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25 Cards in this Set

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  • Back
Localizing infarcts: which ECG leads for posterior wall infarcts?
Might find larger, abnormal R waves in V1-V3
Localizing infarcts: which ECG leads for anterior wall infarcts?
V1-V4
Localizing infarcts: which ECG leads for lateral wall infarcts?
aVL, I, V5, V6
Localizing infarcts: which ECG leads for inferior (apical) infarcts?
II, III, aVF
When the QRS shape/duration indicates abnormal ventricular activation, four general possibilities
1.Abberancy (2’ to L/R block, AV block, hyperkalemia, pharmacological block)
2.Ventricular foci
3.Alternative conduction pathway (pre-excitation, bypass of AV node)
4.Pacing
Two leads used to determine electrical axis (rule of thumb)
aVF (+ means below the x-axis, - means above the x axis) and I (+ means to the right of the y-axis, - means to the left of the x-axis)
Give the estimates for QRS duration under normal, conduction delayed, and conduction blocked (pacing, preexcitation, focal) circumstances
Normal = 100 ms or less
Delayed = 100 ms – 120 ms
Focal = +120 ms
Normal duration of PR interval
120 ms – 200 ms
Rule of thumb for normal length of QT interval
QT should be less than half the RR interval
Leads to assess atrial function
V1 and V2
When looking at V1 and V2 for atrial function, if sinus rhythm is not found, look for these kinds of atrial problems (4)
1.Atrial flutter
2.Atrial fibrillation
3.Atrial tachycardia (SVT)
4.Retrograde (p wave proceeding QRS)
Hyperkalemic ECG changes (4)
1.peaked t waves
2.merging of QRS/T
3.loss of p wave
4.”sine wave” pattern
Hypokalemic ECG changes (2)
1.flattened T waves
2.positive U wave proceeding T wave
ECG hallmarks of RBBB (3)
1.V5/V6: greater negative deflections in QRS
2.V1/V2:extra,large R’ wave
3.Widened QRS
ECG hallmarks of LBBB (3)
1.V5/V6:wide, deep Q waves in QRS
2.wide QRS
3.biphasic waves in V5/V6
Intracardiac electrode ECG allows for more precise measurement of two intervals - what are they?
AH: atrial to bundle of His conduction time (AV conduction time)
HV:bundle of His to ventricular conduction system, typically 40 ms (bundle conduction time)
2’ block (occasional skipped QRS complexes) is due to a conduction problem in the _______ or the ______. The former is more common. These are known as Mobitz Type I and Type II, respectively.
Problem in AV conduction (type I)
Problem in bundle conduction (type II)
Things that cause AV block/delay
Hyperkalemia, RV/Inferior ischemia, digitalis, Beta Blockers, Parasympathetics
In 3’ heart block (dissociated p and QRS waves), how can one tell how far downstream the rescue focus is?
Width of the QRS complex (wider, further downstream)
junctional, fasicular, ventricular
L Bundle
L bundle gives off posterior fascicle, then anterior fascicle
“Tri-fascicular” block
2 fascicle block + prolonged PR interval
Wandering pacemaker
Various atrial foci (p’ waves), less than 100/min, irregular QRS complexes
Junctional Escape
Lone QRS complexes due to failure of upstream foci or proximal AV node block; slower rate (40-60 bpm); retrograde P waves may be seen, with possible inversion in leads with positive QRS’s
Atrial/Junctional Irritability VS Ventricular Irritability
AJ = sympathetics, less parasympathetics, stimulants
Ventricular = hypoxia, hypokalemia
Atrial flutter
Atrial Flutter: atrial foci origin; saw-tooth appearance; 250-350 atrial beats per minute; usually one P wave conducts down to the ventricles for every 2, 3, or 4 P waves fired
-can't have too fast of a heart rate with flutter (ventricular contraction requires AV conduction)