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12 Cards in this Set
- Front
- Back
what is the definition of pain?
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unpleasant sensory and emotional experience associated w/ actual or potential tissue damage, or described in terms of such damage
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define the following;
allodynia hypoalgesia hyperalgesia radiculopathy vasomotor changes sudomotor changes |
allodynia: pain in response to a non-painful stimulus
hypoalgesia: decreased response of a painful stimulus hyperalgesia: increased response to a painful stimulus radiuclopathy: functional abnormality of sensation, motor and/or autonomic activity of a spinal nerve root vasomotor changes: temperature of skin coloration abnormalities sudomotor changes: abnormal sweating |
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what is acute nociceptive pain?
what are the two subcategories? |
pain in response to tissue injury or inflammation
activation of peripheral nociceptors (free nerve endings of A-delta and C fibers) acute somatic and acute viseral |
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how are visceral structures represented on a homunculus?
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they aren't!!
referred pain!! |
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what is neuropathic pain?
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result of injury or acquired abnormalitites of peripheral or central neural structures
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what are the four elements of the "pain process"?
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1. transduction (converting painful stimulus to an action potential)
2. transmission (conduction along neural pathways) 3. perception (discrimination, cognition and affect higher centers) 4. modulation: 1-3 can be modulated at any point in the process |
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what kind of nerve fibers are associated with nociception? differentiate them
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a-delta: myelinated, faster "first pain"
c fibers: unmyelinated "second pain" |
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first order neurons for the head arise from which cranial nerves?
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V, VII, IX, X
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from what 3 sources do second order neurons receive input?
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a-delta
c fibers internuerons in dorsal horn |
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nosceceptive specific second order neurons are found in what lamina?
what about wide, dynamic range? |
lamina 1
lamina 5 |
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describe the site of action for the following analgesics:
opiods local anesthetics antihistamine NSAIDs |
opiods: dorsal horn
local anesthetics: nerve axon/sodium channels antihistamines: injured/inflammed tissues NSAIDs: prostaglandin synthesis |
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what is the Gate Theory?
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with no stimulation: pain gate is "closed" and there is no painful sensation
with painful stimulation: nociceptive afferents block inhibitatory neuron "open" |