Biol 2130 Test 2 Review

Front 1

Protein deficient diet in kids

Back 1

Kwashiorkor

Front 2

Main cause of Coronary Artery Disease (CAD)
Develop plaque within walls of medium to large arteries

Back 2

Atherosclerosis

Front 3

Process by which formed elements are produced

Back 3

hemopoiesis

Front 4

Undifferentiated stem cells are called:

Back 4

hemocytoblasts

Front 5

Hemocytoblasts are found in:

Back 5

Red marrow in certain bones

Front 6

Increases amount of blood in the heart (Exercise) Force of contraction is proportional to load

Back 6

Starling's Law

Front 7

Helps to regulate resting BP
Pressure Receptors in wall of aorta and carotid arteries

Back 7

Baroceptor Reflex

Front 8

Baroceptor Reflex:
If Blood pressure increases, Baroceptor Activity will:

Back 8

Increases the Cardioinhibitory Center (CIC) in the medulla decreasing the Blood Pressure back to normal

Front 9

Baroceptor Reflex:
If blood pressure decreases, Baroceptor Activity will:

Back 9

Decrease the CIC which will increased the blood pressure Back to normal

Front 10

adjust to breathing rate in response to pH, CO2, and O2

Back 10

Chemoceptors

Front 11

Chemoceptors:
Increase in [H+] will result in:

Back 11

Acidosis

Front 12

AV valve between RA and RV:

Back 12

Tricuspid Valve

Front 13

AV valve between LA and LV

Back 13

Bicuspid Valve

Front 14

Heart strings:

Back 14

Cordeae Tendoneae

Front 15

Cordeae Tendoneae attach from AV valve flaps to:

Back 15

Papillary Muscles

Front 16

Tissue Damaged Cells associted with blood vessels release:

Back 16

Prothrombin Activator

Front 17

Plasma protein made by liver cells with Vitamin K (inactive)

Back 17

Prothrombin

Front 18

Prothrombin Activator converts Prothrombin to:

Back 18

Thrombin

Front 19

Thrombin converts fibrinogen (made in liver) to:

Back 19

Fibrin

Front 20

Fibrin strands cross-link to form a:

Back 20

Mesh

Front 21

the Fibrin mesh trap:

Back 21

RBCs and Platelets to form a clot

Front 22

Hormone formed by kidneys to produce RBC's:

Back 22

EPO (erythropoietin)

Front 23

5 types of Anemia

Back 23

Heorrhagic
Aplastic
Hemolytic
Pernicious
Iron-Deficiency

Front 24

Anemia:
Excessive bleeding due to injury or surgery:

Back 24

Hemorrhagic Anemia

Front 25

Anemia:
Failure of stem cells in red marrow to function properly which decrease RBC count:

Back 25

Aplastic Anemia

Front 26

Anemia:
Increase rate of RBC destruction

Back 26

Hemolytic
(parasite)

Front 27

Anemia:
B12 deficiency:

Back 27

Pernicious Anemia

Front 28

Anemia:
low iron which lack hemaglobin reducing RBC

Back 28

Iron deficient Anemia

Front 29

Abnormal excess of RBC Production by red marrow:

Back 29

Polycythemia

Front 30

Anticoagulant that helps prevent abnormal clots in the blood

Back 30

Heparin

Front 31

Hole between 2 atria in fetal skeleton:

Back 31

Foramen ovale

Front 32

Name given to foramen ovale after it seals after birth:

Back 32

Fossa Ovalas

Front 33

Hole in the heart when the foramen ovale fails to close:

Back 33

Interatrial septal Defect

Front 34

Tube in Fetal skeleton connecting aorta with pulmonary trunk:

Back 34

Ductus Artiriosis

Front 35

The ductus arteriosis when seals off is koown as:

Back 35

Ligamentus artiriosum

Front 36

WBC when monocytes leave through cappilary walls the movement is known as:

Back 36

Emmigration

Front 37

Monocytes after emmegration is known as:

Back 37

macrophages

Front 38

"lubb" sound occrurs as:

Back 38

AV valves close

Front 39

"dubb" sound occurs as:

Back 39

SLV's close

Front 40

"pacemaker of the heart"

Back 40

SA node

Front 41

Generates the cardiac impulse causing the heart to beat:

Back 41

SA node

Front 42

An electrical "bridge" or coupling that connects the atria with the ventricles:

Back 42

AV Node

Front 43

delays the spread of the cardiac impulse

Back 43

AV Node

Front 44

Cardiac impulse crosses AV node and spreads rapidly through ventricles by way of AV Bundle and then by way of special myocardial cells:

Back 44

Purkinje fibers

Front 45

Parasympathetic effect are mediated by what neurotransmitter

Back 45

Ach

Front 46

Resting BP and digestive state:

Back 46

Parasympathetic Effects

Front 47

Effect turned on when your stressed
Increase:
HR
BP

Back 47

Sympathetic Effects

Front 48

Lipoprotein synthesized by liver cells with cholesterol and TG attached to them:

Back 48

VLDL
(Very Low Density Lipoproteins)

Front 49

When VLDL lose the TG they become:

Back 49

LDL
(Low density Lipoproteins)

Front 50

Carry cholesterol to the cells of the body and to the walls of arterioles to form plaque:

Back 50

LDL

Front 51

Protein that binds to cholesterol and carries it away from plaque

Back 51

HDL
(High density lipoprotein)

Front 52

How does HDL gets rid of cholesterol:

Back 52

Takes cholesterol to liver which puts it in bile then into ducts into the small intestine then into feces.

Front 53

How many O2 can a hemoglobin carry:

Back 53

4

Front 54

02 in hemoglobin binds to:

Back 54

Iron of the heme

Front 55

Fast resting heart rate >100 bpm

Back 55

Tachycardia

Front 56

Slow resting heart rate < 60 bpm

Back 56

Bradycarida

Front 57

Calculating BP

Back 57

Cardiac Output x Peripheral Resistance

Front 58

amount of blood pumped by the ventricles into the aorta or pulmonary trunk:

Back 58

Cardiac Output

Front 59

Cardiac Output is determined by:

Back 59

Stroke Volume and Heart Rate

Front 60

the volume of blood ejected by the ventricle per beat:

Back 60

Stroke Volume
70ml/beat

Front 61

Number of heart beats per minute:

Back 61

Heart Rate

Front 62

Cardiac Duty Cycle lasts:

Back 62

0.8 secionds

Front 63

Pumping of blood:

Back 63

Systole (0.3 sec)

Front 64

Relaxation and filling of blood

Back 64

Diastole (0.5 sec)

Front 65

Cardiac Duty Cycle consists of:

Back 65

A. Diastole = Fill
V. Systole = Contract
A. Systole = Contract
V.Diastole = Fill

Front 66

foreign chemicals in the body

Back 66

Antigen

Front 67

when exposed to antigens, b bells become:

Back 67

Plasma Cells

Front 68

Plasma cells secrete

Back 68

Antibodies

Front 69

Antibodies bind to antigens to form

Back 69

ag-ab complex

Front 70

ag-ab complex does what:

Back 70

tags foreign materials so they can be destroyed by WBCs.

Front 71

Cancer of the bone marrow

Back 71

Leukemia

Front 72

Abnormal blood clot:

Back 72

Thrombus

Front 73

Broken off blood clot that travels in blood stream:

Back 73

Embolis

Front 74

Anything that narrows arteries

Back 74

Corinary Artery Disease

Front 75

Corinary Artery Disease is caused by

Back 75

Athrosclerosis

Front 76

platlets secrete chemicals that causes clot to form on side of plaque

Back 76

Athrosclerosis

Front 77

WBC:
has odd shaped nucleus and phagocytizes bacteria

Back 77

Neutrophil

Front 78

WBC:
kills parasitic worms

Back 78

Eosinophils

Front 79

WBC:
releases heparin and histomine in response to tissue damage:

Back 79

Basophils

Front 80

this is a vasodiolater, increases the diameter of blood vessles, and increases capillary permeability:

Back 80

histomine

Front 81

WBC:
live in lymph nodes contains B and T cells:

Back 81

Lymphocytes

Front 82

b cells transform into what in response to antigens:

Back 82

plasma cells

Front 83

plasma cells secrete:

Back 83

antibodies

Front 84

WBC:
these turn into macrophages once they leave capillaries:

Back 84

Monocytes

Front 85

Another name for heart attack:

Back 85

Myocardial Infraction

Front 86

Occurs when a group of cardiac muscle cells die:

Back 86

Heart Attack

Front 87

What causes a myocardial infraction:

Back 87

blood clot that forms on atherosclertoic plaque within corinary arteries
the embolis blocks blood vessels

Front 88

What is a small heart attack:

Back 88

"silent"

Front 89

what is a large heart attack:

Back 89

Ventricular Fibrillation

Front 90

resistance of blood vessels to free flow of blood

Back 90

Peripheral Resistance

Front 91

2 changes in diameter of blood vessels

Back 91

Vasoconstrition
Vasodialation

Front 92

Tissue swelling:

Back 92

edemia

Front 93

Right subclavian vein of Lymphatic System:

Back 93

R. lyphatic duct

Front 94

Left subclavian vein of Lymphatic System:

Back 94

Thoracic Duct

Front 95

Lymph flows up to become:

Back 95

Blood plasma

Front 96

WBCs move by:

Back 96

pseudopodial locomotion

Front 97

Vit K is necessary to make:

Back 97

Prothrombin in liver

Front 98

Abnormal shaped valve in heart:

Back 98

Prolapse

Front 99

Narrow shaped valve in heart:

Back 99

Stenosis

Front 100

Angiotensinogen is a

Back 100

Inactive plasma protein made by liver cells:

Front 101

When BP drops, the kidneys release:

Back 101

Renin

Front 102

Renin converts Angiotensinogen to:

Back 102

ANG1

Front 103

ACE created by the lungs convert ANG1 to

Back 103

ANG2

Front 104

ANG 2 is a potent

Back 104

Vasoconstrictor

Front 105

jaundice that blocks bile secretion by obstructing ducts that carry bile from liver to small intestine:

Back 105

Obstructive

Front 106

Jaundice which has an abnormal rate of RBC destruction which leads to increase of amount of bilirubin carried by the blood

Back 106

Hemolytic