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64 Cards in this Set

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fragmentation of cell nucleus/chromatin
karyorrhexis
nuclear changes
coagulation necrosis
chromatin clumping

condensation/shrinking of cell nucleus
pyknosis
fading of chromatin

destruction/dissolution of nucleus
karyolysis
condensation of chromatin

shrinkage of nucleus
karyopyknosis
fragmentation of chromatin

fragmentation of nucleus
karyorrhexis
nuclear changes are morphologic hallmarks of
irreversible cell injury
necrosis
acute/early stage inflammation
neutrophils
PMN=
neutrophils
most numerous of WBC
neutrophils
chemotactic for neutrophils
C3a
C5a
anaphylatoxins
C3a
C5a
BV :
dilation
hypotension
increased vascular permeability
anaphylatoxins
process of attracting
recruiting cells
chemotaxis
cell moves toward a higher concentration of a chemical substance
chemotaxis
abnormal sac containing air/fluid
cyst
lined by epithelium
cyst
tumor a/w ulcerated infections
granuloma
modified macrophage
epitheliod cell
found in granulomas
epithelioid cell
surround the nodule of epithelioid cells
lymphocytes
plasma cells
fibroblasts
growth of tissue
tumor
abnormal regulation of cell division
tumor
necrosis with putrefaction
gas gangrene
putrefaction
rotting
toxin that damages cell membranes
alpha toxin
lecithinase
alpha toxin
hemolysis from
alpha toxin> damage RBC membrane>hemolysis
ischemia>
coagulation necrosis
cell death within a living body
necrosis
suppuration
abscess
ischemic injury to CNS
liquefaction necrosis
typical of TB

granulomatous inflammation
caseous necrosis
immune mediated vascular damage
fibrinoid necrosis
injured pancreas
trauma to adipose tissue
fat necrosis
brain/spinal cord
liquefaction necrosis
inflammation with
epithelioid macrophages
granulomatous
a/w granulomatous inflammation
infectious agents
sarcoidosis
crohn
mycobacterial
TB
leprosy
granulomas produced by
multinucleated giant cells
langerhans
multinucleated giant cells
granulomatous inflammation a/w
caseous necrosis
caseous necrosis produced by
mycobacterium TB
subtype of chronic inflammation
granulomatous inflammation
epitheliod cells
nodular collections of specialized macrophages
cells involved with acute inflammation
basophils
platelets
tissue mast cells
appear later in cellular phase
macrophage
transition btw

-acute
-chronic inflammation
macrophages
vascular response
cellular response
acute inflammation
increased vascular permeability

leukocytic cellular infiltration

repair
acute inflammation
systemic signs of acute inflammation
fever
tachycardia
leukocytosis (neutrophils)
exudate
straw
pus
principally water
exudate
increased intravascular hydrostatic pressure
transudate
non inflammatory conditions
transudate
acute inflammation rxn
neutrophils
tissue necrosis
proteolytic enzymes
WBC
suppurative
contains

few blood cells
low protein content
low specific gravity
transudate
granulocytes in neutrophils
acute cellular phase of acute inflammation
predominated cellular phase in acute inflammation
PMN
later in cellular phase
macrophage
parasitic rxn
allergic rxn>
eosinophils
vasoconstriction>
vasodilation>
increased vascular permeability
vascular phase of acute inflammation
first defense cell to migrate to injured tissue
by chemotaxis>
leukocytes
leukocytes engulf particulate matter by phagocytosis
cellular phase of acute inflammation