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75 Cards in this Set

  • Front
  • Back

Heart Failure Definition

heart unable to maintain adequate circulation

Pulmonary edema definition

accumulation of fluid in the alveoli and interstitial spaces of the lung

class I heart F

no symptoms with activity

class II heart F

symptoms with ordinary exertion

class III heart F

symptoms with minimal exertion

class IV heart F

symptoms at rest

low-output heart F can occur

initially on left or right side of heart

left-sided heart F

inadequate CO

right-sided heart F

inadequate right ventricle output and systemic venous congestion

forms of left-sided heart F

systolic F


diastolic F



left-sided systolic F

ejection fraction < 40%


pulmonary congestion


peripheral congestion

left-sided diastolic F

inadequate relaxation or 'stiffening' prevents ventricular filling

high-output failure

CO normal


unable to meet tissue demands

symptoms of heart F

fatigue


SOB

causes of high-output failure

severe anemia


hyperthyroidism


arteriovenous fistula


beriberi


Paget's disease

Beriberi

deficiency of thiamine (Vit B1)

Paget's disease

abnormal breakdown and regrowth of bones, which develop an excessive amount of blood vessels



arteriovenous fistula

an abnormal connection between an artery and a vein

risk factors for left-sided heart failure

hypertension


coronary artery disease


angina


MI


mitral and aortic valvular disease

risk factors for right-sided failure

left-sided failure


right ventricular MI


COPD


pulmonary fibrosis



risk factors for high-output heart failure

increased metabolic needs


septicemia


anemia


hyperthyroidism



risk factors for cardiomyopathy

coronary heart disease


infection of inflammation of heart muscle


various cancer treatments


prolonged alcohol use


heredity

left-sided heart F s/s

dyspnea, orthopnea,nocturnal dyspnea


fatigue


displaced apical pulse (hypertrophy)


S3 heart sound (gallop)


pulmonary congestion(dyspnea, cough, bibasilar crackles)


frothy sputum(can be blood tinged)


ALOC


manifestations of organ F (i.e. oliguria)



right-sided heart F s/s

JVD


ascending dependent edema


abdominal distension, ascites


fatigue,weakness


polyuria at rest (nocturnal)


hepatomegaly and tenderness


weight gain

four types of cardiomyopathy

dilated***


hypertrophic


arrhythmogenic right ventricular


restrictive



cardiomyopathy s/s

fatigue/weakness


heart F


>left-sided with dilated type


>right-sided with restrictive type


S3 gallop


cardiomegaly (more severe w/dilated type)


angina (hypertrophic type)

dilated cardiomyopathy

starts in the left ventricle. The heart muscle begins to stretch and become thinner. This causes the inside of the chamber to enlarge. The problem often spreads to the right ventricle and then to the atria as the disease gets worse.

hypertrophic cardiomyopathy

myocardium becomes abnormally thick

arrhythmogenic cardiomyopathy

breakdown on the myocardium surrounding the right ventricle

elevated hBNP occurs in

heart failure

hBNP < 100 pg/dL

no heart failure

hBNP between 100 - 300 pg/dL

suggests heart failure

hBNP > 300 pg/dL indicates

mild heart F



hBNP >600 pg/dL indicates

moderate heart failure

hBNP > 900 pg/dL indicates

severe heart failure

drop in SvO2 indicates

worsening cardiac functioning

heart failure generally results in

increased CVP


increased PAWP


increased PAP


decreased CO

diagnostic testing heart failure

hemodynamic monitoring


us


transesophageal echocardiography TEE


chest x-ray


ECG


cardiac enzymes, electrolytes, ABGs

digoxin toxicity s/s

N/V, loss of apetite


fatigue


muscle weakness


confusion


visual disturbances


bradycardia


arrhythmias



treatment of life-threatening digixon toxicity

administer digoxin immune Fab (Digibind)

meds to correct arhythmias from digoxin toxicity

lidocaine


procainamide


quinidine


propranolol


phenytoin

meds used to treat heart failure

diuretics


afterload-reducing agents


inotropic agents


beta-adrenergic blockers


vasodilators


hBNPs


anticoagulants

diuretics

furosemide


bumetanide


hydrocholorothiazide


spironolactone

loop diuretics

furosemide (Lasix)


bumetanide (Bumex)

thiazide diuretics

hydrochlorothiazide (Hydrodiuril)

K-sparing diuretics

spironolactone (Aldactone)

administer furosemide IV no faster than

20 mg/min

loop and thiazide diuretics may cause

hypokalemia,


K supplements may be nec

teach P taking loop and thiazide diuretics

ingest foods and drinks high in K

after-load reducing agents action

help the heart pump more easily by altering the resistance to contraction

after-load reducing meds

ACE inhibitors


angiotensin receptor II blockers


Ca channel blockers


phosphodiesterase-3 inhibitors

after-load agents are

contraindicated for P w/ renal deficiency

ACE inhibitors

enalapril (Vasotec)


captopril (Capoten)

angiotensin receptor II blockers

larsartan (Cozaar)

calcium channel blockers

diltiazem (Cardizem)


nifedipine (Procardia)

phosphodiesterase-3 inhibitors

milrinone (Primacor)

monitor P taking these cardiac meds for hyptension for 2 hours following the first dose

ACE inhibitors

ACE inhibitors can cause

angioedema


decreased sense of taste


rash

angioedema

swelling of the tongue and throat

monitor P taking ACE inhibitors for

increased levels of K

inform P that ACE inhibitors

can cause dry mouth


BP needs to be monitr for 2 hrs post initial dose


notify MD:


if rash,


decreased sense of taste or


swelling of face or extremeties occurs

inopropic agents

digoxin (Lanoxin)


dopamine/dobutamine (Dobutrex)


milrinone (Primacor)

digoxin

take apical pulse for 1 min


hold med if pulse is less than 60 bpm


notify provider

monitor for N/V

digoxin

dopamine, dobutamine, milrinone

administered IV


closely monitor: EKG, BP and urine output

P instructions for self medication of digoxin

count pulse for 60 seconds


take med at same time/day


do not take at same time as antacids(2 hrs apart)


report s/s toxicity


have med levels and K levels taken regularly



common beta-adrenergic blockers (beta-blockers)

carvedilol (Coreg)


metoprolol (Lopressor)

beta-blockers my be used for P who has

sustained increased levels of sympathetic stimulation and catecholamines


> includes chronic heart F

nursing considerations for P taking beta-blockers

monitor BP, pulse, activity tolerance, orthopnea


check orthostatic blood pressure readings

client ed for P taking beta-blockers

weigh daily


regularly check BP


follow MD instructions on increasing dosage

vasodilators

nitroglycerine (Nitrostat)


isosorbide mononitrate (Imdur)

vasodilators effect

prevent coronary artery vasospasm and reduce preload and afterload, decreasing myocardial oxygen demand

vasodilators used to treat

angina


contol BP



vasodilators can cause

orthostatic hypotension



common side effects of nitroglycerin (Nitrostat)

HA


encourage P to sit and lie down slowly