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74 Cards in this Set

  • Front
  • Back
THE HEART IS ALSO A
THE HEART AS A PUMP
THE HEART HAS ATRIAL
SYNCYTIUM
THE HEAT HAS VENTRICAL
SYNCYTIUM
FIBROUS INSULATOR EXISTS BETWEEN ATRIUM AND VENTRICLE
CARDIAC MUSCLE TO PUMP THE BLOOD
THE CARDIAC MUSCLE HAS THESE TYPES OF FILAMENTS
THICK = MYOSIN, THIN =ACTIN
CARDIAC MUSCLE HAS THSE LOW RESISTANCE INTERCALATED DISKS WHICH CONTAIN THIS
GAP JUCTION
TRUE OR FALSE CARDIAC MUSCLE IS VERY WELLL CONTECTED OR NOT SO MUCH
WELL CONNECTED
CARDIAC MUSCLEIS PRIMARILY MADED UP OF ONE PRINCIPLE ELEMENT
PROTEIN
ACTION POTENTIAL OF CARDIAC MUSCLE AT REST IS
-85 TO -95 MILLIVOLTS
THE ACTION POTENTIAL OF CARDIAC MUCSLE IS
105 MILIVOLTS
HOW LONG DOES THE CARDIAC PLATEAU LATS
-0.2 -0.3 SEC IN VENTRICALUR MUSCLE ( NOTE THIS IS MUCH LONGER THEN SKELETAL MUSCLE)
PHASE 0
Fast Na+ channels open then slow Ca++ channels
PHASE 1
K+ CHANNELS OPEN
PHASE 2
CA++ CHANNELS OPEN MORE
PHASE 3
K+ CHANNELS OPEN MORE
PHASE 4
RESTING MEMBRAINE POTENTIAL
WHAT ION CAUSES THE PLATUE PHASE OF THE MEMBRANCE POTENTIAL
CA++
REFACTORY PERIOD OF CARDIAC MUSCLE DURING THIS TIME THE MEMBRANE IS AT REST AND CAN NOT BE RE-EXCITED TO DUE
INACTIVATED NA+ CHANNELS
THE REFACTORY PERIOD IN THE VENTRICLES LAST
0.25 -0.30 SEC IN VENTRICLES
THE REFACTORY PERIOD IN THE ATRIA LAST
0.15 SEC I N ATRIA
LIST ONE OF THE RESULTS OF THE ACTION POTENTIAL IN CARDIAC MUSCLE
RELEASE OF CA++ FROM THE SARCOPLASMIC RETICULUM R/T T TUBLES BAGS O CALCIUM WHICH THEN NEED ATP TO PUMP CA++ OUT
CARDIAC CYCLE
SYSTOLE & DIASTOLE
SYSTOLE
MUSCLE STIMULATED BY AP AND CONTRACTING
DIASTOLE
MUSCLE REESTABLISHING NA+/K+/CA++ GRADIENT IS RELAXING
EKG COMPONETS
P WAVE, QRS –VENTRICAL WAVE, T WAVE VENTRICULAR REPOL
ATRIAL PRESSURE WAVE
A WAVE (ATRIAL CONTRACTION), C WAVE (VENTRICULAR CONTRACTION), V WAVE (FLOW OF BLOOD INTO ATRIA)
A, C, V WAVE FORMS ARE RELATED TO WHAT PRESSURES WAVE ATRIAL OR VENTRICULAR
ATRIAL
ANOTHER NAMBE FOR ISOVOLUMIC RELAXATION
DIASTOLE
THIS WILL SLOW BLOOD FLOW INTO THE VENTRICLE
DIASTASIS
ATRIAL SYSTOLE –EXTRA BLOOD ACCOUNTS FOR WHAT ____% OF THE FILLING
25
ANOTHER NAME FOR ISOVOLUMIC CONTRACTION
SYSTOLE
DURING DIASTOLE A-V VALUES OPEND OR CLOSED
OPEN
DURING SYSTOLE A-V VALUES OPEN OR CLOSED
CLOSED (VENTRICULAR PRESS> ATRIAL PRESS)
ENGERY OF BLOOD EQUATION
P +MV2/2 = PRESSURE +KINETIC ENERGY
EF AT END DIASTOLIC VOL
120 ML
EF AT END SYSTOLIC VOL
50 ML
EJECTION VOL (STROKE VOL) =
70ML
CARDIAC OUTPUT CALCULATION HR 70 * STROKE VOLUME (70) =
4900 ML/MIN 5L
TO CALCULATE DV 180 – SV 20 = 160 , 160* HR 100 =
16000 ML/MIN
AORTIC PRESSURE STARTS __I/D___ DURING SYSTOLE AFTER THE AORTIC VALVE OPENS
INCREASING
AORTIC PRESSURE AT THE END STARTS TO ____I/D___ TOWARD THE END OF THE EJECTION PHASE
DECREASE
AORTIC PRESSURE DECREASES SLOWLY DURING DIASTOLE BECAUSE OF THE ______ OF THE AORTA
ELASTICITY ( IT WILL EXPAND, STRECHT ,RETRACT)
THESE ITEMS HELP PREVENT BACK FLOW (CT)
CHORADE TENDINEAE ATTACHED TO A-V VALVES
THESE ATTACH TO CHORADETENDINEAE AND PREVENT BACK FLOW DURING SYSTOLE
PAPILLARY MUSCLE
FRANK STARLING MECHANISM
WITHIN PHYSIOLOGICAL LIMITS THE HEART PUMPS ALL THE BLOOD THAT COMES TO IT WITHOUT EXCESSIVE DAMMING IN THE VEINS
WITHIN PHHYSIOLOGICAL LIMITS THE HEART PUMPS ALL THE BLOOD THAT COMES TO IT WITHOUT EXCESSIVE DAMMING IN THE VEINS
FRANK STARLING MECHANISM
EXTRA STRECTCH ON CARDIAC MYOCYTES MAKES ACTIN AND MYOSIN FILAMENTS _______INTERDIGITATE TO A MORE OPTIMAL DEGRESS OF FORCE GENERATIO
INTERDIGITATE
AUTONOMIC -SYMPATHETIC STIMULATION CAUSES I/D HR + INCR CONTRACTILITY WITH HR = 180-200 AND C.O. = ______L/MIN
Increased 15-20L/min
AUTOMONIC – PARASYMPATHEITIC STIMULATION I/D HR MARKEDLY AND DECR CONTRACTILITY SLIGHTLY.
DECREASES
DURING PAPARSYMPATHEIC STIMULATION VAGAL FIBERS GO TO ______?
ATRIA
TACHYCARDIA = FAST HEART REATE AND CAN DECREASE C.O. WHY?
NOT ENOUGH TIME TO SUFFICANTLY FILL THE HEART DURING DIASTOLE.
VASCULAR DISTENSIBILITY IS
THE FRACTIONAL INCREASE IN VOLUME FOR EACH mmHg RISE IN PRESSURE
VEINS ARE ____ TIMES MORE DISTENSIBLE THAN ARTERIES
8 TIMES
PULMONARY ARTERIES ARE RELATIVELY DISTENSIBLE TRUE OR FALSE
TRUE
VASCULAR DISTENSIBILITY= INCRESE IN VOLUME , INCREASE IN PRESSURE X
ORIGINAL VOLUME
HEIGHT OF PRESSURE PULSE IS THE SYSTOLIC PRESSURE
120 mmHg
THE LOWEST POINT IS DIASTOLCI PRESSURE
80mmHg
WHAT IS THE DIFFENCE BETTWEEN SYSTOLIC AND DIASTOLIC PRESSURE IS
PULSE PRESSURE 40mmHg
THE CAPACITANCE OF VEINS IS _____ TIMES THAT OF ARTERIES
24 TIMES
ATERIAL COMPLIANCE – DECREASE IN COMPLIANCE = I/D IN PULSE PRESSURE
INCREASE PULSE PRESSURE
ATERIAL COMPLIANCE - INCREASE IN COMPLIANCE = I/D IN PULSE PRESSURE
DECREASE PULSE
INCREASE STROKE VOL INCREASE PULSE PRESSURE AND IF YOU DECREASE ______ YOU DECREASE PULSE PRESSURE
STROK VOL
WHAT % OF BLOOD IS IN THE VEINS
60%
THESE LOCATIONS IN THE BODY SERVE AS RESERVIORS ARE
SPLEEN, LIVER, LARGE ABDOMICAL VEINS
______ PRESSURE TENDS TO INCREASE VENOUS PRSSURES IN THE LEGS
ABDOMINAL PRESSURE
RIGHT ATRIAL PRESSURE CAUSED BLOOD TO BACK UP INOT WHAT SYSTEM, THERE FORE INCREASING _____ PRESSURE
VENSOUS SYSTEM, VENOUS
RIGHT ATRIUM PRESSURE IS CALLED
CENTRAL VENOUS PRSSURE
CENTRAL VENOUS PRESSURE IS NORMALLY 0 mmHg, but can be ______ mmHg
20-30
INTRACELLULAR FLUID _____L =
28.0 L
PLASMAS = ___ L =
3.0 L
INTERSITIAL FLUID = ____
11.0L
WHAT TWO FACTORS DECREASE TOTAL BODY WATER
INCREASING OBESITY AND AGE
DISTRIBUTION ACROSS A CELL MEMBRANE IS REALTED TO OSMOTIC FORCES
ELECTROLYTES NA+ CA++ K+
DISTRIBUTION ACROSS CAPILLARIES IS DETEMINED BY HYDROSTATIC AND ______ _______ FORCES
COLLOID OSMOTIC