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41 Cards in this Set

  • Front
  • Back
Describe the mitral stenosis?
narrowing of the mitral vale orifice
Most common etiology is Mitral stenosis
Rheumatic heart disease
etiologies affecting the mitral valve orifice b/c of Mitral stenosis
Rheumatic heart disease
o Calcification of the mitral annulus (may extend to base of leaflets)
o Obstruction due to thrombus, tumor (myxomas), vegetations
o Congenital MS
 Parachute mitral valve where only one papillary muscle is attached to the MV
what is parachute mitral valve? and is the eitology of what disease?
only one papillary muscle is attach to the mitral valve
and is one eitology of mitral stenosis
Describe RHD
Leaflets become scarred and contracted. Adhesions fuse the commissures and restrict motion of both leaflets. The leaflets become tethered (confined) in a downward position, creating a funnel-shaped structure. The narrowed orifice inhibits flow from the LA to the LV and thus becomes a barrier to normal flow. This results in increased diastolic left atrial flow.
Describe the heodynamics of MS
•  in MVA causes a diastolic pressure gradient (pressure difference) between the LA and LV
 Valve area of 2 cm2 or more = minimal transvalvular pressure gradient and patient is usually asymptomatic
 Valve area < 2 cm2 = significant pressure gradient. This leads to pressure overload of the LA, pulmonary vascular bed, and RV
•  in LA pressure may lead to LA enlargement
 occurs with at least moderate MS
• LA enlargement/pressure may cause atrial fibrillation
• Chronic increase in LA pressure causes an increase in pulmonary resistance, leading to reduced lung compliance and difficulty breathing. The right heart has to work harder to pump blood into the lungs. Tricuspid regurgitation usually develops when the right heart is affected.
Other complications of MS include:
Thrombus formation Due to LA enlargement, loss of function, and stasis of blood flow. LAA common site for thrombus
 LA enlargement causes stretching of the annulus and MR
 Pulmonary hypertension occurs because of the increased resistance to pulmonary venous flow.
What is the common site for thombus when mitral stenosis occur?
Left atria area
What is the sign/ and symptoms for M/s
1. Dyspnea upon exertion
• Dyspnea and pulmonary edema occur because of the changes in the pulmonary vasculature, which reduce lung compliance
2. Hemoptysis
• Increased pulmonary pressure results in the rupture of pulmonary capillaries, leading to bleeding or hemoptsis
3. CP
• Pulmonary HTN can contribute to CP
4. Palpitations
• A-Fib
5. Orthopnea
6. Paroroxysmal nocturnal dyspnea
7. Fatigue
8. Syncope
9. Hoarseness
• Impingement upon the laryngeal nerve due to LA enlargement can lead to hoarseness
10. Stroke
11. EKG indicates A-Fib, LAE, LVH
12. Auscultation characterized by diastolic, low-pitched, rumbling murmur
Dyspnea and pulmonary edema occur because of the changes in the pulmonary is what disease and describe what pathology of the heart
dyspnea upon exertion and describe MS
What can contribute to chest pain during MS?
pulmonary HTN
increased pulomanry pressure results in the ruptrue of pulmonary capillaries, leading to bleeding called?
hemoptysis in mitral stenosis
impingement upon the laryngel nerve due to LA enlargement can lead to what?
hoarseness (eitology of Ms)
EKG in MS indicate what?
A-fib,, LAE, LVH
diastolic, low-pitched, rumbling murmur is called?
ausculation (eitology of MS)
What are the four treatment for ms
1. Prophylactic antibiotic therapy
2. Commissurotomy
3. Percutaneous ballon mitral valvuloplasty
4. Mitral valve replacement
2-D of MS
1. Thickened MVLs (increased echogenicity)
2. Restricted valve motion
3. Diastolic doming of the anterior mitral leaflet (PLAX)
• Hockey stick appearance
o Due to the high pressure overload in the LA and also fusion between the tissues of the leaflets
• Usually seen with at least moderate MS
4. Commissural fusion (PSAX)
5. Anterior motion of the posterior MVL
♠ Due to tethering
6. Decreased mitral valve orifice
• Significant = 1 cm2
7. Fibrosis and shortening of chordae tendineae
8. Small LV (isolated MS)
9. LAE
• Spontaneous contrast (smoke)
• Thrombus formation
10. Pulmonary Hypertension (P HTN)
11. RVH and dilatation
What is the appearance of the AMVL in diastolic of MS?
hockey stick b/c hgih pressure in LA and fusion b/w the tissues of the leaflets
Describe M-mode of MS
1. Thickened mitral leaflets (increased echogenicity)
2. Decreased E-F slope (0 – 30 mm/s indicates significant MS)
3. Anterior motion of the posterior mitral leaflet
4. Decreased A wave of the MVL
5. Reduced D-E excursion (< 15 mm)
6. LAE
7. Pulmonary HTN
8. RV hypertrophy and dilatation
• Flattened or paradoxical IVS
• Due to volume overload/pressure
9. Early diastolic dip of IVS
• Due unrestricted flow into the RV
indicate significant of MS in E-F slop
decrase EF slop (0-30)mm/s
reduced d-e excursion in MS to what?
less than or equal to 15mm
describe in 2D of MVA planimetry of MS
1. Taken in the PSAX at the MV level
2. The very tips of the MVLs should be centered in the middle of the image
3. The measurement is performed at the beginning of diastole when the MV is at its widest excursion
4. The valve area is traced along the inside border of the mitral orifice
What is the normal CSA for MS
4-6cm2
What is the severe CSA for MS
< 1.0 cm2
What is mild CSA of MS
What is moderlate CSa of MS
mild-1.5-2.5
moderate-1-1.5
Pitfalls of 2-D Planimetry
1. Improper imaging plane
2. Inappropriate gain settings
3. Poor lateral resolution – echo dropout; borders are not visualized well
4. Shadowing from calcification – can’t delineate borders
5. Mitral Commissurotomy (surgical procedure to increase mitral orifice size)
Doppler Features of MS
1. Spectral broadening of diastolic flow (turbulent)
2. Increased diastolic velocities (>1.3 m/s)
3. Decreased E-F slope (diastole): Flattened slope
• Increased pressure half-time
4. Increased A wave
5. Pulmonary venous flow pattern
• Decreased systolic flow, prolonged duration
• Increased pressure half-time of diastolic flow
6. Pulmonary Hypertension
• SPAP (systolic pulmoanry artery pressure)
• Use tricuspid regurgitation to calculate RV systolic pressure
• RV systolic pressure = 4 x (Vmax TR2) + RAP
• Substitute 10 mmHG for RAP (right atria pressure)
In MS describe A wave related to doppler and M mode?
Doppler-increased A wave
M mode-deceased awave
doppler MV calculation determine what
mean pressure gradient
peak pressure gradient
MVA
Describe pressure half time
 rate of pressure decline across the stenotic mitral orifice is determined by the cross-sectional area of the orifice: the smaller the orifice, the slower is the rate of pressure decline.
 Defined as the time interval (milliseconds) between the maximum early diastolic transmitral pressure gradient and the time point where the pressure gradient is half the maximum value.

MVA = 220
P1/2


1. The Doppler signal is obtained demonstrating the highest velocity
2. The velocity representing ½ the original pressure gradient is determined by dividing the peak velocity by the square root of 2 (1.414) or multiplying the peak velocity by 0.71
3. A line is drawn along the slope until it intercepts the half value
4. Vertical lines are drawn from the peak velocity to the baseline and from the half value to the baseline
5. The distance between these two lines represents time on the horizontal axis and is equal to the pressure half-time.
6. This number is divided into 220 to obtain the MV area.
what happen to the pressure half time if the smaller the orifice
increase pressure 1/2 time b/c take longer to run through
What is pressure half time serenity values
Severe
> 220 msec
What is normal value of pressure half time of MS?
30-60 msec
What is the normal and severe value of MVA of MV (MS)
normal 4-6cm2
sev-<1cm2
What is the mild and severe value of mean pressure gradient
< or equal 5 -mild
>12 severe
describe color flow doppler of MS
• Increased velocities results in aliasing
• Turbulent flow results in mosaic color
• MS demonstrates a “candle flame” in the LV
Pitfalls of pressure half time of MS
• Decreased pressure half time/increased mitral valve area
♠ Significant aortic insufficiency
♠ Decreased LV compliance
♠ Sudden change in LA compliance (e.g. tachycardia, acute MR)
• Increased pressure half time/decreased mitral valve area
♠ Decreased rate of LV relaxation
♠ Immediate post mitral valve balloon valvuloplasty
Complication of MS
♠ MR
♠ Pulmonary hypertension
♠ A-fibrillation
♠ LA thrombus and embolization
♠ Increased risk of endocarditis
♠ Decreased cardiac output
♠ Mitral annular calcification (MAC)
why is it MR during MS
• Due to shortened and thickened chordae tendineae
• LAE interferes with coaptation of the leaflets
.
MAC can mimic what?
pericardial effusion, masses ,or mitral stenosis
Describe mac (mitral annular calcification)
• May interfere with leaflet motion if calcification involves MVLs and/or AVLs
• Complications associated with MAC include MR, LAE, and MS