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43 Cards in this Set
- Front
- Back
Localized to the ___
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cerebellum
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Etiologies?
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stroke, trauma, tumor, diseases, ETOH (alcohol toxicity)
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Generalized damage that includes the cerebellum caused by
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MS
Friedreich's ataxia infection neuronal degeneration vascular lesions |
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True or False
People with ataxic dysarthria often have motor or balance impairments as well |
true
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True or False
The cerebellum has direct connections to LMNs and UMNs |
False
Indirectly connected to LMNs |
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The cerebellar control circuit is vitally important for __ and ___
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motor planning
error control |
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Damage to the Cerebellum results in ___ and ____ rather than ____ which is seen in UMN lesions less severely and more profoundly with LMN lesions.
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discoordination
abnormal involuntary movements (cerebellar tremor) weakness |
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The floccunodular lobe connects to the ____ for modulating _______
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vestibular mechanism
equilibrium and orientation of head and eyes (eye movements) |
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The anterior lobe contains a projection area for ____ and is an important component in ___
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spinocerebellar proprioceptive information
the regulation of posture, gait and tone in trunk |
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The posterior lobes are important as a mechanism in ____
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coordinating skilled, sequential, and voluntary muscle activity.
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Function is impaired on the side ____ to the lesion
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ipsilateral
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Lesions to the cerebellum/cerebellar tracts cause
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hypotonia
incoordination (errors in force, speed, timing range and direction of movements) |
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Lesions causing speech problems are typically ____, in the ____, or _____.
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bilateral
in the vermis (midline) generalized |
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Input channels
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spinocerebellar pathways
vestibulocerebellar tracts corticopontocerebellar tracts |
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Output channels
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dentorubrothalamic to cortex
rubrospinal, vestibulospinal and reticulospinal to LMNs |
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What is important for motor control, particularly for cerebellar coordination of movement
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sensory information/feedback
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Whose axons leave the cerebellar cortex?
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Purkinje cells
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The ___ and ____ are most involved in motor speech control
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vermis
cerebellar hemispheres |
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Function of cerebellum
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uncertain
helps coordinate timing between single components and movement scales size of muscular actions coordinates sequence of agonist & antagonist muscle groups steadiness of movements initiation of fast limb movements |
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General Characteristics
Coordination |
disrupted smooth coordination of movement
general discoordination of motor acts failure to coordinate sensory data with motor performance |
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General Characteristics
Complex motor acts |
decomposition of movement into component parts
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General Characteristics
Equilibrium |
disequilibrium
broadbased gait difficulty with balance (standing and seated) staggering frequent falls |
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General Characteristics
Ability to gauge distance, speed and power of movement is characterized by: |
dysmetria, the inability to gauge distance, speed and power of movement resulting in overshooting/undershooting target, jerkiness, and rebound effect.
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General Characteristics
diadochokinesia |
Dysdiadochokinesia: inability to perform rapid alternating movements of muscles
AMRs and SMRs may be irregular, poorly timed, and with excessive excursion |
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General Characteristics
___tonia |
DYStonia
also seen in flaccid muscle lack normal tone decreased resistance to passive movement w/ MS may be spastic (mixed) |
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General Characteristics
Tremor |
intention/kinetic tremor (doesn't begin until movement is initiated)
terminal tremor (increases toward end of movemetn) |
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General Characteristics
Rate of Movement |
voluntary movements are initiated slowly and remain slow throughout (not a great way to distinguish between spastic and ataxic)
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General Characteristics
Stretch reflexes |
usually normal
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General Characteristics
eye movements |
may be irregular (nystagmus)
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General Characteristics
common pathological reflex |
pendulous reflex: knee swings several times
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Flocconodular lesions result in
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truncal ataxia
disturbance of gait nystagmus |
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Anterior lobe lesions result in
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gait ataxia
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Posterior lobe lesions result in
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limb ataxia
hypotonia intention tremor incoordination (ipsilateral to lesion) |
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Lesion to the vermis or bilateral cerebellar hemispheres or diffuse damage results in
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ataxic dysarthria!
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Confirmatory signs (6)
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hypotonia
slow voluntary movements jerky movements wide-based gait intention tremor dysmetric jaw, face, tongue, AMRs |
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Speech Characteristics
Articulation |
imprecise consonants,
inconsistent errors BUT ESPECIALLY: vowel distortions irregular and transient articulatory breakdowns irregular AMRs |
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Speech Characteristics
Resonance |
usually intact
occasional hypernasality nasal emission rare |
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Speech Characteristics
Phonation |
vocal quality fairly normal
may see excessive loudness harsh voice sometimes seen |
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Speech Characteristics
Prosody |
MOST COMMON AND OBVIOUS
excess and equal stress prolongation of phonemes and intervals between phonemes explosive speech/loudness variations slowed rate monopitch and monoloudness scanning speech (pause after each syllable) |
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Best distinguishing features
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irregular/transient articulatory breakdowns
irregular AMRs distorted vowels excess/equal prosodic stress prolonged phonemes prolonged intervales loudness variations monopitch & monoloudness slowed rate |
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What is a progressive degenerative spinocerebellar disease of adolescence and early adulthood that primarily involves long tracts of the spinal chord but affects the cerebellum as well?
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Friedriech's Ataxia
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"lack of order"
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ataxia
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Main output channel from cerebellum
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superior peduncle
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