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48 Cards in this Set

  • Front
  • Back
antidysrhythmic meds CLASS I
sodium channel blockers stabilize cardiac membranes
Divided into 3 groups, IA, IB, IC
antidysrhytmics Class IA
procainamide (Pronestyl, Procanbid) oral, I
quinidine gluconate (Quinaglute Dura-Tabs)
antidysrthymics Class IA
quinidine sulfate (Quinidex Extentabs)
antidysrhythmics Class IA
disopyramide (Norpace)
antidysrhythmics Class IA
antidysrhythmics Class IB
prototype: lidocaine (Xylocaine) IV
mexiletine (Mextil)
antidysrhythmics Class IB
Tocainide (Tonocard)
antidysrhythmics Class IB
antidysrythmics CLass IC
Prototype: propadfenone (Rythmol) oral
felcainide (Tambocor)
antidysrhythmic Class IC
Class II antidysrythmic
beta-adrenergic blockers prevent sympathetic nervous system stimulation of the heart.

prototype: propranolol hydrochloride (Inderal)
esmolol hydrochloride (Breviblock)
Class II antidysrhythmic
acebutolol hydrochloride (Sectral)
Class II antidysrhythmic
Class III antidysrhythmic meds
potassium channel blockers prolong the action potential and refractory period of the cardiac cycle

prototype: amiodarone (Cordarone, Pacerone)- oral, IV
Soltalol (Betapace)
Class III antidysrhythmic
Ibutilide (Corvert)
CLass III antidysrhythmic
Doefetilide (Tikosyn)
Class III antidysrhythmic
Class IV antidysrhythmic
calcium channel blockers depress depolarization and decrease oxygen demand of the heart

Proto: verapamil (Calan)- oral, IV
diltiazern (Cardizem)
Class IV antidysrhythmic
other antidysrhythmic meds
adenosine (Adenocard) IV
digoxin (Lanoxin) oral, IV
Class IA MOA
decrease electrical conduction
decrease automaticity
decrease rate of repolarization
Class IA use
supraventricular tachycardia
ventricular tachcardia
atrial flutter
atrial fibrillation
Class IB MOA
decreased electrical conduction
decrease automaticity
increase rate of repolarization
Class IB use
short term use for ventricular dysrhythmias
Class IC MOA
decreace electrical conduction
decrease excitability
increase rate of polarization
Class IC use
SVT
Class II MOA
decrease heart rate
slow rate of conduction through SA node
decrease atrial ectopic stimulation
Class II use
A-fib, atrial flutter, paroxysmal SVT, hypertension, angina
Class III MOA
decrease rate of repolarization
decrease electrical conduction
decrease contractility
decrease automaticity
Class III use
coversion of a-fib- oral route
recurrent ventricular fibrillation
recurrent ventricular tachycardia
Class IV calcium channel blockers
veramapil, diltiazem MOA
decrease force of contraction
decrease heart rate
slow rate of conduction through the SA and AV nodes
Class IV calcium channel blockers
veramapil, diltiazem USEs
atrial fibrillation and flutter
SVT
hypertension
angina pectoris
adenosine MOA
decrease electrical conduction through AV node
adenosine use
paroxysmal SVT
Wolf-Parkinson white syndrome
digoxin MOA
decrease electrical conduction through AV node
Increase myocardial contraction
digoxin use
HF, a-fib, and flutter
paroxysmal SVT
Procainamide adverse
systemic lupus syndrome
neutropenia and thrombocytopenia
cardiotoxicity

Monitor procainamide level : 4-8 mcg/mL, BP, vitals, ECG, blood counts
lidocaine adverse
CNS effects, respiratory arrest

administer phenytoin to control seizure activity
propafenone adverse
bradycardia, heart failure, dizziness, weakness
propranolol adverse
hypotension, bradycardia, heart failure, fatigue
amiodarone adverse
pulmonary toxicity, sinus bradycardia and AV block, which may lead to heart failure
visual disturbances
liver and thyroid dysfunction, GI disturbances
CNS effects
phlebitis with IV admin
hypotension, bradycardia, AV block
verpamil adverse
bradycardia, hypotension, heart failure, constipation
adenosine adverse
sinus bradycardia, flushing of the face
digoxin adverse
bradycardia, hypotension, toxicity, nausea, vomiting, dysrhythmias- hold dose if heart rate less than 60/min
antidysrhythmic adverse
toxicity is a major concern for antidysrhythmics
can lead to increased cardiac dysrhythmias
antidysrhythmics contraindications
heart block, bradycardia, liver, kidney, respiratory problems
antidysrhythmics interventions/client education
Monitor ECG and blood pressure
monitor for toxicity
monitor digoxin level
antidysrhythmics interactions
concurrent use of atenolol, esmolol, or propanolol may cause additive effects of both meds
beta blockers may cause heart failure
methylxanthines
dipyridamole
corticosteroids
diuretics
thiazides
amphotericin B
(decreased potassium levels)
antacids and metoclopramide