• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/141

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

141 Cards in this Set

  • Front
  • Back
Immittance
physiological test of M.E.'s ability to pass energy thru
Function Tests
-complement to behavioral tests
-doesn't test exact hearing sensitivity
admittance
how easily energy flows thru M.E. system (infers ET funct)
impedance
resistance to energy flow; how a system opposes energy flow thru it
when is admittance max?
P of EAM = P of M.E.
age of tympanometry
reliable after 3 mo age
hermetic seal
no air leaks from probe tip to TM
minimum admittance
+200 daPa added to EAM
what does tympanometry measure?
-vol b/w probe tip & TM
-impedance of M.E.
what can you infer w/tympanometry?
-mass & stiffness
-funct of M.E. & ossicles
-M.E. vol
When is equiv. vol of EAM-TM large?
TM perforation or PE tubes
when is equiv. vol small?
OM w/effusion or cerumen impaction
what causes increased mass in the M.E.?
fluid or disarticulated ossicles
what causes stiffness in M.E.?
otosclerosis
Nml Equiv. vol EAM to TM
infant = 0.3 to 0.9
child = 0.6 to 1.5
adult =0.6 to 2.0
referral schedule for tympanometry
>0.3 vol in under 6 yrs
>0.4 vol 6 yr +
6-8 wk
where does energy flow best?
P of EAM = P of M.E. (max admittance)
normal range for peak admittance
+50 to -100 (0 = atmos P is perfect)
what can you infer when peak admittance = 0?
normal ET funct (balanced mass & stiffness of M.E.)
normal values of peak compensated static acoustic admittance
infant = 0.25 to 0.92
child = 0.25 to 1.5
adult = 0.3 to 1.7
tympanometry units
-determined by equip.
-mmho (actual energy)
-cm^3 = cc
-mL (vol)
Aside from immitance device & probe tip, what equip. req for tymp.?
226Hz probe tone loudspeaker, monitor mic, P pump & manometer, ipsilateral reflex loudspeaker
tympanogram shows (on pic)
admittance across TM
type A(s) tymp
ossicular chain not vibrating fully-->reduced energy thru M.E.
what tymp implies a nml conductive system?
type A
otosclerosis results in which tymp?
type A(s)
is ET funct in type A(s) tymp?
yes (but ossicles stiff)
Tymps that convey pathologies where absent acoustic reflex
Types A(s), A(d), & B.
type A(d) tymp
moderate conductive HL
causes of disarticulated ossicular chain
-severe infection
-TBI
flaccid TM that vibrates w/large amplitude
type A(d) tymp
type B tymp
always check vol
increased mass that cause ossicles to not vibrate freely; ET not working
type B tymp
type C tymp
P EAM > P M.E.
type of tymp when ET not working well
type C tymp
risks w/type C tymp
cholestotoma (neg P pulls TM toward M.E. space)
what admittance is ART tested?
max admittance (P EAM = P M.E.) where energy flows best
ART frequency & intensity stim range
500, 1KHz, 2KHz, 4KHz
70-110 dB SPL
ART threshold search
if admittance drops, decrease by 10 dB SPL
what dB level are we looking for in ART?
doesn't matter--just presence/absence of reflex
definition of acoustic reflex threshold (ART)
min intensity to elicit change of min 0.2 mL in admittance
what does change in admittance show in ART?
increased impedance (ossicular chain stiffens w/stapedius contraction)
in what test will cochlea respond even w/40 dB HTL?
acoustic reflex present
no acoustic reflex in SNHL usually caused by
nerve tumor
ART w/type A(s) tymp
no reflex b/c ossicular chain already stiff (fixed stapes)
T or F: cerumen impaction will cause absent acoustic reflex
T. r/o w/otoscopy
T or F: ART tests hearing sensitivity
F. (no response at nml/mild threshold levels)
physiological test that helps dx exact site of lesion
ART
contralateral reflex pathway
. . .superior olivary complex---->facial nucleus-->CNVII-->stapedius muscle
efferent in acoustic reflex pathway
CNVII (carries motor info from brain to body)
afferent in reflex pathway
CNVIII (carries sensory info to brain)
ipsilateral reflex pathway
peripheral-->cochlear nucleus-->superior olivary complex-->facial nucleus-->CNVII-->stapedius muscle
causes a higher acoustic reflex threshold
worse conductive HL (absorbs stim)
process to find acoustic reflex decay
find ART, increase by 10 dB SPL, present stim 10 sec
abnormal acoustic decay
50% decrease in response before 10 sec is up
use for acoustic decay?
help find retrocochlear HL (b/c neural fatigue faster than norm)
(screening) test that indirectly assess the M.E.
otoacoustic emissions (OAE)
OHC
-efferent
-lateral
-resp to soft sounds
-3 rows
cochlear cells that are motile (dance)
OHC
what is measured in OAE?
the unique sound created by P waves from vibrating OHC
IHC
-afferent
-respond to loud sounds
-no stereocilia & not embedded in tectorial membrane
probe tip in OAE
helps block noise & trap emission
info derived about the nerve in OAE
none--only tests to the cochlea (can even get an emission if sound not traveling to brain)
OAE test considerations
-test in quiet enviro
-no muscle mvmt
OAE frequency
-high freq = larger resp
-low freq blend w/random noise
criteria for present emission
+6 dB S/N over random noise
750-8kHz
frequencies tested in DPOAE
first test for differential dx
OAE (in SNHL b/c test only goes to cochlea, not nerve beyond)
SNHL with present emission
nerve is responsible for HL (inferred)
SNHL with absent emission
cochlea is responsible for HL
no emission found in OAE test
-otoscopy to r/o outer ear
-tympanometry to r/o M.E.
types of OAE
-spontaneous = 50% ppl get OHC dance sans stim
-evoked = clin. useful
definition of evoked OAE
OHC dance in response to stim
types evoked OAE
-transient evoked (TEOAE)
-distortion product (DPOAE)
what is TEOAE?
broadband clicks that dx only flat HL
what is DPOAE?
2 stim freq generate a 3rd measurable prod = (2F1-F2)
which evoked OAE is freq specific?
DPOAE (b/c you can manipulate the 2 stim freq to get a prod on a certain region of the BM)
which evoked OAE is binary?
TEOAE (b/c it is multi-freq)
DPOAE can measure what frequencies?
20-20,000HZ, but high freq have better resp
what does a large emission mean?
Great dif. b/w sig. & noise means nothing. We care if it's present or not.
T or F: evoked OAEs are threshold tests
F (NOT a threshold test, we aren't manipulating intensities, doesn't ascertain hearing sensitivity)
dB HTL that an emission will be present
0 dB HTL to 30 dB HTL
assumption about hearing level with present emission
no more then mild HL (no emission in moderate-profound HL)
M.E. assumptions w/present emission
no pathology, no conductive component
assumption about inner ear with present emission
intact cochlea capable of resp to SOFT sounds
OAE results in moderate to profound HL
no emission (but we still don't know exact degree HL)
steps after getting absent emission?
use other dx tests to find site of lesion (OAE is a complement test)
OAE referral schedule
rescreen OAE-->refer = ABR (by 3 mo in newborn population)
clinical uses for evoked OAE
-screen (special populations)
-monitor ototoxicity
-dx pseudohypoacusis
-differential dx (AC = BC)
possible reasons for absent emission
-too much enviro noise or mvmt
-birthing fluid in ears
-operator error
-HL
Who sets OAE pass/refer criteria?
equip/manufacturer (non-standard; no calibration)
absent emission, but nml audiogram
OAE will show cochlear changes before they appear on audiogram (good monitoring tool)
umbrella term for all tests that look at time b/w aud stim & change in neural activity (resp)
auditory evoked potentials (AEP)
definition of latency
time b/w stim & resp (in msec)
AEP latency intervals
-early = 0-15 msec
-middle = 16-60 msec
-late = 50-200 msec
other acronyms for auditory brainstem response
BAER, AER, BAEP, BSER, early resp
purpose of ABR (what affects CNVIII?)
-screening
-thresholds
-nerve tumor
-dyssynchrony
-M.S.
ABR testing considerations
-quiet (sleeping preferred)
-no mvmt
another test for differential dx
ABR (in SNHL b/c looks @ nerve & beyond)
Gives site of lesion in SNHL when used together?
OAE = cochlear & ABR = neural
test to dx M.S.?
ABR (slow resp, especially w/fast rate in demyelinated)
age of ABR resp
-34-35 wk gestation
-2 yr for mature resp
age conditions req for ABR to work
-pneumotized M.E.
-synchronization (of CNVIII)
process of pneumotization
-1st gasp stim ET
-replaces liquid in M.E. w/air
characteristics of an adult ABR resp
-absolute latency
-interpeak latency
-all neural generators resp (wv I-VII)
far-field measurements
-electrodes on scalp (vs field = needle direct on nerve)
electrode montage
-forehead/vernix; reference EEG on pinna/mastoid, ground
electrode that p/u actual aud resp
forehead
electrode that p/u body EEG
reference
types of ABR
-transient clicks
-tone pips
ABR used for hearing screening
transient clicks (broadband = range 1-4KHz; all high freq)
ABR used to collect thresholds
tone pips (attempt at freq-specific)
intensity units for tone pip ABR
dB nHL (compared to normed data)
Neurons resp fast to transient click ABR b/c?
sharp = fast rise & fall time; no complex info to process
far-field req how many sweeps for morphology?
min of 10,000 (more = better pic, takes more time)
ABR clicks/sec
11 (faster = fatigues neural generators)
how to use ABR rate to dx pathology
use neural fatigue (slow = resp & faster = falls apart) to dx retrocochlear
in ABR collect own norms for
-age
-gender
-latency-intensity function
definition of absolute latency
wave peak over time
definition of interpeak latency
distance b/w waves (I-V = 4 msec)
ABR interpretation looks at:
-morphology
-latency: absolute/interpeak/interaural
ABR wave I corresponds to
cochlear nucleus (CNVIII)
ABR wave III corresponds to
superior olivary complex (according to B.P.)
ABR wave V corresponds to
inferior colliculus (all synapse here)
ABR in conductive HL
all waves = delayed latency; nml interpeak latency
ABR in SNHL
wv I morphology poor; short interpeak (I delayed, V nml)
ABR in retrocochlear HL
all waves = poor morphology; long interpeak (I nml, V delayed)
interaural latency max difference
0.2 msec or less
interaural latency is slower in 1 ear
suspect nerve tumor (unilateral)
age for conditioned play audm
2 yr and up
age for VRA
(maybe 4 mo) 5mo to 2yr
age for BOA
birth to 4-5-ish mo
pediatric behavioral tests
-BOA
-VRA
-conditioned play audm
-speech audm
behavioral observation audm (BOA) responses
-blink, widen, engage w/eyes
-change sucking pattern
-startle (always 65 dB)
test w/very rapid habituation?
BOA (subtle & limited resp)
VRA procedure
-condition (noise)
-resp (head turn)
-reward (lighted toy)
= operant conditioning
approx dB level VRA will test
20 dB (depends on age & stim)
test that utilizes behavioral conditioning & shaping
conditioned play audm (train + praise)
-block in box
-pegs in board
exp of conditioned play audm activities
speech audm used for
AC & BC thresholds (not freq-specific)
provides freq-specific info
-filtered speech
-narrow-band noise
-DPOAE
-pure tone tests