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28 Cards in this Set
- Front
- Back
Etiology: Disturbances in automaticity
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Altered rates caused by various pacemaker cells.
Example: sinus bradycardia |
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Etiology: Disturbances in conduction
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Speed at which impulse travels through the SA>AV>Purkinje fibers.
Example: AV heart block |
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Etiology: Reentry of impulses
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Reactivated muscle for a second time by the same impulse.
Example: atrial fibrillation |
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Cardiac Dysrhythmias: Risk factors
* pathophysio - atrial kick |
Myocardial ischemia: angina and MI
Hypoxia Vagal stimulation (autonomic nervous system) Lactic acidosis Electrolyte imbalances Drug toxicity Shock |
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Clinical manifestations
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Reduced cardiac output
Palpitation, dizziness, syncope, pallor, diaphoresis, altered mental status, hypotension / shock, edema, oliguria, SOB, chest pain, fatigue, seizures Heart rate below 50 or above 140, very irregular heart rate, or rate that does not change with exercise. |
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Lead Patterns
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Electricity travels from the negative electrode to the positive one.
Lead II Right arm or shoulder lead is negative. Left leg or abdomen/lower chest is positive. Third lead is a ground. Follows the same direction as an impulse traveling from SA node to ventricles. MCL1 is a modification of a 12 lead’s V1 lead. |
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P wave
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First upright wave
Close to atrial contraction |
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QRS wave
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3 waves or less
Q is first downward wave, R is second upright wave, and S is second downward wave. Close to ventricular contraction. |
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P-R interval
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Space between beginning of P-wave and the beginning of the Q wave (if Q absent, then the R wave)
Impulse traveling from the SA node into the AV node. |
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ST segment, T wave
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Ventricular repolarization
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EKG analysis basic steps
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Calculate heart rate, which should be between 60-100.
Measure regularity of R waves (R to R interval) and P waves (P to P interval). Examine P waves for their preceding each QRS (R wave) and their sameness. Measure the P-R interval, which should be between 0.12-0.20 seconds. Measure the duration (or width) of the QRS, which should be less than 0.12 seconds. Examine the ST segment, which should be neither elevated or depressed. Examine the T wave, which should be upright and 1/3 the height of the QRS. |
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EKG paper
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EKG paper has small boxes on it.
As it comes out of the machine, the horizontal axis of the boxes measures time in seconds. Each small box represents 0.04 seconds. |
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0.12 seconds or more difference between different R-R intervals
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rhythm is irregular.
** R waves or QRS complexes are very close to ventricular contraction. |
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P-R interval
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between 0.12-0.20 seconds.
IF NOT - Indicates a slowing of conduction in the AV node. |
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QRS
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less than 0.12 seconds.
IF NOT - Indicates a slowing of ventricular conduction. |
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ST segment
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neither elevated or depressed IF NOT Indicates abnormal recovery of ventricle.
Seen in MI and other conditions. |
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T wave
T-wave is ventricular repolarization. |
should be upright and 1/3 the height of the QRS
ABN: Upside down or inverted is seen in myocardial ischemia. Peaked or flattened T waves are seen in hyperkalemia and hypokalemia. |
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Characteristics of normal sinus rhythm (NSR or SR)
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Rhythm
Regular P-P and R-R intervals, varying only up to 3 mm (less than 3 small boxes). Rate 60-100 beats / minute P waves (atrial contraction) One precedes each QRS (ventricular contraction) P-R interval: 0.12-0.20 seconds QRS complex: less than 0.12 seconds QT interval: less than 0.40 seconds |
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(NSR or SR)Outcome management
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Control the dysrhythmia rate.
Remove the dysrhythmia. Reduce potential complications. |
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Atrial dysrhythmias, automaticity: Sinus Tachycardia (ST)
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Etiology/risk factors
Heart failure, fluid loss, shock, respiratory distress, drugs, exercise, stress, pain Clinical manifestations ECG: NSR except for HR > 100 Outcome management Treat cause, O2, beta blockers |
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Sinus Bradycardia (SB)
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Etiology/risk factors
Vagal stimulation (including Valsalva), drugs, MI, hyperkalemia, athletes Clinical manifestations ECG: NSR except for HR < 60 |
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Sinus Bradycardia (SB)
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Outcome management
If symptomatic: Treat cause Atropine, epinephrine, or dopamine Temporary pacemaker |
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Premature atrial contractions (PAC)
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Etiology/risk factors
Ectopic foci Valve problems, CHF (atrial enlargement) Stress, CAD, medications, pulmonary problems Palpitation Clinical manifestations ECG: a P wave is early, differ from the other P waves, and an early QRS follows the early P. Outcome management Treat cause, O2 |
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Atrial dysrhythmias, reentry of impulses: Atrial Flutter
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Etiology/risk factors
Ectopic foci or rapid reentry, with atrial contraction up to 350 times per minute CAD, mitral valve disease, PE, cardiac surgery |
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Atrial Flutter: Clinical manifestations/ Outcome management
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Clinical manifestations
ECG: inverted or bidirectional, saw-toothed P-waves, with possibly a constant P-wave to QRS ratio such as 2:1 or 3:1 from AV blocking. Outcome management Cardioversion (similar to defibrillation) or Drugs: Convert to NSR: procainamide, flecainide, propafenone, dofetilide, ibutilide Slow ventricular response: verapamil, diltiazem, amiodarone, adenosine |
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Atrial Fibrillation
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Etiology/risk factors
Rapid chaotic atrial depolarization, up to 700 times per minute. No atrial contraction: loss of atrial kick (decreasing cardiac output 20-30%), thrombi formation in atria AV node blocking of some impulses Apical-radial pulse deficit Hypoxia, CHF |
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Atrial Fibrillation: Clinical manifestations
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Clinical manifestations
ECG: erratic baseline without P-waves, very irregular R to R intervals |
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Atrial Fibrillation: Outcome management
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Outcome management
Anticoagulants for thrombi Diltiazem, metoprolol, cardioversion |