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29 Cards in this Set
- Front
- Back
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6 areas fed by the RCA
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right atrium
most of right ventricle Diaphragmatic surface of lt ventricle Posterior 1/3 of IV septum SA node- 60% of people AV node- 80% of people |
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6 areas fed by the LCA and it's branches
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Left atrium
Most of left ventricle Portion of right ventricle Anterior 2/3 of IV septum AV bundle Supplements bld supply to SA/AV nodes |
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2 branches of the LCA
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Left anterior descending
Circumflex |
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What does dominant mean in relation to coronary arteries
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blood supply to diaphragmatic surface of the heart
origin of posterior IV artery |
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Which CA is most often dominant
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RCA
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Veins that drain directly into the heart
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thebesian
anterior cardiac veins coronary sinus |
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Drains the anterior aspect of the RV
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anterior cardiac veins
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Drains the LV
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coronary sinus
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2 things that will decrease CPP
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decrease in DBP
increase in LVEDP |
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When does the greatest flow rate through the RCA occur
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systole
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What is the affect of adenosine as it relates to the heart
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vasodilation
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What are 3 factors that affect myocardial oxygen demand
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HR
Force of contraction Wall tension |
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Which 3 factors affect wall tension
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EDP- directly
Radius of ventricular chamber-directly Thickness of ventricular wall- indirectly |
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When during the cardiac cycle is the greatest amount of O2 consumed by the myocardium
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isovolumetric ctx- 50%
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Cardiac work formula
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MAP X CO
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TPR formula
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MAP-CVP/CO
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SVR formula
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MAP-CVP/CO X 80
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Total pulmonary vascular resistance formula
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Mean Pulm Pressure - LAP/ CO
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How does an increase in BP increase blood flow
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Increases pressure gradient
decreases resistance |
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What two factors affect the critical closing pressure
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Blood pressure
Vasomotor tone- SNS |
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Recipricol of resistance
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conductance
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CO that goes to the heart
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5%
250 mL/min 80-100 mL/min/100 gm heart tissue |
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Primarily responsible for decreasing blood flow in the subendocardial blood vessels of the LV during systole
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increased LV pressure
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Area that is most prone to MI's
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subendocardial region of LV
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What happens to cerebral blood vessels if CPP is inadequate
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vasodilation
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What happens to coronary blood vessels if CPP is too great
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vasoconstriction
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Vasodilator produced in response to decreased coronary perfusion
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adenosine
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How does the SNS cause vasodilation in the coronary arteries
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indirectly by increasing HR and SOC, therefore the MVO2 of the heart
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MVO2 of the heart at rest
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8-10 mL/min/100 gm
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