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63 Cards in this Set

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which part of the bp is related to resistance?
volume?
only time this doesn't apply?
resistance = systolic
volume = diastolic
doesn't apply w/ eisenmonger
what is eisenmongers?
L to R shunt causing pulm HTN which reverses everything so now have R to L shunt
side of heart w/ decr pressure and resistance? - consequence (in relation to the valve opening/closing)
R
valves will open first and close 2nd
side of heart w/ inc pressure and resistance? consequence (in relation to the valve opening/closing)
L
valves will close first, open 2nd
when inhale, which side of heart gets inc volume? why?
R
b/c when inhale, bring in O2, and will cause vasodilation of the pulm vessels
when exhale, which side of heart gets inc volume?
exhale - L
3 physio effects for wider S2 split
bring in inc O2
inc volume in RV
delay opening and therefore closing of pulm valve
only dz to give FIXED wide S2 split
ASD
opening snap?
Tricuspid stenosis
Mitral stenosis
ejection click?
Aortic stenosis
Pulmonic stenosis
Midsystolic click
when does it occur when stand up?
when lay down?
MVP
standing: closer to S1
lay down: closer to S2
Reasons for soft S1
one of the valves isn't closing:
Tricuspid regurg
Mitral regurg
Valve is missing
Soft S1 w/ cyanosis
tricuspid atresia
mitral atresia
Reasons for loud S1
stiff valve bangs shut (tricuspid / mitral stenosis)

ventricle contracting harder
Reasons for soft S2
one of the valves isn't closing or valve is missing
Aortic regurg
Pulmonic regurg
Soft S2 and cyanosis
Aortic / pulmonary atresia
Reasons for loud S2
stiff valve (aortic / pulmonic stenosis)

high pressure in front of the valves
Reasons for S3
volume overload
dilated ventricle
decompensation
when is S3 normal?
adolescent females
pregnant women
Reasons for S4
pressure overload
hypertrophy
compensation
at what point do females have an inc incidence of heart dz? why? what do they complain of?
at menopause - due to decr estrogen - complaint = GI issue
what does estrogen cause the liver to produce?
lipoproteins
TBG
Angiotensinogen
Clotting factors (esp fibrinogen; not F11)
Pt comes in asking for OCPs. Current meds include thyroxin. What do you advise the pt?
inc thyroxin dosage to counteract hypothyroid effect - since estrogen inc TBG, more T3 will be bound and so have a transient hypothyroid effect
young adult comes in w/ an inc systolic bp (NL diastolic) - what are you thinkin?
drugs!
pressure in:
RA
RV
pulm artery
LA
LV
aorta
RA: -/0-8
RV: 16-24/0-8
pulm artery: 32-48/16-24
LA: 16-24/8-10
LV: 140/8-10 - must always be 20 higher than systolic in aorta
aorta: 120/80
what does the pulmonary capillary wedge pressure represent? what are the components?
total volume in lungs
components = volume from bronchial arteries and volume from pulm artery
max volume on right side of heart?
min volume on left side of heart?
max on R = 8
min on L = 8
AVO2 diff in:
RA, RV, LA, LV, pulm artery
RA: 75
RV: 75
LA: 100
LV: 97
pulm artery: 75
reason for difference in AVO2 difference between LA and LV
bld lost to the thesbian veins
SV, HR, TPR
which changes in whites? med?
blacks/hisp? med?
elderly? med?
whites: HR - give b-blocker
blacks/hisp: SV - give diuretic
elderly: TPR - CCB
how to treat inc ICP?
1. put on ventilator (inc O2, decr CO2) - vasoconstricts vessels in CNS
2. mannitol - gets rid of extra volume
3. acetazolamide - decr CSF
4. Burr hole - creates vacuum for brain to move back into place
all systolic murmurs
why?
AS, PS, MR, TR, VSD
valves that are supposed to be open or valves that should be closed
holosystolic murmurs
TR, MR, VSD
holosystolic inc on inspiration
TR
holosystolic inc on expiration
MR, VSD
holosystolic inc on expiration w/ radiation to axilla
MR
systolic ejection murmur radiating to the neck
AS
systolic murmur inc when lean forward, making fist, or squatting
AS
crescendo-decrescendo murmur
AS
Young athlete suddenly dies on the field - histo?
fibers are hypertrophied and disorganized
young boy comes in for physical required b/f basketball season. auscultation shows murmur louder upon standing or w/ valsalva. doc also notices radial pulse w/ 2 peaks. what do you tell young boy?
can't play. has hypertrophic cardiomyopathy.
rx w/ beta blocker and fluid
ECHO whole family
blowing diastolic murmur
AR, PR
blowing murmur inc on insp
PR
blowing murmur inc on exp
AR
murmur that can cause another murmur (2 of em)
AR - causes MR
PR - causes TR
diastolic murmur w/ widened pulse pressure
AR
diastolic murmur radiating to the back
PR
diastolic rumbles
TS
MS
cause of CHF (in terms of muscle)
overstretched muscle fibers due to inc EDV and ESV

what happens to contractility?
decr so decr CO and EF
if decr CO, what's consequence?
shock
CHF after MI - MCC?
left coronary artery infarct

how much myocardium is lost?
what's the ejection fraction?
lose 40%
EF: <45%
pt comes in w/ orthopnea and crackles on lung exam - dx?
left sided heart failure
pt comes in w/ bilateral lower extremity edema and hepatomegaly - lungs are clear on auscultation - dx?
right sided heart failure

MCC?
left sided heart failure
S3 heart sound
dilated cardiomyopathy

what happens to contractility?
decr
S4 heart sound
hypertrophic cardiomyopathy

what happens to contractility?
treatment?
inc

rx: beta blocker to decr HR to allow for max filling time
which type of heart failure is a systolic dysfxn? diastolic?
systolic: dilated
diastolic: hypertrophic
ways to rx CHF (general)
give O2
decr volume
inc contractility
decr TPR
ways to decr volume in CHF?
give furosemide
decr Na intake
decr volume intake

why is furosemide the best loop to give?
b/c hearing loss is reversible and also dilates lymphatic channels so lose volume faster
ways to inc contractility in CHF
digitalis
dobutamine
dopamine

which do you give in hospital? which to send home with?
to send home: digitalis
in hospital: the other 2
ways to decr TPR in CHF
ACEI
ARBs
B-blocker

which will decr mortality
beta blocker
MOA of digitalis
blocks Na/K pump by competing w/ K+ - causes inc Ca++ so inc contractility

also stimulates the vagus

side effects?
blue/yellow around light
ventricular arrhythmias initially from inc Na inside -> narrow QRS
optic nerve can swell from trapped Na
dig toxicity if have hypokalemia or hypercalcemia
how does MOA of dopamine change when give at low dose or high dose
low dose - will be D1 and beta 1 agonist
at high dose, will work at alpha 1
what 3 drugs can we use in heart failure that will decr mortality?
beta blockers
spironolactone (how)
nitrates (in who)
spironalactone will block cardiac fibrosis caused by aldosterone from ACEI use
nitrates used in blacks