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54 Cards in this Set

  • Front
  • Back

Etiology of burns

Thermal


Chemical


Electrical

Describe Thermal Burns

Result from direct/indirect contact with flame, hot liquid, or steam




Influenced by: Contact time, temp, type of insult

Describe Chemical Burns

Acids, Bases, Industrial accidents, assualts.


Most likely to cause full thickness damage

Chemical Burns severity are influenced by...

Alkaline - more severe than acidic


Contact time - continues until irrigated


Chemical Concentration


Amount of chemical

Describe electrical burns

Low and high voltage currents


Entrance wounds - depressed or charred


Exit wound - larger explosive




Skin may not be severely damaged despite deep tissue injury due to difference in resistance

What concomitant injuries can you have with electrical burns

Fractures, muscle necrosis, neurological injuries, cardiac, pulmonary, other organ failure

Electrical burn severity influenced by...

High voltage current causes more damage


AC burn injuries are more severe


Contact time

How to classify and characterize burns

Depth of burn


Total Body Surface Area


Severity

Describe depth of burn injuries

-In terms of degrees


-Level of tissue involved


-Usually not all one depth


-Infection can inc depth


-Chemical burns take 24-72 hours to fully develop

Describe SUperficial burns

"First Degree"


-Dry, bright red, or pink skin that blanches upon pressure


-No dermal vessel damage


-Resolves in 3-5 days without scarring




Ex: Sun burn, minor flash burn

Describe superficial Partial Thickness burns

"Superficial second degree burn"


-Painful.moist, weeping, blistered with local erythema and edema


-Blanches to pressure with immediate capillary refill


-Heal within 10-14 days with minimal or no scarring


Ex. Brief contact burns, flash burns, brief contact with dilute chemicals

Describe Deep partial thickness burns

"Deep second-degree burns"


-Mottled areas of red with white eschar, blistering possible, may have areas of insensitivity


-Blanches to pressure with Slow capillary refill


-May take 3 or more weeks to heal


-Scarring, pigment changes, contractures possible


Ex. Severe sunburn, scald, flash burn, brief contact with dilute chemicals



Describe Full thickness burns

"Third Degree Burns"


-Initially look red then become mottled white/black dry, leathery eschar, and painful


-Burned areas insensate to light touch


-Scarring and contractures likely


-Most require surgical debridement and grafting


Ex. Prolonged contact with flam, immersion scald injury

Describe Subdermal Burns

"Fourth Degree burns"


-Charred, mummified appearance


-Exposed deep tissues


-Burned areas insensate to light touch


-May have permanent nerve damage


-Req surgery (fasciotomy, escharotomy, grafting) and possible amputation




Ex. Electrical burn, s trong chemical burn

What is TBSA

Total Body Surface Area


-Rule of Nines


-Lund and browder chart


-Palm method - size of palm equals 1%

Describe the Lund Browder Classification

-Takes into account variation of body proportion from child to adult


-Appropriate for children under age 16


-Preferred by pediatric burn units

Describe the Lund and Browder Chart

-Most accurate to calculate TBSA


-Subdivides body segments into % TBSA based on age


-Requires chart to calculation

Describe the rule of nines

Divides body into areas of 9% or multiples of 9s


-perineum is 1%


-Head, front and back of each UE and LE


-Ant - post trunk each equal 18%




-consistently overestimates the size of burns in young teenagers

Describe the palmar method for TBSA

Uses area of plantar surface of the patients hand to determine burn size




Highly unreliable, inaccurate

How is burn severity determined

(Size, depth, age)




Minor: out patient


Moderate: In patient


Major: Specialized burn unit

Adult percentages for FT and PT burn classficiation

(FT% - PT%)


Minor: <2 , <10


Mod: 2-5 , 10-20


Major: >5 , >20

Child percentages for FT and PT burn classficiation

(FT% - PT%)


Minor: <1 , <5


Mod: 1-5 , 5-10


Major: >5 , >10

What are the "Zones" of the integ system?

Zone of coagulation


Zone of stasis


Zone of Hyperemia

Describe Zone of coagulation

-Central portion, irreparable damage


-Characterized by coagulation, ischemia, necrosis

Describe Zone of Stasis

-Area of cellular injury and compromised perfusion


-Conversion: Widening and deepening of necrosis



Describe Zone of Hyperemia

Outer edges, minimal cellular injury

Complications of bandaging burns

Bandages that are too tight


Undue pressure from splints


Improper pt positioning

Effect of burns on Cardiovascular system

Burn shock - massive fluid shift causing hypovolemia and edema


Results in dec blood volume --> necrosis, organ failure, and death

Who is at risk for burn shock?

Patients with more than 15% TBSA

What to be aware of regarding the CV system and its relation to burn care

-Fluid resuscitation is vital


-BP generally dec due to hypovolemia


-Resting HR 100-120 bpm


-Monitor peripheral pulses


-Must monitor and manage edema

Effects of burns on the pulmonary system

Inhalation inuries cause up to 20% of burn deaths


-Smoke inhalation, carbon monoxide, pulmonary edema, pneumonia, ARDS

What to be aware of regarding the pulmonary system and its relation to burn care

Suspect lung involvement if signed facial hair, carbonaceous sputum, closed space injury, burns to face/neck/torso




Monitor for signs of breathing difficulties


Monitor oxygen saturation


Encourage aggressive pulmonaray hygiene

Effects of burns on metabolism

Basal metabolic rate doubles or triples


Inc in core temp


Sustained hyperglycemia


Inc fat catabolism


Dec in body mass


Peaks 7-17 days post major brun injury




Greater nutritional needs

Effects of burns on the immune system

75% of burn pt deaths due to infection


-Sepsis and infection common


-- Endogenousand exogenous bacteria


--Decreasedtissue perfusion reduces immune system effectiveness


--Neutrophilsless effective


--Eschar,blister fluid, residual topical agents excellent medium for bacterial growth


--Openwound for extensive periods of time



What to be aware of regarding the immune system and its relation to burn care

Aggressive debridement and rapid skin coverage is necessary to reduce risk of infection




Prophylactic topical anitmicrobials

Other system complications due to burns

Multi-organ system dysfunction


CNS dysfunction


Acute kidney failure


GI dysfunction/peristalsis/ileus/ulcers


-Encourage early, controlled mobility

Psychological dysfunction and burns

PTSD


Anxiety/depression/disturbed sleep


Extremely common

What to be aware of regarding psychological dysfunction and its relation to burn care

Pt. education, control pain and anxiety


Promote pt independence


Involve family and friends

PT interventions for burns

Coordination, communication, and documentation




-Reinforce goals


-Participate in pt rounds


-Pt. edu


-Give pt control over their rehab

Patient/Client related instruction

-Ways to control pain


-What to expect during procedures


-How to care for wounds


-Proper positioning


-Edu on importance of skin care/scar management

Precautions for procedural interventions

Screen for domestic violence


Anticipate/prevent complications when possible


-Contractures


-Infection


-Deconditioning


-Pulmonary dysfunction


-Pressure ulcers


-Adequate pain control

Debridement for wounds

-Foreign debris, residual topical agents, exudate, hair, necrotic tissue


-Remove blisters (open and closed)


-Consider enzymatic debridement is appropriate

Infection control with burns

Sterile technique for large TBSA burns


Topical antimicrobials are standard


-Silver sulfadiazine


-Mafenide


-Bacitracin




Signs of infection


-Inc erythema/pain, foul odor, purulence


-Inc in necrosis, fever, inc tachycardia

Dressings for burns

Most common


-Topical antimicrobial covered with nonadherent impregnated gauze, bulky gauze dressing




Limit bulk to allow movement


Short stretch compression wrap to dec edema and scarring

Scar management for burns

Moisturizer


Protect from friction/shear


Scar mobilization


Compression - req if wound take 3+ wks to close


Consider silicone gel sheets/pads, ultrasound, paraffin


Darker-skinned individuals > incidence of hypertrophic scarring and keloids

What is the vancouver scar scale

Rates 4 scar qualities


-Vascularity


-Pliability


-Pigmentation


-Height




Scores range from 0-14


Lower score indicates less severe scar tissue

Procedural interventions

ROM


Mobility training


Breathing exercises


Aerobic Exercise - target HR 50-70% of max predicted HR

Devices and equipment for burns

Splints/Braces


-Immobilize and protect grafts fractures, peripheral nerve injuries




Dynamic Splints


-Assists with contractures

Physical agents and modalities

Whirl pool


Pulsed lavage w/ suction


Ultra sound


Paraffin

Medical Interventions

Pharmacological management


-Ensure adequate control of pain and anxiety


-Time procedures with medications

Surgical interventions

Debridement


-Early debridement often performed on pts with medium and large FT burns


-Escharotomy - Incision through eschar and subcutaneous tissue to release tissue constricting circulation


-Fasciotomy - incision through fascia to release pressure/improve distal circulation


-Skin Graffting - Mainstay for FT or DPT burns/autographs/xenografts for temporary coverage


-Split thickness grafts - removes epidermis and part of dermis


-FT grafts - more durable and most cosmetic



What can cause graft failure

Infection


Eschar


Insufficient immobilization


Fluid collection under graft


Prior surgery - areas that have been previously compromised

Surgical substitutes to grafting?

Skin substitutes


-Bi layered dressing with epidermal and dermal analog


-Used on donor sites and wounds




Cultured epithelial autographs


-Cultures pt own cells


-Grown in lab and stapled sutured in place

Benefits of skin substitutes

Dec water loss


Maintain wet, warm environment


Provide physical barrier to infection


Thermal Insulation


Decrease pain


Promote tissue granulation


Less fq dressing changes


Dec contraction


Enhance healing