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43 Cards in this Set

  • Front
  • Back
Localized to the ___
cerebellum
Etiologies?
stroke, trauma, tumor, diseases, ETOH (alcohol toxicity)
Generalized damage that includes the cerebellum caused by
MS
Friedreich's ataxia
infection
neuronal degeneration
vascular lesions
True or False

People with ataxic dysarthria often have motor or balance impairments as well
true
True or False

The cerebellum has direct connections to LMNs and UMNs
False

Indirectly connected to LMNs
The cerebellar control circuit is vitally important for __ and ___
motor planning
error control
Damage to the Cerebellum results in ___ and ____ rather than ____ which is seen in UMN lesions less severely and more profoundly with LMN lesions.
discoordination
abnormal involuntary movements (cerebellar tremor)
weakness
The floccunodular lobe connects to the ____ for modulating _______
vestibular mechanism
equilibrium and orientation of head and eyes (eye movements)
The anterior lobe contains a projection area for ____ and is an important component in ___
spinocerebellar proprioceptive information
the regulation of posture, gait and tone in trunk
The posterior lobes are important as a mechanism in ____
coordinating skilled, sequential, and voluntary muscle activity.
Function is impaired on the side ____ to the lesion
ipsilateral
Lesions to the cerebellum/cerebellar tracts cause
hypotonia
incoordination (errors in force, speed, timing range and direction of movements)
Lesions causing speech problems are typically ____, in the ____, or _____.
bilateral
in the vermis (midline)
generalized
Input channels
spinocerebellar pathways
vestibulocerebellar tracts
corticopontocerebellar tracts
Output channels
dentorubrothalamic to cortex
rubrospinal, vestibulospinal and reticulospinal to LMNs
What is important for motor control, particularly for cerebellar coordination of movement
sensory information/feedback
Whose axons leave the cerebellar cortex?
Purkinje cells
The ___ and ____ are most involved in motor speech control
vermis
cerebellar hemispheres
Function of cerebellum
uncertain
helps coordinate timing between single components and movement
scales size of muscular actions
coordinates sequence of agonist & antagonist muscle groups
steadiness of movements
initiation of fast limb movements
General Characteristics

Coordination
disrupted smooth coordination of movement
general discoordination of motor acts
failure to coordinate sensory data with motor performance
General Characteristics

Complex motor acts
decomposition of movement into component parts
General Characteristics

Equilibrium
disequilibrium
broadbased gait
difficulty with balance (standing and seated)
staggering
frequent falls
General Characteristics

Ability to gauge distance, speed and power of movement is characterized by:
dysmetria, the inability to gauge distance, speed and power of movement resulting in overshooting/undershooting target, jerkiness, and rebound effect.
General Characteristics

diadochokinesia
Dysdiadochokinesia: inability to perform rapid alternating movements of muscles
AMRs and SMRs may be irregular, poorly timed, and with excessive excursion
General Characteristics

___tonia
DYStonia
also seen in flaccid
muscle lack normal tone
decreased resistance to passive movement
w/ MS may be spastic (mixed)
General Characteristics

Tremor
intention/kinetic tremor (doesn't begin until movement is initiated)
terminal tremor (increases toward end of movemetn)
General Characteristics

Rate of Movement
voluntary movements are initiated slowly and remain slow throughout (not a great way to distinguish between spastic and ataxic)
General Characteristics

Stretch reflexes
usually normal
General Characteristics

eye movements
may be irregular (nystagmus)
General Characteristics

common pathological reflex
pendulous reflex: knee swings several times
Flocconodular lesions result in
truncal ataxia
disturbance of gait
nystagmus
Anterior lobe lesions result in
gait ataxia
Posterior lobe lesions result in
limb ataxia
hypotonia
intention tremor
incoordination (ipsilateral to lesion)
Lesion to the vermis or bilateral cerebellar hemispheres or diffuse damage results in
ataxic dysarthria!
Confirmatory signs (6)
hypotonia
slow voluntary movements
jerky movements
wide-based gait
intention tremor
dysmetric jaw, face, tongue, AMRs
Speech Characteristics

Articulation
imprecise consonants,
inconsistent errors
BUT ESPECIALLY:
vowel distortions
irregular and transient articulatory breakdowns
irregular AMRs
Speech Characteristics

Resonance
usually intact
occasional hypernasality
nasal emission rare
Speech Characteristics

Phonation
vocal quality fairly normal
may see excessive loudness
harsh voice sometimes seen
Speech Characteristics

Prosody
MOST COMMON AND OBVIOUS
excess and equal stress
prolongation of phonemes and intervals between phonemes
explosive speech/loudness variations
slowed rate
monopitch and monoloudness
scanning speech (pause after each syllable)
Best distinguishing features
irregular/transient articulatory breakdowns
irregular AMRs
distorted vowels
excess/equal prosodic stress
prolonged phonemes
prolonged intervales
loudness variations
monopitch & monoloudness
slowed rate
What is a progressive degenerative spinocerebellar disease of adolescence and early adulthood that primarily involves long tracts of the spinal chord but affects the cerebellum as well?
Friedriech's Ataxia
"lack of order"
ataxia
Main output channel from cerebellum
superior peduncle