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34 Cards in this Set

  • Front
  • Back
What is AKI?
Acute kidney injury
What is ARF?
A severe AKI
How is ARF managed?
RRT (renal replacement therapy)
What are 3 lab values, along with +/- oliguria, that are indicative of an AKI?
Increased Urea

Increased Creatinine

Increased K+


There are three categories/causes of AKI, what are they?
Pre renal causes

Renal/intra-renal/intrinsic


Post renal causes



What are the pre renal causes of AKI, which make up for 55-60% of all ARF
Hypovolemia

Vasodilation (decreased SVR)


Decreased CO


Renal vascular obstruction


*They are all low perfusion

What happens to the Na values in pre renal AKI? What can happen to the renal tubules?
Because Na is failed to be excreted, Na increases, water retention also increases. The renal tubules can undergo acute tubular necrosis because of lack of blood flow.
What are the intrarenal causes of AKI?
Renal ischemia

Nephrotoxins



Blocked tubules


Renal diseases


Acute Tubular necrosis


*Anything that causes direct damage to the kidneys


What is the most common reason behind ARF?
ATN
What is ATN caused by (there are 2 main)
Ischemia

Nephrotoxins

What are the post renal causes of AKI
BPH

Prostate cancer

Calculi

*anything that obstructs urinary flow





What are the three phases of ATN
Initiation phase

Maintenance phase


Recovery phase



How long is the initiation phase of ATN?
From hours to days
How long is the oliguric (maintenance) phase?
from days to weeks
What's the difference between anuric, oliguric, or non oliguric?
Anuric (no urine, less than 100 ml a day)

Oliguric (low urine output, less than 400 ml a day)


Non oliguric (more than 500 ml a day, little waste, blood not filtered)

What does the body become acidotic during the maintenance phase, and what's the body do to compensate?
Cannot excrete bicarb as a buffer



For compensation it does


Kussmaul's respirations


Become hyperkalemic

Why does hypercalcemia ensue?
Kidney's cannot activate Vitamin D

So it impairs GI absorption


Leads to hypocalcemia


But PTH secretion causes more


Calcium and PO4 from the bone to be released



What are other concerns during the maintenance phase?
Waste product accumulation of urea and creatinine

Anemia

Neurological disorders




What to monitor in the recovery phase?
Gradual increase in urine output

Monitor for hypotension, hypovolemia, Decreased K, Na, and dehydration




If dehydration occurs, it'll result in hypotension, which will make us start all over again








What marks the recovery phase?
GFR increases

BUN/Creatinine decreases

What are diagnostic tests we can do for ARF?
Labs - Creatinine, BUN, CBC, electrolytes

Creatinine clearance test

CT

Renal scan





The best thing to do with ARF, is to prevent it. But how?
Hydrate post angiogram

Fluids for hypovolemia

Manage heart failure

Monitor renal function and nephrotoxic drugs




Monitoring of fluid and electrolyte status





The management of ARF includes
Fluid management

Treat hyperkalemia


Nutritional therapy


Renal replacement therapy

How do you treat hyperkalemia?
Stabilize myocardiom with calcium gluconate



Enhance K removal with


-Kayexalate


-Diuretics (potassium removing ones like Lasix)


- Dialysis




Shift potassium back into cells with


-glucose and insulin combination


-salbutamol


-NaHCO3




Dietary restrictions


Stop K enhancing meds





Why is nutritional therapy required?
To stop catabolism which creates urea



To stop PTH from leeching more Ca and Phosphate from the bones

The most common form of RRT for AKI is?
Hemodialysis

- done when you need a rapid effect over short time

- done intermittently 4 hr/day, 3 or 4 times a week


CRRT

- over 24 hours or longer

- slower rate of change

- preferred for hemodynamically unstable patients








What are some nephrotoxic chemicals (outside of drugs)?
insecticides, organic solvents, cleaning agents
What are high risk populations?
Trauma

Elderly


Burns


UTI


Chemotherapy

What is recovery based on? 3 factors
Age, general condition

Duration of Oliguria


Severity of Nephron damage



1. A patient developed shock aftera severe MI and now has RF. The pt fam asks the nurse why the ptdeveloped acute

A.A decrease in the blood flow through the kidneys


B.An obstruction of urine flow from the kidneys==

A. A decrease in the blood flow through the kidneys
2.The serum K level of a pt in ARF is 6.2. Which of thefollowing interventions may be ordered (select all that apply, circle on scantron,don’t get them all u don’t get the question right)

A.Polystyrenesulfonate (Kayexalate)


B.Regularinsulin


C.50%dextrose


D.Dpotassiumreplacement therapy


E.Clalciumgluconate

ABCE
3.In the Oliguric(Maintenance) phase of ARF the nurse should assess the ptfor

A.Pulmonaryedema


B.Metabolicalkalosis


C.Hypotension


D.Hypokalemia

A.Pulmonary edema
4.In the oligruicphase of ARF the nurse can expect to find which of the following breathingpatterns?

A.Normal


B.Apneic


C.Cheyne-Stokes


D.Kussmaul’s

D.Kussmaul’s
5.Duringthe oliguric(maintenance phase) the nurse would expect to find which of thefollowinglab values? (Select all that apply)A.Hyponatremia

B.Hyperkalemia


C.Hypercalcemia


D.Hypophosphatemia


E.Hypocalcemia

ABE