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76 Cards in this Set
- Front
- Back
How is P2 loaded?
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Axial and torsional planes
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Who is predisposed to P2 fractures?
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Horses turning on hindlimbs such as western performance horses or jumpers
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Categories of P2 fractures:
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dorsal/ palmar-plantar IA OC chip, palmar-plantar eminence, axial, comminuted
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Where do P2 OC fractures occur?
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Most often palmar-plantar, immediately medial or lateral to midline
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Treatment of P2 OC fractures:
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arthroscopic removal if determined to cause lameness
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Approaches to surgical removal of P2 OC fractures:
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dorsal or palmar-plantar arthroscopy preferred, open approach through DFTS for palmar-plantar or arthrotomy dorsally
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Mechanism of palmar-plantar P2 eminence fractures:
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hyperextension of pastern joint with tension on palmar-plantar attachments of SDFT, middle scutum, and distal sesamoidean ligaments
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How are uniaxial P2 eminence fractures treated?
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Lag screw fixation or pastern arthrodesis
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How are biaxial P2 eminence fractures treated?
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Pastern arthrodesis (with interfragmentary compression through plate or in addition to plate)
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Treatment of axial P2 fracture:
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lag screw fixation + casting for 4 weeks
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Treatment of comminuted P2 fracture:
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lag screw fixation for minimally comminuted fractures, single LCP or DCP with interfragmentary screws, 2 LCP or DCP for pastern arthrodesis, lag screw fixation + TPC
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Which P2 fracture fixation option optimizes chances for adequate reconstruction of distal P2 articular surface?
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Plate fixation with independent lag screw fixation of fracture
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When is TPC used for P2 fractures?
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Fractures than can not be reduced and stabilized with implants and euthanasia is not an option
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What decreases the prognosis for P2 fracture fixation repair?
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Fragments entering DIP joint, compromise to vasculature and soft tissue structures, concurrent fracture of NB
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Treatment of PIP OA:
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IA corticosteroid injection, alcohol facilitated ankylosis, pastern arthrodesis
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Arthrodesis options for PIP OA:
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2-3 5.5 transarticular screws, LCP or DCP with or without adjacent transarticular screws, minimally invasive placement of 1 or 2 LCP with or without transarticular screws, laser ablastion with 2 5.5 transarticular screws
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What types of OC lesions affect PIP joint?
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SCL more often than OCD, usually distal articular surface of P1 (more than proximal articular surface of P2)
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Treatment of PIP SCL:
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solitary SCL treated with transosseous drilling, curettage of SCL, and grafting with tricalcium phosphate granules or hydrogel with parathormone; multiple SCL treated with pastern arthrodesis
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Treatment of PIP luxation or subluxation:
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for dorsal subluxation due to upright conformation, NSAIDs and controlled exercise; for dorsal subluxation due to tension on DDFT, release of medial head of DDFT; for others, pastern arthrodesis
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What causes dorsal PIP subluxation?
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Damage to SL and SL extensor branches or occasionally contracture of distal sesamoidean ligaments or excessive tension on DDFT
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What causes palmar-plantar PIP subluxation?
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Failure of distal sesamoidean ligament, middle scutum, SDFT insertions on P2 due to hyperextension
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Categories of P1 fractures:
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proximal IA OC fractures (dorsal and palmar-plantar), fractures of the shaft or diaphysis
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Cause of dorsal proximal IA P1 OC fractures:
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hyperextension of the MC/MTP joint with impact of proximal P1 on the dorsal aspect of distal MC/MT3
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Most common location of dorsal proximal IA P1 OC fractures:
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dorsomedial eminence of P1
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Treatment of dorsoproximal IA P1 OC fracture:
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small fragments can be left if not causing lameness, larger fragments are removed with dorsal fetlock arthroscopic approach
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Post-op care dorsoproximal IA P1 OC fractures:
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rest 6-12 weeks depending on damage to MC3 cartilage
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Types of palmar-plantar proximal IA P1 OC fractures:
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1: avulsion from axial, proximal, plantar-palmar rim of P1, mostly articular, extensive attachment of short sesamodian ligament; 2: large, abaxial, partially articular fractures extending 2-3 cm distally with minimal articular cartilage
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Radiographic projection for palmar-plantar type 1 P1 OC fracture:
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proximo-20-dorso-15-lateral to distal palmaromedial or proximo-20-dorso-15-medial to distal palmarolateral
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Treatment of type 1 P1 OC fractures:
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arthroscopic removal
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Treatment of type 2 P1 OC fractures:
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removal only if causing lameness, even if radiographic fusion is not evident
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Post-op care for palmar-plantar type 1 P1 OC fractures:
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6-12 weeks rest depending on degree of short sesamoidean ligament injury
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Characteristics of dorsal frontal P1 fractures:
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short, extending 2-5 cm distal in the dorsolateral cortex, complete, minimally displaced
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Treatment of dorsal frontal P1 fractures:
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complete: lag screw fixation using 1 or 2 3.5 cortex screws with arthroscopic reduction of articular surface; incomplete: stall rest or lag screw fixation
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Most common characteristics of P1 diaphyseal fractures:
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sagittal, distal from articular surface from mid-sagittal groove
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Mechanism of P1 diaphyseal fractures:
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torsion with axial weight bearing applied to sagittal groove of P1 from sagittal ridge of MC/MT3
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Treatment of short P1 sagittal fractures:
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if incomplete, conservative therapy or lag screw fixation
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Prerequisite of P1 fracture repair:
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intact strut of bone spanning from MC/MTP joint to PIP joint
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Treatment of complete, non-displaced P1 diaphyseal fractures:
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lag screw fixation with a proximal 5.5 cortex screw placed within 5mm of the distal point of the sagittal groove, and the rest of the fracture fixed with 4.5 cortex screws, post-operative cast (hind limb) or bandage/ splint (fore limb)
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Treatment of mild to moderate displaced P1 diaphyseal fractures:
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open, dorsal with I or S shaped incision for articular alignment and interfragmentary screw placement
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Benefits of I shaped dorsal approach to P1 diaphyseal fractures:
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exposes entire dorsal surface, preservation of collateral ligaments
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Disadvantages of S shaped dorsal approach to P1 diaphyseal fractures:
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transection of collateral ligament of fetlock joint
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Treatment of severely comminuted diaphyseal P1 fractures:
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salvage TPC or external skeletal fixation device
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Disadvantages of cast only fixation of diaphyseal P1 fractures:
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cast does not provide resistance to axial collapse of the fracture
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What is the cause of palmar MC fragmentation?
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Accumulation of stress and sclerosis of the palmar MC condyles because of hyperextension during racing
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Radiographic projection for palmar MC fragmentation?
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125-dorsopalmar
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Radiographic appearance of palmar MC fragmentation:
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differently shaped defects with intense sclerosis of the palmar condyle, secondary OA
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Lesion associated with palmar MC fragmentation:
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acellular necrotic bone over the entire distal palmar MC condyle, with a sclerotic zone deep to the necrotic bone
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Treatment of palmar MC fragmentation:
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none (conservative) because develop in region not accessible arthroscopically
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Types of PSB fractures:
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apical, mid-body, basal, abaxial, sagittal, comminuted
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What predicts prognosis with apical PSB fractures?
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Extent of loss of SL insertion and preexisting SL desmitis
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What percent of the PSB is involved with articular apical fractures?
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PSB fractures involving ¼ - 1/3 of the bone are always articular
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Treatment for apical PSB fractures:
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for fragments up to 1/3of the PSB, palmar arthroscopic removal, large fragments need internal fixation
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Treatment of mid-body PSB fractures:
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internal fixation with cerclage wire (STB) or lag screws (TB)
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How is lag screw fixation of mid-body PSB fractures performed?
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Oblique fracture declining from medial to lateral has screw placed proximolaterally from the apex embedded in the insertion of the SL branch; oblique fractures declining from lateral to medial has screw placed from base in a fossa between the oblique and straight distal sesamoidean ligament or from a contralateral approach for proximal to distal screw insertion
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What affects the prognosis for basal PSB fractures?
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Involvement of distal sesamoidean ligament and thin fragment profile
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Treatment of basal PSB fractures:
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if less than 25% of base, remove fragment, if more than 25% consider internal fixation
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Radiographic projection for abaxial PSB fracture:
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60 degree skyline of abaxial PSB surface to determine if IA or not
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Treatment of abaxial PSB fracture:
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if IA, remove by arthroscopy, if not IA, conservative management
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Where and when do sagittal PSB fractures occur?
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Axial margin of PSB in conjuction with other MCP joint injuries such as condyle fractures
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Treatment of sagittal PSB fractures:
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if no other injuries, lag screw fixation from lateral to medial with 3.5mm cortex screws
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What is chronic proliferative or villnodular synovitis?
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Development of soft tissue mass in dorsal MCP joint secondary to chronic fibrosing synovitis or advancing OA
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Radiographic features of chronic proliferative synovitis:
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cortical lysis on dorsal distal MC3 with enlarged ST mass
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Best diagnostic imaging for diagnosis of chronic proliferative synovitis:
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ultrasound
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What is critical for surgical decision regarding chronic proliferative synovitis?
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Extent of OA
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Treatment of palmar-plantar MC/MT3 OCD:
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none, unless accessible via arthroscopy, which is rare
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Treatment of dorsal sagittal ridge or parasagittal MC/MT3 OCD:
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arthroscopic removal
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Treatment of distal MC/MT3 SCL:
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if accessible by arthroscopy and dorsal to the transverse ridge of MC/MT3, debridement through arthroscopic approach, if not accessible arthroscopically, does not communicate with joint, or located palmar-plantarly, transosseous approach with debridement and graft therapy
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Cause of fetlock luxation:
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rupture of either medial or lateral collateral ligament, usually from entrapment of the distal limb in a hole
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Treatment of closed fetlock luxation:
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cast immobilization for 6 weeks
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Treatment of open fetlock luxation:
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joint and wound lavage and debridement, support in splint until infection cleared, then support in cast or arthrodesis
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Disadvantages of open approach to DFTS:
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delay in initiation of exercise post-operatively which increases risk of adhesion formation, increase wound dehiscence if exercise is initiated too early
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Options for annular ligament desmotomy:
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open approach with incision over entire length of ligament, closed approach with paramedian incision over entire length of ligament, 1 cm proximal incision with blind transection with curved bistoury, 2 cm proximal incision, tenoscopy transection with 90 degree angled blade, bistoury, or RF probe
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Post-op care for DFTS tenoscopy:
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bandaging for 3-4 weeks, walking after 5 days, intrathecal HA
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Most common type of ruminant MC/MT fracture:
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distal metaphysis from forced fetal extraction, SH type 1 or 2 in non-neonates, mid-diaphyseal in adults
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Fixation of neonatal ruminant distal metaphyseal MC/MT fractures:
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external coaptation or external skeletal fixation
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Internal fixation of ruminant MC/MT fractures:
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external fixation in smaller animals, internal fixation with double plating (1 dorsal) for larger animals
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