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48 Cards in this Set
- Front
- Back
antidysrhythmic meds CLASS I
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sodium channel blockers stabilize cardiac membranes
Divided into 3 groups, IA, IB, IC |
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antidysrhytmics Class IA
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procainamide (Pronestyl, Procanbid) oral, I
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quinidine gluconate (Quinaglute Dura-Tabs)
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antidysrthymics Class IA
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quinidine sulfate (Quinidex Extentabs)
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antidysrhythmics Class IA
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disopyramide (Norpace)
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antidysrhythmics Class IA
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antidysrhythmics Class IB
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prototype: lidocaine (Xylocaine) IV
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mexiletine (Mextil)
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antidysrhythmics Class IB
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Tocainide (Tonocard)
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antidysrhythmics Class IB
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antidysrythmics CLass IC
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Prototype: propadfenone (Rythmol) oral
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felcainide (Tambocor)
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antidysrhythmic Class IC
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Class II antidysrythmic
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beta-adrenergic blockers prevent sympathetic nervous system stimulation of the heart.
prototype: propranolol hydrochloride (Inderal) |
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esmolol hydrochloride (Breviblock)
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Class II antidysrhythmic
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acebutolol hydrochloride (Sectral)
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Class II antidysrhythmic
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Class III antidysrhythmic meds
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potassium channel blockers prolong the action potential and refractory period of the cardiac cycle
prototype: amiodarone (Cordarone, Pacerone)- oral, IV |
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Soltalol (Betapace)
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Class III antidysrhythmic
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Ibutilide (Corvert)
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CLass III antidysrhythmic
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Doefetilide (Tikosyn)
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Class III antidysrhythmic
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Class IV antidysrhythmic
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calcium channel blockers depress depolarization and decrease oxygen demand of the heart
Proto: verapamil (Calan)- oral, IV |
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diltiazern (Cardizem)
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Class IV antidysrhythmic
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other antidysrhythmic meds
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adenosine (Adenocard) IV
digoxin (Lanoxin) oral, IV |
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Class IA MOA
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decrease electrical conduction
decrease automaticity decrease rate of repolarization |
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Class IA use
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supraventricular tachycardia
ventricular tachcardia atrial flutter atrial fibrillation |
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Class IB MOA
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decreased electrical conduction
decrease automaticity increase rate of repolarization |
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Class IB use
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short term use for ventricular dysrhythmias
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Class IC MOA
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decreace electrical conduction
decrease excitability increase rate of polarization |
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Class IC use
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SVT
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Class II MOA
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decrease heart rate
slow rate of conduction through SA node decrease atrial ectopic stimulation |
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Class II use
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A-fib, atrial flutter, paroxysmal SVT, hypertension, angina
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Class III MOA
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decrease rate of repolarization
decrease electrical conduction decrease contractility decrease automaticity |
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Class III use
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coversion of a-fib- oral route
recurrent ventricular fibrillation recurrent ventricular tachycardia |
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Class IV calcium channel blockers
veramapil, diltiazem MOA |
decrease force of contraction
decrease heart rate slow rate of conduction through the SA and AV nodes |
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Class IV calcium channel blockers
veramapil, diltiazem USEs |
atrial fibrillation and flutter
SVT hypertension angina pectoris |
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adenosine MOA
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decrease electrical conduction through AV node
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adenosine use
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paroxysmal SVT
Wolf-Parkinson white syndrome |
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digoxin MOA
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decrease electrical conduction through AV node
Increase myocardial contraction |
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digoxin use
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HF, a-fib, and flutter
paroxysmal SVT |
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Procainamide adverse
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systemic lupus syndrome
neutropenia and thrombocytopenia cardiotoxicity Monitor procainamide level : 4-8 mcg/mL, BP, vitals, ECG, blood counts |
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lidocaine adverse
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CNS effects, respiratory arrest
administer phenytoin to control seizure activity |
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propafenone adverse
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bradycardia, heart failure, dizziness, weakness
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propranolol adverse
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hypotension, bradycardia, heart failure, fatigue
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amiodarone adverse
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pulmonary toxicity, sinus bradycardia and AV block, which may lead to heart failure
visual disturbances liver and thyroid dysfunction, GI disturbances CNS effects phlebitis with IV admin hypotension, bradycardia, AV block |
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verpamil adverse
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bradycardia, hypotension, heart failure, constipation
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adenosine adverse
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sinus bradycardia, flushing of the face
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digoxin adverse
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bradycardia, hypotension, toxicity, nausea, vomiting, dysrhythmias- hold dose if heart rate less than 60/min
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antidysrhythmic adverse
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toxicity is a major concern for antidysrhythmics
can lead to increased cardiac dysrhythmias |
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antidysrhythmics contraindications
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heart block, bradycardia, liver, kidney, respiratory problems
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antidysrhythmics interventions/client education
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Monitor ECG and blood pressure
monitor for toxicity monitor digoxin level |
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antidysrhythmics interactions
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concurrent use of atenolol, esmolol, or propanolol may cause additive effects of both meds
beta blockers may cause heart failure methylxanthines dipyridamole corticosteroids diuretics thiazides amphotericin B (decreased potassium levels) antacids and metoclopramide |