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25 Cards in this Set
- Front
- Back
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INFLAMMATION WOUND REPAIR DOMINATED BY WHAT CELLS?
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NO AND MO
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PROLIFERATION WOUND REPAIR DOMINATED BY WHICH CELLS?
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ENDOTHELIAL CELLS AND FIBROBLASTS
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MATURATION/REMODELING WOUND REPAIR GOVERNED BY?
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COLLAGEN DEPOSITION
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CLOT FORMATION AT SITE OF INJURY WHICH CELL FIRST
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NO
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MO PRODUCE?
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NO: KILLS BACTERIA
MMP: ELIMIANTE CELLAR DEBRIS,PHAGOCYTOSSI OF BAC, CELL DEBRIS, AND NO |
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PRIMARY STIMULIS FOR PROLIFERATIVE PHASE WOUND REPAIR
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ENDOTHELIAL GROWTH FACTOR AND TGF-ALPHA (PRODUCED BY PLATELETS AND MO)
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PLATELETS AND MO PRODUCE WHAT THAT IS THE MAIN STIMULUS FOR PROLIFERATIVE PHASE WOUND REPAIR?
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ENDOTHELIAL GROWTH FACTORS AND TGF-ALPHA
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FIBROBLASTS PROLIFERATIVE WOUND REPAIR SECRETE WHAT?
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KO GROWTH FACTOR 1 AND 2
IL-6 STIMULATE KO TO MIGRATE, PROLIFERATE AND DIFFERENTIATE IN EPIDERMIS |
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PROVISIONAL MATRIX FORMED IN WOUND REPAIR MADE OF WHAT?
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COLLAGEN 3
GAGS FIBRONECTIN |
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TGF-B WOUND REPAIR
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DECREASES MMP
INCREASE COLLAGEN 1 INCREASES CELL ADHESION PROTEINS |
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WHAT TURNS OFF PROLIFERATIVE PHASE WOUND REPAIR?
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INTERFERON INDUCIBLE PROTEIN 10 IP-10
INHIBITS EGF-INDUCED FBIBROBLAST MOTILITY, LIMITS FIBROBLAST RECRUITMENT, NEGATIVE MITOGENIC EFFECT ON FIBROBLASTS |
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CONTRACTURE OF WOUND DURING WOUND REPAIR
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FIBROBLASTS DIFFERENTIATE INTO MYOFIBROBLASTS
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HYDROCOLLOIDS
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CARBOXYMETHYCELLULOSE, GELATIN, PECTIN, ELASTOMERS, ADHESIVES
OCCLUSIVE DEBRIDEMENT OF NECTROTIC TISSUE |
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HYDROGELS
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MOISTURE
DEBRIDEMENT |
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ALGINATES
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CALCIUM ALGINATE
DEBRIDEMENT S0DIUM ABSORBED FROM WOUND TURNS PAD INTO GEL |
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OVEREXPRESSION OF ? CORNOID LAMELLA
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P53
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POROKERATOSIS
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FIRST LINE
CYRO SECOND LINE 5FU ALDARA VITAMIN D3 ANALOGS |
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PRUIGO PIGMENTOSA
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MINOCYCLINE
DAPSONE |
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CHOLESTASIS
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RIFAMPIN
NATREXONE, NALMEFENE, NALAXONE |
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CHOLESTASIS OF PREG
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UROSODEOXYCHOLIC ACID
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UREMIC ITCH
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UVB
GABAPENTINNALTREXONE |
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MAL ASSOC PRURITUS
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PAROXETINE
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ITCH ASSOC W HEM
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UVB
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PXE RETINAL
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BEVACIZUMAB
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RELAPSIN POLYCHONDRITIS
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SYSTEMIC STEROIDS
NSAIDS DAPSONE |
