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Myocardial Infarct (MI)
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Rhythm - Regular
Rate- 80 bpm P wave- normal, QRS complex, - normal ST segment does not return to baseline |
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Asystole
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Rhythm - flat
Rate- 0 bpm Carry out CPR and defibrillation |
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Coronary Artery Disease (CAD)
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Blockage of one of the coronary arteries by plaque and cholesterol deposits
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PTCA
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Percutaneous transluminal coronary angiography
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The coronary arteries are accessed via skin incision, and the procedure is performed within the blood vessels.
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Radioplaque dye is used to show coronary arteries
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Stents and ballon catheters are
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deployed through the incision if needed
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Stents
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a tube use to prevent flow constriction
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Ballon Catheter
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A catheter with an inflatable ballon at its tip used to open or enlarge a blocked artery
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Stenting
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LOOK AT P.P
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ACID
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Automated implanted cardioverter defibrillator
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Pacemaker
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Cardo Terminology
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CABG
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Coronary artery bypass graft
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CHF
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Congestive heart failure
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Dyspnea
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difficulty breathing
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MI
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Myocardial infarction
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Angina
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Chest Pain
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