• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/15

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

15 Cards in this Set

  • Front
  • Back
Causes of vericose veins
conditions that produce venous stasis or increased intra-abdominal pressure (i.e. pregnancy, obesity, constipation, tight clothes), congenital weakness of valves, family history, occupations that mandate long periods of standing.
ANY conditions that destroys or distends a valve may lead to VV
what are vericose veins
dilated, tortuous veins engorged with blood and resulting from improper venous valve function.
Primary: originate in superficial veins
Secondary: originate in deep veins
S/S of vericose veins
purplish ropelike veins particularly in calves (venous pooling)
edema of calves and ankles
leg heaviness (worse in evening & warm weather)
dull aching in legs after prolonged standing or walking
ache during menses as a result of increased fluid retention.
Deep Vein Thrombosis (DVT)
Thrombophlebitis
an acute condition characterized by inflammation & thrombus formation; may occur in deep or superficial veins.
DVT may affect small veins (i.e. soleal venous sinuses) or large veins (i.e. vena cava, femoral, iliac, subclavian).
Usually progressive  Pulmonary Embolism (PE)
Superficial thrombophlebitis: rarely causes PEs.
DVT causes
Causes: idiopathic but usually results from endothelial damage, accelerated blood clotting, reduced blood flow.
Predisposing factors: prolonged bed rest, trauma, surgery, childbirth, use of hormonal BC.
Superficial thrombophlebitis
caused by infection, IV drug abuse, trauma, and chemical irritation due to extensive use of IVs for medications.
pathophys of DVT's
thrombus formation is faster in areas where bloodflow is slower – greater contact between platelet and thrombin accumulation. Thrombus expands rapidly inflamed endothelium  fibrosis.
S/S of DVT's
may be asymptomatic but could include: pain, chills, fever, swelling/cyanosis of the affected arm/leg due to impaired circulation, heat, tenderness along length of affected vein, possibly varicose veins due to impaired venous return.
Hypertension defined:
an elevation of either systolic and/or diastolic pressure.
primary HTN
essential, begins slowly but progresses to a serious state; if untreated, can become life-threatening. Genetics + environmental factors.
secondary HTN
usually the result of renal disease or some other pathology.
Causes of essential HTN
Advancing age- elderly people may have Isolated Systolic Hypertension (ISH) due to loss of vascular elasticity.
Diabetes mellitus
Excessive alcohol consumption
Excess renin
Family history
High saturated fat intake
High sodium intake
Mineral deficiencies (Ca, K, Mg)
Obesity
Race (more common in blacks)
Causes of Secondary HTN
1. brain tumor
2. coarctation of Aorta
3. excessive alcohol consumption
4. hormonal BC, cocaine, sympathetic stimulants, estrogen replacement therapy, NSAIDs, Cushing’s syndrome, pre-eclampsia/eclampsia, renal artery stenosis.
BP is?
the product of peripheral resistance and cardiac output.
cardiac output is increased by?
conditions that increase HR, Stroke Volume, or both.